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CPB 25
CPB 25
CPB 25
Untreated Type II Diabetes 1. Hyperglycaemia increased hepatic gluconeogenesis, decreased peripheral glucose use **Ketosis is minimal/absent in type II because of low levels of insulin ketogenesis 2. Hypertriglyceridaemia The amount of Fas being released swamps the capacity of oxidation and ketogenesis. Excess FAs converted to TGs which are packaged and secreted as VLDL by the liver.