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    Lohrhen Lheighh Cahreeniyow
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    Mild preeclampsia is caused by reduced uteroplacental blood flow due to unknown etiologic agents, which leads to reduced perfusion of the renal glomeruli and production of thromboplastin, resulting in proteinuria. This triggers the release of uterine renin and a cascade that causes vasoconstriction, hypertension, fluid retention, and edema through angiotensin conversion and aldosterone production.

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    Mild preeclampsia is caused by reduced uteroplacental blood flow due to unknown etiologic agents, which leads to reduced perfusion of the renal glomeruli and production of thromboplastin, resulting in proteinuria. This triggers the release of uterine renin and a cascade that causes vasoconstriction, hypertension, fluid retention, and edema through angiotensin conversion and aldosterone production.

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    12 views1 page

    Pa Tho Physiology

    Uploaded by

    Lohrhen Lheighh Cahreeniyow
    Mild preeclampsia is caused by reduced uteroplacental blood flow due to unknown etiologic agents, which leads to reduced perfusion of the renal glomeruli and production of thromboplastin, resulting in proteinuria. This triggers the release of uterine renin and a cascade that causes vasoconstriction, hypertension, fluid retention, and edema through angiotensin conversion and aldosterone production.

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    Attribution Non-Commercial (BY-NC)
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    PATHOPHYSIOLOGY OF MILD- PREECLAMPSIA

    ` PREDISPOSING FACTORS Age Race PRECIPITATING FACTORS Lifestyle Hydramnios Multiple gestation Grand multiparity Secondary illnesses

    ETIOLOGIC AGENT (unknown)

    REDUCED UTEROPLACENTAL BLOOD FLOW

    REDUCED PERFUSION

    THROMBOPLASTIN PRODUCTION RENAL GLOMERULI AFFECTED PROTEINURIA

    RELEASE OF UTERINE RENIN

    ANGIOTENSIN I CONVERSION TO ANGIOTENSIN II ALDOSTERONE PRODUCTION SODIUM REABSORPTION

    VASOCONSTRICTION

    RELEASE OFADRENAL HORMONES HYPERTENSION

    FLUID RETENTION

    EDEMA

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