Professional Documents
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Graded Pat 3
Graded Pat 3
Age: 48
Gender: Female
Served/Veteran: Unobtainable
3 HISTORY OF PRESENT ILLNESS: (Be sure to OLDCART the symptoms in addition to the hospital course)
Patient is a 48 year old African American woman who presented to the Emergency Department of Bayfront Medical
Center on 3/11/14 after a family member found her in her home nonverbal and unable to move the right side of her body.
After CT scan and MRI, she was found to have had a hemorrhagic stroke of large basal ganglia bleed and left
intraparenchymal hemorrhage and was admitted to the Neurointensive Care Unit. Patient had an abnormal
electroencephalogram (EEG) which showed evidence of a focus of left temporal paroxysmal activity and suggested that
the patient was at risk for seizures. Patient was diagnosed with right hemiplegia and neurogenic dysphagia. On 3/24/14 a
complete upper gastrointestinal endoscopy with placement of G-tube was performed because of the patients neurogenic
dysphagia. The patient is now on A2 and remains nonverbal and unable to move the right side of her body, however, she
is able to follow commands and move the left side of her body.
5 PATHOPHYSIOLOGY: (include APA reference and in text citations) (Mechanics of disease, risk factors, how to
diagnose, how to treat, prognosis, and include any genetic factors impacting the diagnosis, prognosis or
treatment)
A cerebrovascular accident (stroke) is the interruption of normal blood flow in one or more of the blood vessels that
supply the brain. The tissues become ischemic, leading to hypoxia or anoxia with destruction or necrosis of the neurons.
In its mildest form, a cerebrovascular accident is so minimal that it is almost unnoticed. In its most severe state,
hemiplegia, coma, and death result. Cerebrovascular accidents are classified pathophysiologically as global hypoperfusion
(as in shock), ischemic (thrombotic, embolic), or hemorrhagic. Risk factors for stroke include arterial hypertension and
smoking. Diabetes, insulin resistance, polycythemia, thrombocythemia, atrial fibrillation, and the presence of lipoproteinA all increase the risk for ischemic stroke. Stroke is the third leading cause of death in the US and affects more than
600,00 Americans annually with 160,000 deaths per year. Strokes tend to run in families and are more common in men at
younger ages. The incidence is about 2 times greater in blacks than whites. Blacks between the ages of 55 and 64 who live
in Southern states are about 50% more likely to die of stroke than blacks of the same age who live in the North. People
with both hypertension and type 2 diabetes have a fourfold increase in stroke incidence and an eightfold increase in stroke
mortality. Hemorrhagic stroke (intracranial hemorrhage) is the third most common cause of cerobrovascular accident.
Hypertension, ruptured aneurysms or vascular malformation, bleeding into a tumor, or hemorrhage associated with
anticoagulants or clotting disorders, head trauma, or illicit drug use are common causes. A hypertensive hemorrhage is
associated with significantly increased systolic and diastolic blood pressure measurements over several years and usually
occurs in the brain tissue. A mass of blood is formed and grows, displacing and compressing adjacent brain tissue.
Rupture or seepage into the ventricular system occurs in many cases. Hemorrhages are described as massive (several
centimeters in diameter), small (1 to 2 cm in diameter), slit (lies in the subcortical area), or petichial (the size of a pinhead
bleed). The most common sites for hypertensive hemorrhages are in the putamen of the basal ganglia, the thalamus, the
cortex and subcortex, the pons, the caudate nucleus, and the cerebral hemispheres. Clinical manifestations vary,
depending on the location and size of the bleed. Individuals experiencing intracranial hemorrhage from a ruptured or
leaking aneurysm have one of three sets of symptoms: onset of an excruciating generalized headache with an almost
immediate lapse into an unresponsive state, headache but with consciousness maintained, or sudden lapse into
unconsciousness. If bleeding spreads into the brain tissue, hemiparesis/paralysis, dysphagia, or homonymous hemianopia
may be present. MRI and magnetic resonance angiography (MRA) are used to diagnose stroke. Treatment of an
intracranial bleed, regardless of cause, focuses on stopping or reducing the bleeding, controlling the increased intracranial
pressure, preventing a rebleed, and preventing vasospasm. Occasionally an attempt is made to evacuate or aspirate the
blood. Once a deep unresponsive state occurs, the person rarely survives. The immediate prognosis is grave. If the person
survives, recovery of function often is possible (Huether and McCance, 2012).
5 MEDICATIONS: [Include both prescription and OTC; home (reconciliation), routine, and PRN medication. Give trade and
generic name.]
Name clindamycin
Route IVPB
Frequency Q6H
Home
Hospital
or
Both
Concentration
Route G-tube
Frequency Q8H
Home
Hospital
or
Both
Concentration 100mg/50mL
Route IVPB
Frequency
Home
Hospital
or
Both
Concentration 1,000mg/100mL
Route IVPB
Frequency BID
Home
Hospital
or
Both
Concentration
Dosage Amount 40 mg
Route G-tube
Frequency Daily
Home
Hospital
or
Both
Concentration
Dosage Amount 50 mg
Route G-tube
Frequency BID
Home
Hospital
or
Both
Indication Hypertension
Side effects-fatigue, dizziness, mental status changes, blurred vision, bronchospasm, wheezing, hypotension, constipation, nausea, hyperglycemia, hypoglycemia,
back pain, joint pain
Adverse effects- arrhythmias, bradycardia, HF, pulmonary edema, rashes
Nursing considerations- monitor BP and pulse before and during therapy, assess for hypotension, monitor I&O ratios and daily weight, assess for evidence of
fluid overload, assess for rash, instruct patient not to abruptly stop taking this medication because it can cause rebound tachycardia, life-threatening
arrhythmias or myocardial ischemia, take apical pulse before administering if <50 bpm, hold medication, administer with meals or directly after eating
Name polyethylene glycol 3350 (Miralax)
Concentration
Dosage Amount 17 GM
Route G-tube
Frequency Daily
Home
Hospital
or
Both
10 PERTINENT LAB VALUES AND DIAGNOSTIC TEST RESULTS (include pertinent normals as well
as abnormals, include rationale and analysis. List dates with all labs and diagnostic tests):
Lab
WBC
Dates
8.7
8.3
(3/28/14)
(3/31/14)
Hgb
10.0 L
10.6 L
(3/28/14)
(3/31/14)
Hct
30.7 L
31.9 L
(3/28/14)
(3/31/14)
Platelet count
274
252
(3/28/14)
(3/31/14)
Trend
Upon admit, the patients
WBC were in the high
range indicating the
presence of an infection.
However, they have
dropped down into the
normal range after
receiving antibiotic
therapy.
Upon admit, patients
Hgb level was in the low
range, which was
expected due to her
condition. It is now
slowly trending up. This
value will be closely
monitored because a Hgb
level of less than 7 would
require a blood
transfusion and anemia
has been associated with
poorer outcomes
following hemorrhagic
strokes.
Upon admit, the patients
Hct level was in the low
range and has been
steadily increasing over
the course of treatment.
Analysis
Number of infection
fighting cells. High WBC
indicates the presence of
an infection or
inflammation.
*I forgot to change the search criteria to admission-present before printing the labs at the hospital, which
is why not all values are present above; however, I did review the trend of the all the lab values while at the
hospital and based the explanation in the trend section on this knowledge.
3/11/14- CT scan revealed intracerebral hemorrhage and hemorrhagic stroke of basal ganglia
3/13/14- Electroencephalogram results- Abnormal. The background is slowed and disorganized. There is
evidence of a focus of left temporal paroxysmal activity. Electroencephalogram suggests patient at risk for
seizure.
3/23/14- A swallow study confirmed the diagnosis of neurogenic dysphagia. A G-tube was inserted on
3/24/14.
side of body.
5. Unilateral neglect r/t effects of disturbed perceptual abilities aeb failure to move limbs on affected side.
6. Risk for impaired skin integrity r/t alteration in sensation and immobility.
15 CARE PLAN
Nursing Diagnosis: Risk for aspiration r/t impaired swallowing.
Patient Goals/Outcomes Nursing Interventions to
Rationale for
Achieve Goal
Interventions
Provide References
Patient will maintain patent -Monitor respiratory rate, -Signs of aspiration
airway and clear lung
depth, and effort Q4
should be detected as
sounds during this shift.
hours and PRN with any
soon as possible to
changes. Note any signs
prevent further aspiration
Patient will remain free
of aspiration such as
and to initiate treatment
from aspiration for the
dyspnea, cough, cyanosis, that can be lifesaving
duration of this
wheezing, hoarsness, or
(Ackley and Ladwig,
hospitalization.
fever. If new onset of
2014).
symptoms, perform oral
suctioning and notify
provider immediately.
- Patients respiratory
rate, depth, and ease of
respiration were closely
monitored and remained
within normal limits. No
signs of aspiration noted.
-Auscultation of lung
sounds was shown to be
specific in identifying
clients at risk for
aspiration (Ackley and
Ladwig, 2014).
-Maintaining this
positioning can help
decrease aspiration
pneumonia (Ackley and
Ladwig, 2014).
-Patient showed no
symptoms of nausea or
vomiting.
-Decreased or absent
-Patients bowel sounds
bowel sounds can indicate were auscultated Q4
an ileus with possible
hours and noted and
Evaluation of Goal on
Day care is Provided
hyperactive.
documented as
hypoactive.
-Abdominal distention o
rigidity can be associated
with paralytic or
mechanical obstruction
and an increased
likelihood of vomiting
and aspiration (Ackley
and Ladwig, 2014).
2 DISCHARGE PLANNING: (put a * in front of any pt education in above care plan that you would include
for discharge teaching)
Consider the following needs:
SS Consult
Dietary Consult
PT/ OT
Pastoral Care
Durable Medical Needs
F/U appts
Med Instruction/Prescription
are any of the patients medications available at a discount pharmacy? Yes No
Rehab/ HH
Palliative Care
References
Ackley, Betty J. and Ladwig, Gail B. (2014). Nursing diagnosis handbook. Maryland Heights, MO: Elsevier
Huether, Sue E. and McCance, Kathryn L. (2012). Understanding pathophysiology. St. Louis, MO: Elsevier.
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