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  • Pathophysiology of Acute Gastroenteritis: Perforati-Ng

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    Bryan Voltaire Santos Lannu
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    The document summarizes the pathophysiology of acute gastroenteritis. It lists common etiological factors like E. coli and Salmonella and predisposing factors like age, poor sanitation, and diet. It then describes the process by which ingestion of contaminated foods allows pathogens to circulate in the body, consume nutrients, produce toxins and gas, and cause pain, bleeding, fever, dehydration, and other symptoms.

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    Pathophisio of Age

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    The document summarizes the pathophysiology of acute gastroenteritis. It lists common etiological factors like E. coli and Salmonella and predisposing factors like age, poor sanitation, and diet. It then describes the process by which ingestion of contaminated foods allows pathogens to circulate in the body, consume nutrients, produce toxins and gas, and cause pain, bleeding, fever, dehydration, and other symptoms.

    Copyright:

    Attribution Non-Commercial (BY-NC)
    Download as DOC, PDF, TXT or read online from Scribd
    Flag for inappropriate content
    Download as doc, pdf, or txt
    214 views1 page

    Pathophysiology of Acute Gastroenteritis: Perforati-Ng

    Uploaded by

    Bryan Voltaire Santos Lannu
    The document summarizes the pathophysiology of acute gastroenteritis. It lists common etiological factors like E. coli and Salmonella and predisposing factors like age, poor sanitation, and diet. It then describes the process by which ingestion of contaminated foods allows pathogens to circulate in the body, consume nutrients, produce toxins and gas, and cause pain, bleeding, fever, dehydration, and other symptoms.

    Copyright:

    Attribution Non-Commercial (BY-NC)
    Download as DOC, PDF, TXT or read online from Scribd
    Flag for inappropriate content
    Download as doc, pdf, or txt
    of 1

    PATHOPHYSIOLOGY OF ACUTE GASTROENTERITIS ETIOLOGICAL FACTOR: Escherichia Coli Salmonella Species Staphylococci PREDISPOSING FACTOR: Age (Infant, Adult)

    Adult) Poor Sanitation Poor Hygiene Diet

    Ingestion of contaminated foods Entry of large amount of pathogen in GIT Exposure of pathogens to gastric acid

    Circulation of Toxins in the body Recognition of toxins in the body

    Intraluminal pressure of intestines


    Perforati-ng

    Activity organism to digestive food Production of gas as product

    Consumption of nutrients by pathogens Cellular deprevation of nutrients

    of intestinal lining

    Compres sion of nerve endings

    Rapture of capillaries

    Pain

    Excess gas formation

    Cellular starvation

    Bleeding

    Melena

    Flatulence

    Hunger

    Weakness

    Histamine release

    Prostaglandin

    Release of pyrogen Irritation of hypothalamus Alteration of thermoregulation Fever

    Peristaltic movement Frequency of defacation Water absorption

    Watery stool

    Fluid loss Dehydration

    urine output

    Poor skin turgor

    Sunken eyeball

    thrist

    Dry mucous membrane

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