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  • Pathophysiology HPN Cva

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    tresdos09
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    This document discusses the pathophysiology of a stroke in a 78-year-old patient. It outlines how increased systolic blood pressure and systemic vascular resistance can raise the risk of cerebral vascular accident by increasing the afterload on the heart and reducing blood flow to organs. It then describes the renin-angiotensin system pathway that is activated in response to decreased renal perfusion and blood pressure, ultimately leading to vasoconstriction and increased blood pressure and risk of stroke. Disruption of blood supply to the brain from trauma such as a fall can then cause neurologic deficits depending on the compliance of arteries in the brain.

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    PATHOPHYSIOLOGY HPN CVA

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    This document discusses the pathophysiology of a stroke in a 78-year-old patient. It outlines how increased systolic blood pressure and systemic vascular resistance can raise the risk of cerebral vascular accident by increasing the afterload on the heart and reducing blood flow to organs. It then describes the renin-angiotensin system pathway that is activated in response to decreased renal perfusion and blood pressure, ultimately leading to vasoconstriction and increased blood pressure and risk of stroke. Disruption of blood supply to the brain from trauma such as a fall can then cause neurologic deficits depending on the compliance of arteries in the brain.

    Copyright:

    Attribution Non-Commercial (BY-NC)
    Download as DOCX, PDF, TXT or read online from Scribd
    Flag for inappropriate content
    Download as docx, pdf, or txt
    122 views1 page

    Pathophysiology HPN Cva

    Uploaded by

    tresdos09
    This document discusses the pathophysiology of a stroke in a 78-year-old patient. It outlines how increased systolic blood pressure and systemic vascular resistance can raise the risk of cerebral vascular accident by increasing the afterload on the heart and reducing blood flow to organs. It then describes the renin-angiotensin system pathway that is activated in response to decreased renal perfusion and blood pressure, ultimately leading to vasoconstriction and increased blood pressure and risk of stroke. Disruption of blood supply to the brain from trauma such as a fall can then cause neurologic deficits depending on the compliance of arteries in the brain.

    Copyright:

    Attribution Non-Commercial (BY-NC)
    Download as DOCX, PDF, TXT or read online from Scribd
    Flag for inappropriate content
    Download as docx, pdf, or txt
    of 1

    PATHOPHYSIOLOGY

    Age (78 y/o)

    [Systolic HPN the risk

    Of CVA (stroke)]

    Systemic Vascular Resistance

    Afterload

    Blood Flow to Organs

    Renal Perfusion Blood Pressure Beta-receptor activation

    Hyponatremia Stimulation of Juxtaglomerular cell Hypovolemia

    Renin

    Angiotensinogen Angiotensin I

    ACE

    Arteriolar Vasoconstriction Angiotensin II

    TRAUMA (e.g. FALL)

    Peripheral Resistance Aldosterone

    DISRUPTION OF the BLOOD SUPPLY

    To the Brain Na Reabsorption

    BLOOD PRESSURE

    Compliance of large H2O Reabsorption

    arteries e.g. MCA/ICA

    Plasma Volume (ECF)

    Neurologic Deficit

    Aphasia Loss of Consciousness Body Weakness ICP

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