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ADH and RAAS System
ADH and RAAS System
7/5/12
If thesalinityoverthe range ofnormaland lesswaterin thebody,the bloodosmoticpressurewill increase, osmoreceptorsin thehypothalamusandpituitary glandwillbestimulatedmore activelystimulatedto secreteantidiuretichormoneADHistoincrease thepermeability of therenaltubulesto water, adrenalgland(hormonealdosterone)willnotbe stimulated,then morewateris absorbedandlesssodiumionsandcalciumionsto be reabsorbedinto thebody, bloodosmoticpressurewill drop, this processwill be repeatedso that theosmoticpressureof bloodinthenormal
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Decrease in blood volume causes a decrease in blood pressure in the glomerulus Juxtaglomerular cell stimulation by the sympathetic nervous decrease in osmotic concentration of tubular fluid in the macula densa. 7/5/12 (decreased levels of sodium)
Process can stimulate cells in the kidney jukstaglomerular to release the enzyme renin, Renin then it will circulate throughout the body which will then meet with the angiotensinogen produced in the liver to release the enzyme angiotensin I. Angiotensin I into angiotensin II will 7/5/12 change after Angiotensin Converting
increased arterial pressure reduce the excretion of salt and extra water to increase the volume of cells stimulates the secretion of aldosterone in the adrenal gland which in turn increases salt and water reabsorption by the renal 7/5/12 tubules
Excretion on animal
Excretion tool Animal Picture Malpighian tubules (uric acid) Insecta
Flame cell
Flat worm
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Excretion tool
animal
Picture
Gills
Marine fishes
Gills
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Excretion tool
Animal
Picture
Kidney
Kidney
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