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A&E - Urinary / DM/ Toxicology-Mergencies Traumatized Uts
A&E - Urinary / DM/ Toxicology-Mergencies Traumatized Uts
A&E - Urinary / DM/ Toxicology-Mergencies Traumatized Uts
ToxicologyEmergencies
TRAUMATIZED UTS
MOST COMMON
CASES:
Bleeding urinary
meatus
Testicular torsion
Cystitis
Hernia
Renal colic
Testicular Torsion
( twisting of the
supporting cord of
the testis
tenderness cannot
relieved by scrotal
elevation
Types of Hernia
Strangulated Hernia
2. Incarcerate Hernia
Hernia is trapped
outside
peritoneal cavity
Cystitis
Recommendations:
drink 8 to 10 glasses
of fluids /day
Women should wipe
intercourse
Avoid vaginal
deodorants and
bubble bath.
DKA vs
HHNKA
Diabetes Mellitus
Types of DM
Type I (DKA)
juvenile onset DM
insulin dependent DM
Type II (HHNK)
adult onset DM
non-insulin dependent
DM
Endocrine
emergencies
CAUSE OF DKA
initial presentation of
undiagnosed Type I DM
missed or reduced insulin
doses
illness (infection and stress
hormone excess)
stress
pregnancy
Pathophysiology
produce insulin
hyperglycemia and
hyperosmolar state
hyperosmolarity causes
leading to decreased
blood pH and bicarbonate
concentration causing
ketoacidosis
SIGNS AND
SYMPTOMS
dehydration
ketosis
metabolic acidosis
ketonuria
weakness, anorexia,
vomiting, abdominal
pain
altered mental status
tachycardia, orthostatic
breath
blood sugar,
potassium, sodium, and
other electrolytes.
Ketone levels and kidney
Complications
Hypokalemia and
often, potassium
depletion
Cerebral edema
Hyperglycemia
Ketoacidemia
Fluid and electrolyte
depletion
ESRD
Pulmonary edema
Myocardial Infarction
Treatment
Medical Management
Rapid fluid replacement
bicarbonate
rapid acting insulin
correction of electrolyte
imbalance
correction of underlying
Treatment
Nursing
Responsibilities
monitoring of serum
glucose monitoring
monitoring
monitor cardiac,
medications
regulation
eating schedules
diet
exercise
rest
HYPEROSMOLAR
HYPERGLYCEMIC
NON-KETOTIC
ACIDOSIS
(HHNK)
HHNK
Hyperosmolar
Hyperglycemic
nonketotic state is
one of two serious
metabolic
derangements
that
occurs
in
patients
with diabetes mellitus
and can be a lifethreatening
emergency.
Risk Factors
Usually triggered by
or missed dose of
insulin or antidiabetic
agents
Certain medications
(e.g., corticosteroids,
diuretics, beta
blockers)
Stress
Drug abuse or
excessive
consumption of
alcohol
Overeating
(polyphagia)
Inactivity
Chronic illness
Recent surgery
thirst)
Polyuria (increased
urination)
Warm, dry skin that
100.4 degrees
Fahrenheit, 38
degrees Celsius)
Low systolic blood
Confusion
Vision loss
Hallucinations
Weakness on one side
of the body
Impaired speech
Treatment
The treatment of
HHNS centers on
correcting the
patients dehydration,
hyperglycemia and
electrolyte
imbalances.
2. The patient may
receive intravenous
fluid to correct the
dehydration and
restore electrolytes.
3. Small doses of
intravenous insulin
may be given to treat
the hyperglycemia.
4. Treatment of
precipitating causes
and complications
Prevention
1. Careful monitoring
of glucose levels
2. Ensuring adequate
hydration in patients
at risk of
3. Screening program.
Identify patients at
risk for HHNS.
4. Avoiding excessive
amounts of alcohol.
DKA vs
HHNKA