Glaucoma is a condition characterized by increased pressure within the eye. Acute angle closure glaucoma is a type of glaucoma caused by sudden closure of the anterior chamber angle, leading to a rapid rise in intraocular pressure. Signs include severe eye pain, blurred vision, nausea, mid-dilated and non-reactive pupil, cloudy cornea, very high eye pressure, and optic nerve damage if not treated quickly. Treatment involves intravenous medications to lower pressure urgently along with laser treatment to permanently open the eye's drainage angle.
Glaucoma is a condition characterized by increased pressure within the eye. Acute angle closure glaucoma is a type of glaucoma caused by sudden closure of the anterior chamber angle, leading to a rapid rise in intraocular pressure. Signs include severe eye pain, blurred vision, nausea, mid-dilated and non-reactive pupil, cloudy cornea, very high eye pressure, and optic nerve damage if not treated quickly. Treatment involves intravenous medications to lower pressure urgently along with laser treatment to permanently open the eye's drainage angle.
Glaucoma is a condition characterized by increased pressure within the eye. Acute angle closure glaucoma is a type of glaucoma caused by sudden closure of the anterior chamber angle, leading to a rapid rise in intraocular pressure. Signs include severe eye pain, blurred vision, nausea, mid-dilated and non-reactive pupil, cloudy cornea, very high eye pressure, and optic nerve damage if not treated quickly. Treatment involves intravenous medications to lower pressure urgently along with laser treatment to permanently open the eye's drainage angle.
no no Profuse tearing Pain no ++ (globe) +++(nauseating) ++(on blinking) Photophobia no +++ + ++ Blurred vision no ++ +++ varies Pupil normal smaller Fixed in mid- dilation Same/smaller Cornea normal Keratatic precipitate cloudy Infiltrate, edema Intraocular pressure normal varies Increased markedly Normal or increased Others Large Posterior synechiae Colored halos nausea & vomiting pale, cupped optic disc Signs (Fig. 9.19) Lids may be oedematous, Conjunctiva is chemosed, and congested, (both conjunctival and ciliary vessels are congested), Cornea becomes oedematous and insensitive, Anterior chamber is very shallow. Aqueous flare or cells may be seen in anterior chamber Angle of anterior chamber is completely closed as seen on gonioscopy (shaffer grade 0), Iris may be discoloured, Pupil is semidilated, vertically oval and fixed. It is non-reactive to both light and accommodation, IOP is markedly elevated, usually between 40 and 70 mm of Hg, Optic disc is oedematous and hyperaemic, Fellow eye shows shallow anterior chamber and a narrow angle (latent angle closure glaucoma). Circumcorneal injection about 4 mm from the cornea the anterior ciliary vessels perforate the sclera to supply the ciliary body. They therefore dilate as the result of any congestion of the anterior segment. Comeal oedema the corneal endothelium has a metabolic pump mechanism which pumps water out of the corneal stroma into the aqueous. 40mmhg : pump cannot cope and water fails to pass out of the cornea Dilated pupil closure of the angle of the anterior chamber is the cause of the acute attack, this may be precipitated by dilatation of the pupil. In turn the acute pressure rise causes paralysis of the iris muscles so that the pupil remains fixed in semi-dilatation Optic atrophy and permanent visual loss optic nerve head as the intraocular pressure rises above capillary and arteriolar pressure, causing at first congestion and later a failure of perfusion of vessels supplying the nerve. Management Administered Acetazolamide intravenously and orally, together with topical pilocarpine and beta-blockers.