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  • Preeclampsia Pathophysio

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    Stephen S. Padayhag
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    Preeclampsia is a disease of pregnancy characterized by hypertension and proteinuria after 20 weeks of gestation. It is caused by placental ischemia which leads to vasospasm and decreased blood flow to organs. This results in signs and symptoms such as edema, headaches, visual disturbances, and elevated blood pressure. Complications for both the mother and fetus can be serious if left untreated.

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    Preeclampsia is a disease of pregnancy characterized by hypertension and proteinuria after 20 weeks of gestation. It is caused by placental ischemia which leads to vasospasm and decreased blood flow to organs. This results in signs and symptoms such as edema, headaches, visual disturbances, and elevated blood pressure. Complications for both the mother and fetus can be serious if left untreated.

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    89 views5 pages

    Preeclampsia Pathophysio

    Uploaded by

    Stephen S. Padayhag
    Preeclampsia is a disease of pregnancy characterized by hypertension and proteinuria after 20 weeks of gestation. It is caused by placental ischemia which leads to vasospasm and decreased blood flow to organs. This results in signs and symptoms such as edema, headaches, visual disturbances, and elevated blood pressure. Complications for both the mother and fetus can be serious if left untreated.

    Copyright:

    Attribution Non-Commercial (BY-NC)
    Download as DOCX, PDF, TXT or read online from Scribd
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    of 5

    PREECLAMPSIA --- A disease condition encountered 20th and above weeks of

    pregnancy characterized by increasing hypertension, proteinuria and significant edema.

    Predisposing Factors: Precipitating Factor:


    -Primigravida -pregnancy
    -age (17 years old))
    -family history of hypertension

    Vasospasm

    Decrease Blood Supply and O2


    perfusion to vital organs
    PLACENTA LIVER KIDNEY

    Tissue Ischemia
    Tissue Ischemia Beta- Decrease Renal
    receptor Perfusion

    Reduce fetal nutrients activation


    and O2 supply Vascular stasis

    Juxtaglomerular Glomerular
    Epigastric cells Degenerator
    Pain Angioten-
    Intrauterine Angiotensinogen Renin sin 1 Increase
    growth Glomerular
    Abruptio Permeability
    retardation
    placenta ACE
    Fetal weight = 2,500
    grams
    Arteriolar
    Vasoconstriction
    Angiotensin 2 Proteinuria

    Increase Peripheral Adrenal Cortex Chemical


    Resistance Stimulation Properties
    Protein =
    BP = Increase Blood Increase +3
    160/100 Pressure Aldosterone

    Increase Na
    reabsorption

    Increase water
    reabsorption

    PREECLAMPSIA
    Diffusion of fluid from
    the blood stream into
    interstitial space

    Edema Oliguria

    Hematocrit
    3+ edema in Generalized
    40%lesser than the lower H2O Retention
    normal range extremities
    HCT=22.9
    Pulmonary Cerebral
    edema edema

    Hypoxia
    Cyanosis
    Convulsion
    LEGEND:

    ETIOLOGY

    DISEASE PROCESS

    SIGNS AND SYMPTOMS

    DIAGNOSTIC RESULT

    COMPLICATIONS

    DISEASE PROCESS

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