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  • Kidney S: Pathophysiology of Pregnancy Induced Hypertension

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    Khristine Anne Fabay
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    The document summarizes the pathophysiology of pregnancy induced hypertension. It explains that reduced resistance to angiotensin and norepinephrine by the mother leads to increased blood volume, cardiac output, and generalized vasoconstriction, raising blood pressure. This decreases oxygen and nutrient perfusion to organs like the kidneys, uterus, and placenta. Kidney issues include increased permeability causing proteinuria, decreased glomerular filtration rate, and water retention exacerbating edema. Complications for the placenta and fetus include growth restriction and low birth weight.

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    The document summarizes the pathophysiology of pregnancy induced hypertension. It explains that reduced resistance to angiotensin and norepinephrine by the mother leads to increased blood volume, cardiac output, and generalized vasoconstriction, raising blood pressure. This decreases oxygen and nutrient perfusion to organs like the kidneys, uterus, and placenta. Kidney issues include increased permeability causing proteinuria, decreased glomerular filtration rate, and water retention exacerbating edema. Complications for the placenta and fetus include growth restriction and low birth weight.

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    11 views2 pages

    Kidney S: Pathophysiology of Pregnancy Induced Hypertension

    Uploaded by

    Khristine Anne Fabay
    The document summarizes the pathophysiology of pregnancy induced hypertension. It explains that reduced resistance to angiotensin and norepinephrine by the mother leads to increased blood volume, cardiac output, and generalized vasoconstriction, raising blood pressure. This decreases oxygen and nutrient perfusion to organs like the kidneys, uterus, and placenta. Kidney issues include increased permeability causing proteinuria, decreased glomerular filtration rate, and water retention exacerbating edema. Complications for the placenta and fetus include growth restriction and low birth weight.

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    PATHOPHYSIOLOGY OF PREGNANCY INDUCED HYPERTENSION

    Reduced resistance of the mother to the pressor effects of angiotensin & nor- e Increase thromboxane A2 & decrease prostacyclin
    INCREASE BLOOD VOLUME/ INCREASE CARDIAC OUTPUT INJURY TO ENDOTHELIAL CELLS (DUE TO POOLING OF BLOOD IN THE VENOUS CIRCULATION PLATELETS WILL GO TO THE SITE OF INJURY GENERALIZED VASOSPASMS VASOCONSTRICTION

    INCREASE BLOOD PRESSURE DECREASE OXYGEN & NUTRIENT PERFUSION TO THE DIFFERENT ORGANS

    THROMBOCYTOPENIA

    KIDNEY S
    E D E M A

    UTERU S
    DECREASE OXYGEN & NUTRIENT TO PLACENTA

    PANCR EAS
    ELEVATED SERUM AMYLASE CREATININE RATIO

    INCREASE PERMEABILITY PROTEINURIA

    DECREASE GFR ELEVATED BUN, URIC ACID, CREATININE LEVEL INCREASE TUBULAR REABSORPTION OF Na WATER RETENTION
    PULMONARY EDEMA

    PLACENTAL INSUFFICIENCY

    ORTHOPNEAVISUAL DISTURBANCES CEREBRAL EDEMA (BLURRING OF VISION) (HEMORRHAGE LEADS TO BLINDNESS) VERTIGO

    RESTRICTED UTERINE GROWTH

    LOW BIRTH WEIGHT

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