This document outlines the pathophysiology and risk factors for abdominal aortic aneurysm (AAA). It describes the process as beginning with predisposing factors like age, gender, and smoking that cause damage to the aortic wall. Over time, this results in thinning of the wall, loss of elastic tissue, and accumulation of lipids and inflammatory cells that form plaques. The plaques enlarge the vessel and put it at risk of rupture, which can lead to hemorrhage and death. Complications of AAA include embolism, hypertension, renal failure, and shock due to blood loss from rupture. Signs and symptoms may include back or abdominal pain, pulsations, and hypotension. Diagnostic tests involve ultrasound, CT
This document outlines the pathophysiology and risk factors for abdominal aortic aneurysm (AAA). It describes the process as beginning with predisposing factors like age, gender, and smoking that cause damage to the aortic wall. Over time, this results in thinning of the wall, loss of elastic tissue, and accumulation of lipids and inflammatory cells that form plaques. The plaques enlarge the vessel and put it at risk of rupture, which can lead to hemorrhage and death. Complications of AAA include embolism, hypertension, renal failure, and shock due to blood loss from rupture. Signs and symptoms may include back or abdominal pain, pulsations, and hypotension. Diagnostic tests involve ultrasound, CT
This document outlines the pathophysiology and risk factors for abdominal aortic aneurysm (AAA). It describes the process as beginning with predisposing factors like age, gender, and smoking that cause damage to the aortic wall. Over time, this results in thinning of the wall, loss of elastic tissue, and accumulation of lipids and inflammatory cells that form plaques. The plaques enlarge the vessel and put it at risk of rupture, which can lead to hemorrhage and death. Complications of AAA include embolism, hypertension, renal failure, and shock due to blood loss from rupture. Signs and symptoms may include back or abdominal pain, pulsations, and hypotension. Diagnostic tests involve ultrasound, CT
- Gender: male - Genetics Precipitating factors: - Cigarette smoking - Atherosclerosis - Trauma - Hypertension - Obesity,diet (high cholesterol and fat), lack High wall tension Hypermeable vessels allow establishment of chemotactic gradients for endothelial cell migration Elimination of elastin from the media Increased expression and activity of matrix metalloproteinases Accumulation of lipids in foam cells, extracellular free cholesterol crystals, calcifications, thrombosis, and ulcerations Chemotactic gradient also allows recruitment of inflammatory cells Atherosclerosis reduced amount of vasa vasorum Medial Neovascularization Degradation of extracellular matrix in media allows endothelial cell migration Degradation of elastin and collagen Inflammatory Infiltration production of proteases Production of cytotoxic mediators Decrease in smooth muscle cells density Abdominal Aortic Aneurysm Wall thinning and focal dilation Plaque formation Ulceration of the wall Damage of endothelial lining Development of intraluminal thrombus Narrows the vessel Hypertension Rupture of aneurysm Hemorrhage Death - Feeling of heart beat on the abdomen when lying - abdominal/back pain - Duplex ultrasonography - X-ray examination - Echocardiography - CT scan - MRI Altered comfort: Acute pain related to nerve compression Decreased cardiac output related to increased systemic vascular resistance Anxiety related to threat to health status Swept along with the blood and becomes an emboli Arterial embolism Emboli travels to the brain Emboli travels to the heart Stroke Myocardial infarction Death Emboli travels to the renal artery Complete blockage of blood flow Kidney failure Emboli lodges in the interosseous or digital artery Ineffective cerebral tissue perfusion related to occlusion in the carotid artery - Altered level of consciousness - restlessness - Changes in motor/sensory responses Excess Fluid Volume related to compromised regulatory mechanism - Intake greater than output - oliguria - changes in urine specific gravity - Decreased Hb/hematocrit (Hct) - altered electrolytes - Urinalysis - CBC - GCS Altered comfort: Acute pain related to coronary artery occlusion - chest pain - Restlessness - Facial grimacing - Fatigue - Peripheral cyanosis - Weak pulse - Pulse Risk for super infection related to presence of bacteria on the bladder Legends: Nursing dx------------------------ Complications------------------- Signs and symptoms----------- Medications---------------------- Surgical interventions--------- Disease condition--------------- Diagnostic tests------------------
Hypovolemic shock - tachycardia - hypotension - pale clammy skin - decreased urine output -altered sensorium - abdominal tenderness on palpation - Diuretics - beta-blockers - ACE inhibitors - Angiotensin II receptor antagonists - Calcium channel blockers - Open aneurysm repair - Endovascular grafting Risk for fluid volume deficit related to hemorrhage Deficient knowledge (preoperative and postoperative care) related to newly identified need for aortic surgery cyanosis mottling of toes Ineffective peripheral tissue perfusion related to occlusion the digital artery Risk for super infection related to presence of bacteria on the bladder
"Acute Coronary Syndrome Non ST Elevation Myocardial Infarction, Hypertensive Cardiovascular Disease, Diabetes Mellitus Type 2, and Community Acquired Pneumonia" Intro