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    Pathway DM

    Uploaded by

    Rifa'atul Mahmudah
    Type I diabetes (DM Type I/IDDM) is an autoimmune disease where the immune system destroys the pancreatic beta cells, resulting in insulin deficiency. It accounts for about 15% of diabetes cases and is often diagnosed in children and young adults. Type II diabetes (DM Type II/NIDDM) results from the body's ineffective use of insulin and accounts for about 85% of diabetes cases. Genetic factors and lifestyle contribute to insulin resistance. It is most common in those over age 40. Both types of diabetes can lead to serious complications if not managed properly, including damage to blood vessels, nerves, eyes, kidneys, heart, and other organs. Complications become more likely with poorer blood

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    Pathway DM

    Uploaded by

    Rifa'atul Mahmudah
    21 views3 pages
    Type I diabetes (DM Type I/IDDM) is an autoimmune disease where the immune system destroys the pancreatic beta cells, resulting in insulin deficiency. It accounts for about 15% of diabetes cases and is often diagnosed in children and young adults. Type II diabetes (DM Type II/NIDDM) results from the body's ineffective use of insulin and accounts for about 85% of diabetes cases. Genetic factors and lifestyle contribute to insulin resistance. It is most common in those over age 40. Both types of diabetes can lead to serious complications if not managed properly, including damage to blood vessels, nerves, eyes, kidneys, heart, and other organs. Complications become more likely with poorer blood

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    pathway diabetes mellitus

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    Type I diabetes (DM Type I/IDDM) is an autoimmune disease where the immune system destroys the pancreatic beta cells, resulting in insulin deficiency. It accounts for about 15% of diabetes cases and is often diagnosed in children and young adults. Type II diabetes (DM Type II/NIDDM) results from the body's ineffective use of insulin and accounts for about 85% of diabetes cases. Genetic factors and lifestyle contribute to insulin resistance. It is most common in those over age 40. Both types of diabetes can lead to serious complications if not managed properly, including damage to blood vessels, nerves, eyes, kidneys, heart, and other organs. Complications become more likely with poorer blood

    Copyright:

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    21 views3 pages

    Pathway DM

    Uploaded by

    Rifa'atul Mahmudah
    Type I diabetes (DM Type I/IDDM) is an autoimmune disease where the immune system destroys the pancreatic beta cells, resulting in insulin deficiency. It accounts for about 15% of diabetes cases and is often diagnosed in children and young adults. Type II diabetes (DM Type II/NIDDM) results from the body's ineffective use of insulin and accounts for about 85% of diabetes cases. Genetic factors and lifestyle contribute to insulin resistance. It is most common in those over age 40. Both types of diabetes can lead to serious complications if not managed properly, including damage to blood vessels, nerves, eyes, kidneys, heart, and other organs. Complications become more likely with poorer blood

    Copyright:

    © All Rights Reserved
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    of 3

    DM Type I/ IDDM

    DM Type II/ NIDDM


    -

    Immunology
    Factor
    Environmen
    t Factor
    (Mumps virus,
    Rubella/ toxic)

    Genetic
    (Antigen
    HLA)

    About 85% of all


    cases
    Genetic (mutation
    gen Glucokirase
    {kr.7})
    Most common in
    over 40s in White
    population
    And over 25s in
    Black and South
    Asian Population

    Deficiency insulin lead to the use of


    glucose by cells
    Leukocytes

    Autoimmune Cell precipitate lipid in vascular

    infiltration

    Destruction of the Pancreatic

    Insulin Secretion
    (-)/

    Transport glucose
    Hyperglycaemia

    Impaired of
    Insulin resistance
    Insulin secretion
    Hyperinsulinemia
    Depressed of cell

    Insulin Secretion
    (-)/
    Lipid

    Glicogenesis

    Hypertrigliseri
    Hypercholeste

    LDL

    HDL

    Risk
    infection
    Lipolysi
    s

    Glucosuria
    Water secretion

    Kidney disease

    Polyuria

    Death

    Ketoneuria,

    Intracell

    Heart
    compensation

    Intravascular
    Osmotic of the cell

    Weight loss,
    fatigue, malaise,
    energy

    Schwann
    cell
    destroyed
    Hypoxia
    peripheral cells
    Peripheral
    Neuropathy

    Intravascul

    Intracell
    Dehydratio

    Aterosclerosi
    s

    Hyperosmolarit

    Infark Miokard

    Sorbitol,
    fruktosa

    Risk of injury

    Diabetic
    Ketoacidosis (DKA)

    Reproducti
    on Organ

    Lipolysis,
    glycogenesis

    Nutrition less
    than body needs

    osmotic
    pressure a

    Disorder balance acid


    bases

    Artery
    Coronaria

    Compensation
    protein rupture

    Progressiv
    e Damage
    glomerulu
    s

    Hyperglycaemia

    Polyfagi

    Mechanism

    BUN
    GFR

    Gluconeogenesis

    And forming body of

    Hypertensio
    n

    Glucose

    Fatty acids go to

    Sediment lipid in
    vascular

    Cell glucose

    Protein

    Carbohydra
    te

    Cerebro
    Vascular

    Hemoconsentrati
    Acidosis metabolic
    Coma
    Death

    Polydipsia

    Nausea,
    vomiting
    hypovolem
    Deficient fluid

    Microaneurism
    Impaired vision

    Motoric and
    sensory disorder
    Pain sensation

    Osmoreceptor in
    Hypothalamus

    Disorder blood
    vessels coz the flow
    onto retina

    Risk injury

    Low sensibility
    Ulcus diabeticum
    Surgery
    Disability

    Impaired of
    skin
    integrity
    Self-image
    disorder

    Atropi of
    muscle

    Death

    Decompensati
    on cordis

    Sexual
    disfunction

    Stroke
    Death

    Disorder
    sexual pattern

    Insulin Secretion
    (-)/
    Hyperglycaemia
    Provision of
    insulin , diet ++
    Glucose to the

    Hypoglycaemia

    Impaired function
    of the brain

    Activation autonom
    Hypothalamus
    Hormone

    Dizzy, blurred
    vision, mental
    acuity ,
    consciousness
    Risk injury

    Coma

    Crisis

    Palpitation,
    sweating , tremor,

    Self-image
    disorder

    Death

    Change the
    process family

    Searching
    information

    Lacking
    knowledge

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