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    PATHOPHYSIOLOGY OF LIVER CIRRHOSIS (CLIENT CENTERED) Alcohol intake Hepatocyte damage Liver inflammation Alterations in blood and lymph flow Liver necrosis ADH and aldosterone detoxification so increased levels metabolism of protein and carbohydrate fat metabolism bilirubin metabolism, And/or biliary tree damage or obstruction Fluid shift to extravascular compartment Malnutrition Wt. Loss plasma proteins bile in the gastrointestina l tract and urobilinogen Conjugated and unconjugated

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    PATHOPHYSIOLOGY OF LIVER CIRRHOSIS (CLIENT CENTERED) Alcohol intake Hepatocyte damage Liver inflammation Alterations in blood and lymph flow Liver necrosis ADH and aldosterone detoxification so increased levels metabolism of protein and carbohydrate fat metabolism bilirubin metabolism, And/or biliary tree damage or obstruction Fluid shift to extravascular compartment Malnutrition Wt. Loss plasma proteins bile in the gastrointestina l tract and urobilinogen Conjugated and unconjugated

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    25 views1 page

    (Client Centered) Pa Tho Physiology

    Uploaded by

    jake90210
    PATHOPHYSIOLOGY OF LIVER CIRRHOSIS (CLIENT CENTERED) Alcohol intake Hepatocyte damage Liver inflammation Alterations in blood and lymph flow Liver necrosis ADH and aldosterone detoxification so increased levels metabolism of protein and carbohydrate fat metabolism bilirubin metabolism, And/or biliary tree damage or obstruction Fluid shift to extravascular compartment Malnutrition Wt. Loss plasma proteins bile in the gastrointestina l tract and urobilinogen Conjugated and unconjugated

    Copyright:

    Attribution Non-Commercial (BY-NC)
    Download as DOCX, PDF, TXT or read online from Scribd
    Flag for inappropriate content
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    PATHOPHYSIOLOGY OF LIVER CIRRHOSIS (CLIENT CENTERED)

    ↑ Alcohol
    intake

    Hepatocyte damage

    Liver inflammation

    Alterations in blood
    and lymph flow

    Liver necrosis

    ↓bilirubin
    ↓ ADH and ↓ metabolism metabolism,
    aldosterone of protein and And/or biliary
    detoxification carbohydrate tree damage
    so increased ↓ fat or obstruction
    levels metabolism

    Fluid shift to
    extravascular Malnutrition ↓ plasma ↓ bile in the Conjugated and
    compartment ↓Wt. Loss proteins gastrointestin unconjugated
    al tract and hyperbilirubine
    mia
    ↑urobilinoge
    Dyspnea n
    Edema
    Jaundice
    Dark urine

    ↑serum
    ammonia
    Liver fibrosis and scarring Liver failure

    Liver flap
    Inability to
    Portal hypertension metabolize
    ammonia to
    urea
    Foul breath Pyuria
    Splenomegaly
    Hepatic
    Anemia encephalopathy
    Thrombocytop
    enia
    Agitation Confusion to
    Leukopenia Death
    hepatic coma

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