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  • Benign Prostatic Hyperplasia Hypertensive Nephroschelorosis

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    Riin Irasusta
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    Benign prostatic hyperplasia can develop due to age, genetics, and race, causing urinary retention and obstruction. Untreated urinary tract infections from the bacteria entering the urinary tract can lead to kidney damage, chronic kidney disease, and systemic complications like respiratory distress and anemia as infection spreads and kidney function declines.

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    patho icu

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    Benign prostatic hyperplasia can develop due to age, genetics, and race, causing urinary retention and obstruction. Untreated urinary tract infections from the bacteria entering the urinary tract can lead to kidney damage, chronic kidney disease, and systemic complications like respiratory distress and anemia as infection spreads and kidney function declines.

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    13 views2 pages

    Benign Prostatic Hyperplasia Hypertensive Nephroschelorosis

    Uploaded by

    Riin Irasusta
    Benign prostatic hyperplasia can develop due to age, genetics, and race, causing urinary retention and obstruction. Untreated urinary tract infections from the bacteria entering the urinary tract can lead to kidney damage, chronic kidney disease, and systemic complications like respiratory distress and anemia as infection spreads and kidney function declines.

    Copyright:

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    Download as PDF, TXT or read online from Scribd
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    of 2

    Precipitating Compromised immune system Urinary tract obstruction Diet Urinary Retention Large numbers of alpha-1 andreg.

    receptors in smooth muscle of stroma and capsule of the prostate

    Predisposing Age Hereditary Race

    Chronic HPN

    stimulted Inc. smooth muscle tone Benign prostatic hyperplasia Urethral compression Obstruction of the bladder outlet Outpouching pf the lining of bladder Restrict flow of urine Urinary frequency Bladder weakens

    Presence of micro organism at Urinary Tract


    Inflammatory response untreated

    Narrowing of preglomular arteries


    Dec. glomeral blood flow Glomerular ischemia Hypertensive Nephroschelorosis Hardening of the kidney wall

    Micro organism ascends to kidney Microorg. Reaches the bloodstream urosepsis

    Ischemic renal injury Acute kidney injury Reduction of renal blood flow Dec. glomerular filtration rate Kidney damage Chronic kidney disease Endproduct of CHON accumulates

    Bladder diverticula

    Bacteria releases endotoxin


    Spread of infection Fluid leaks in to the lungs

    creatinine

    BUN

    Dec. oxygen absorption Respiratory distress Less oxygen in the blood

    Dec. production of erythropoetin anemia

    hypocalcemia

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