JVP - WL Gan

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Waveform Palpation

Jugular venous pressure

Respiratory effect Patient position Compression

Hepatojugular reflux

Double waveform Increase Not palpable Occludable

The University of Manchester | W.L. Gan | 2012 No effect No effect No effect Palpable

Decrease with inspiration

Venous pulsation

Increase with patient lying supine

Single waveform Non occludable

Arterial pulsation

Waveform a wave x descent c wave v wave y descent 1. Heart failure

Atrial contraction

Relaxation of atrium and the tricuspid valve moves downward Passive filling of right atrium against closed tricuspid valve Filling of right ventricle after tricuspid opening 1. Pericardial effusion 2. Cardiac tamponade Atrial fibrillation Kussmaul's sign

Ventricular contraction and resulting bulging of tricuspid into the right atrium JVP rises with inspiration, drops with expiration 3. Constrictive pericarditis

2. Fluid overload

Causes of raised JVP

3. Superior vena caval syndrome 4. Cardiac tamponade Absence a wave Large a wave Cannon a wave Slow y descent 5. Constrictive pericarditis

Waveform abnormalities

Prominent v wave

cv waves, steep y descent Steep x and y descents

Pulmonary hypertension, tricuspid stenosis Tricuspid regurgitation Tricuspid stenosis Constrictive pericarditis

Atrial flutter, complete heart block, ventricular tachycardia

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