ACLS/ PALS

Questions
1. Why is
excessive
ventilation
detriment
al to the
pt?
2. How to
identify
ischemia?

Answers
Excessive ventilation will cause an increase in intrathroasic pressure
leading to decrease venous return to the heart and decrease cardiac
output. It can also cause cerebral vasoconstriction, reducing blood
flow to the brain due to hyperventilation.
-

3. VF
Treatment

4.

5.

6.

7.

8.

9.

d.
How would
you give
drugs
through
the ET
tube?
If Amio is
not available,
what
alternative
drug can you
give for Vf?
Which
rhythms are
considered to
be
bradycardia?
Which H/B
do we not
soly rely on
atropine?
At which
point would
you start
pacing a pt?
If Atropine
doesnt work,
what other
drugs are

Uncomfortable pressure, fullness, squeezing or pain in the

center of the chest that last for serveral minutes.
Chest pain spreading to the shoulder, neck, one or both arms, or
jaw, shoulder blades.
Chest discomfort with light headeness, dizziness, fainting,
sweating
a. CPR -> Shock -> 1mg Epi q 3-5 min -> Shock -> 300 mg
initial, 150 mg subsequent Amiodarone.
b. Advanced Airway -> 1 breath every 6 second
i. ETCo2, in VF < 10 mmhg (Improve CPR)
c. Rosc-> Maintain ETCo2 >40 mmhg

Increase the dosage to 2-2.5 times then dilute the dose in 5-10 mL of
NS then inject into the tube.

Lidocaine @ 1 to 1.5 mg/kg. Rp of 0.5 to 0.75 mg/kg over 5 to 10 mins.

Sinus brady, 1st degree H/B, 2nd degree type I & II, and 3rd degree H/B

2nd type II and 3rd degree. Go with pacing instead

When the pt is symptomatic showing AMS, hypoperfusion, chest pain. SET THE
PACING AT 60BPM.
Epi drip at 2-10 mcg/min
Dopamine drip at 5-20 mcg/kg/min.

there for
bradycardia?
10. What is the
post rosc
treatment?
11. What is the
drug
treatment for
post-rosc
hypotension?
12. What is the
temperature
you wish to
maintain in
post rosc?

Maintain o2 sat > 94%, avoid hyperventilation, reach a goal of 35-45 mmhg.
Hypovolemia- 1-2 L
Epi- 0.1-0.5 mcg/kg
Dopamine 5-10 mcg/kg
32 C/89.6 F.