This document discusses the pathophysiology and complications of acute pancreatitis. It involves damage to pancreatic cells leading to inflammation, edema, and obstruction of pancreatic enzymes which become activated inside the pancreas causing autodigestion, fatty necrosis, and complications such as pancreatic necrosis, septic shock, multiple organ failure and hemorrhage.
This document discusses the pathophysiology and complications of acute pancreatitis. It involves damage to pancreatic cells leading to inflammation, edema, and obstruction of pancreatic enzymes which become activated inside the pancreas causing autodigestion, fatty necrosis, and complications such as pancreatic necrosis, septic shock, multiple organ failure and hemorrhage.
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Attribution Non-Commercial (BY-NC)
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This document discusses the pathophysiology and complications of acute pancreatitis. It involves damage to pancreatic cells leading to inflammation, edema, and obstruction of pancreatic enzymes which become activated inside the pancreas causing autodigestion, fatty necrosis, and complications such as pancreatic necrosis, septic shock, multiple organ failure and hemorrhage.
Copyright:
Attribution Non-Commercial (BY-NC)
Available Formats
Download as DOCX, PDF, TXT or read online from Scribd