The document summarizes the pathophysiology of congestive heart failure (CHF). It lists common causes like myocardial infarction and hypertension. It then describes how a large infarct can activate baroreflexes, increasing blood return to the damaged heart. This leads to constriction of downstream arteries and blood accumulating in the heart, overstretching cardiac muscle cells. Eventually, this decreases heart contractility and causes ventricular hypertrophy, resulting in ineffective pumping and congestion - the defining features of CHF.
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"Acute Coronary Syndrome Non ST Elevation Myocardial Infarction, Hypertensive Cardiovascular Disease, Diabetes Mellitus Type 2, and Community Acquired Pneumonia" Client Centered Pathophysiology
The document summarizes the pathophysiology of congestive heart failure (CHF). It lists common causes like myocardial infarction and hypertension. It then describes how a large infarct can activate baroreflexes, increasing blood return to the damaged heart. This leads to constriction of downstream arteries and blood accumulating in the heart, overstretching cardiac muscle cells. Eventually, this decreases heart contractility and causes ventricular hypertrophy, resulting in ineffective pumping and congestion - the defining features of CHF.
The document summarizes the pathophysiology of congestive heart failure (CHF). It lists common causes like myocardial infarction and hypertension. It then describes how a large infarct can activate baroreflexes, increasing blood return to the damaged heart. This leads to constriction of downstream arteries and blood accumulating in the heart, overstretching cardiac muscle cells. Eventually, this decreases heart contractility and causes ventricular hypertrophy, resulting in ineffective pumping and congestion - the defining features of CHF.
Copyright:
Attribution Non-Commercial (BY-NC)
Available Formats
Download as DOC, PDF, TXT or read online from Scribd
The document summarizes the pathophysiology of congestive heart failure (CHF). It lists common causes like myocardial infarction and hypertension. It then describes how a large infarct can activate baroreflexes, increasing blood return to the damaged heart. This leads to constriction of downstream arteries and blood accumulating in the heart, overstretching cardiac muscle cells. Eventually, this decreases heart contractility and causes ventricular hypertrophy, resulting in ineffective pumping and congestion - the defining features of CHF.
Copyright:
Attribution Non-Commercial (BY-NC)
Available Formats
Download as DOC, PDF, TXT or read online from Scribd
"Acute Coronary Syndrome Non ST Elevation Myocardial Infarction, Hypertensive Cardiovascular Disease, Diabetes Mellitus Type 2, and Community Acquired Pneumonia" Client Centered Pathophysiology