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  • Pathway Gagal Jantung PDF

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    Iffah Ahmad
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    This document outlines the pathway of heart failure, beginning with abnormalities in the myocardium or increased pressure loads that decrease cardiac contractility and output. This leads to forward heart failure where blood supply to tissues is reduced, causing metabolic changes and fatigue. In response, the sympathetic nervous system is activated, increasing stress hormones. Backward heart failure then occurs as elevated pressures in the heart cause fluid backup in the lungs and systemic veins, leading to pulmonary and systemic edema. Rising right heart pressures further impair cardiac function in a worsening cycle that can progress to right heart failure and death if not treated.

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    This document outlines the pathway of heart failure, beginning with abnormalities in the myocardium or increased pressure loads that decrease cardiac contractility and output. This leads to forward heart failure where blood supply to tissues is reduced, causing metabolic changes and fatigue. In response, the sympathetic nervous system is activated, increasing stress hormones. Backward heart failure then occurs as elevated pressures in the heart cause fluid backup in the lungs and systemic veins, leading to pulmonary and systemic edema. Rising right heart pressures further impair cardiac function in a worsening cycle that can progress to right heart failure and death if not treated.

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    9K views2 pages

    Pathway Gagal Jantung PDF

    Uploaded by

    Iffah Ahmad
    This document outlines the pathway of heart failure, beginning with abnormalities in the myocardium or increased pressure loads that decrease cardiac contractility and output. This leads to forward heart failure where blood supply to tissues is reduced, causing metabolic changes and fatigue. In response, the sympathetic nervous system is activated, increasing stress hormones. Backward heart failure then occurs as elevated pressures in the heart cause fluid backup in the lungs and systemic veins, leading to pulmonary and systemic edema. Rising right heart pressures further impair cardiac function in a worsening cycle that can progress to right heart failure and death if not treated.

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    of 2

    PATHWAY GAGAL JANTUNG

    Kelainan miokardium
    (kardiomiopati, miokarditis)
    Peningkatan beban tekanan (sentralstenosis
    aorta, periferhipertensi sistemik)
    Peningkatan beban volume (regurgitasi
    katup, pirau, peningkatan beban awal)
    Kontraktilitas ventrikel
    Beban sistol me
    Kontraktilitas
    Hambatan pengosongan ventrikel
    COP
    Beban jantung me
    Right heart failure
    Gagal Jantung
    Gagal pompa v. kiri
    Forward Failure
    Suplai darah jaringan
    Metab. anaerob
    Asidosis metabolik
    ATP
    Fatigue
    Intoleransi aktivitas
    Suplai O
    2
    di otak
    kesadaran
    Sinkop
    Me respon simpatik
    Merangsang pengeluaran
    Katekolamin
    Vasokonstriksi arteria perifer
    Renal Flow Vasokonstriksi
    kulit
    Menghambat
    pelepasan panas
    Pengeluaran
    keringat
    Perfusi yg kurang
    k otot rangka
    Kelemahan &
    keletihan
    RAA
    Merangsang
    simpatik
    adregenik
    Aldosteron me
    ADH me
    Retensi Na
    +
    /H
    2
    O
    Preload + afterload
    Kelebihan volume cairan
    vaskular
    Backward Failure
    EDV
    Oedema sistemik
    Penurunan perfusi
    oksigen ke jaringan
    Tirah baring
    Gangguan Integritas
    kulit
    LVEDP
    LAP
    LAP diteruskan k
    belakang (vaskular paru)
    Tekanan kapiler &
    vena paru-paru
    Tekanan hidrostatik>tekanan
    onkotik vaskular
    Transudasi cairan ke dlm
    alveoli
    Oedema paru
    Iritasi mukosa
    paru
    Iritasi mukosa paru
    Penumpukan sekret
    Menghambat pertukaran O
    2
    &
    CO
    2
    di paru
    Gangguan pertukaran gas
    Gagal pompa v. kanan
    Tekanan diastole
    Bendungan a. kanan
    Bendungan vena sistemik
    Penimbunan as. laktat
    Hepar
    Hepatomegali
    Mendesak diafragma
    Sesak napas
    Pola napas tidak
    efektif
    Beban v. Kanan me
    Hipertropi v. kanan
    penyempitan v.
    kanan
    By. Iffah (C 121 10 103)


    Referens:
    Noer, Sjaifoellah. 1996. Buku ajar ilmu penyakit dalam jilid I, edisi ketiga. FKUI : Jakarta
    Price, Sylvia A. 2002. Patofisiologi (konsep klinis proses-proses penyakit), edisi keempat. EGC: Jakarta.

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