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  • Hiperglikemia Menempel Pada Arteri Estrogen Agregasi Trombosit

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    Andrianus Atu
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    This flow diagram summarizes the pathophysiological process of cerebral vascular disease (CVD) that can lead to stroke. It shows how risk factors like oral contraceptives, alcohol, smoking, diabetes, and hypertension can increase estrogen, ethanol, nicotine levels and lead to hyperlipidemia, atherosclerosis, and vascular changes narrowing arteries. This reduced blood flow and increased viscosity can cause ischemia, hypoxia in brain tissue leading to cell death, edema, and neurological deficits affecting speech, movement, swallowing depending on the region of brain injury. Progressive injury can worsen leading to rebleeding, herniation, and death.

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    Patoflo Cvd

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    This flow diagram summarizes the pathophysiological process of cerebral vascular disease (CVD) that can lead to stroke. It shows how risk factors like oral contraceptives, alcohol, smoking, diabetes, and hypertension can increase estrogen, ethanol, nicotine levels and lead to hyperlipidemia, atherosclerosis, and vascular changes narrowing arteries. This reduced blood flow and increased viscosity can cause ischemia, hypoxia in brain tissue leading to cell death, edema, and neurological deficits affecting speech, movement, swallowing depending on the region of brain injury. Progressive injury can worsen leading to rebleeding, herniation, and death.

    Copyright:

    © All Rights Reserved
    Download as XLSX, PDF, TXT or read online from Scribd
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    9 views4 pages

    Hiperglikemia Menempel Pada Arteri Estrogen Agregasi Trombosit

    Uploaded by

    Andrianus Atu
    This flow diagram summarizes the pathophysiological process of cerebral vascular disease (CVD) that can lead to stroke. It shows how risk factors like oral contraceptives, alcohol, smoking, diabetes, and hypertension can increase estrogen, ethanol, nicotine levels and lead to hyperlipidemia, atherosclerosis, and vascular changes narrowing arteries. This reduced blood flow and increased viscosity can cause ischemia, hypoxia in brain tissue leading to cell death, edema, and neurological deficits affecting speech, movement, swallowing depending on the region of brain injury. Progressive injury can worsen leading to rebleeding, herniation, and death.

    Copyright:

    © All Rights Reserved
    Download as XLSX, PDF, TXT or read online from Scribd
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    Download as xlsx, pdf, or txt
    of 4

    PATOFLOW DIAGRAM CVD

    kontrasepsi oral

    alkohol

    perokok

    estrogen

    etanol

    nikotin

    DM

    Hiperlipidemia

    Hipertensi

    hiperglikemia

    menempel pada arteri

    viskositas
    darah

    penyempitan
    vaskuler

    Atrial Fibrilas

    curah jantung

    agregasi trombosit

    emboli

    trombus

    aliran darah ke otak < 25 ml/100gr/mnt

    ruptur aneurisma intracranial & AVM

    perdarahan subarachnoid TIK menekan jar. Otak & inj

    aterosklerosis
    tekanan dinding

    pernafasan aerob ggl di neuron


    pernafasan anaerob di mitokondria
    asam laktat

    hipoperfusi

    vaskuler

    iskemia otak

    kerusakan endotel vaskuler

    asidosis
    metabolik

    kejang

    p perfusi & vasospasme

    ke otak

    ruptur vaskuler

    perdarahan intraserebral

    adenosin trifosfat (ATP)


    lobus otak

    ggl. Pompa Na-K

    depolarisasi dinding sel


    influk air ke sel

    (penumbra)
    revitalisasi dg obat t-PA
    pemulihan stroke
    kalsium intraseluler , pelepasan glutamat

    retensi cairan
    di serebral

    ggn.gerak
    ekstremitas
    atas & bwh

    jaringan otot iskemik

    edema
    serebral

    talamus

    batang otak (pons)

    N. VII
    ggn. Memori &
    sensasi kec.penciuman
    ggn. Pengecapan &
    gerak otot wajah

    ggn.aktivitas
    kelemahan tgn & kaki
    kanan, M5

    menghancurkan membran sel


    infark serebral
    perdarahan berulang
    sel otak mati

    basal ganglia

    vasospasme
    serebral
    resistensi vaskuler
    aliran darah
    terhambat
    iskemia serebral

    somnolent
    p aliran
    N=90x/mnt
    darah ke sakit kepala
    otak
    ggn perfusi
    E3,M5,Vaphasia
    jar. Otak

    perluasan
    daerah
    cidera

    ggn reabsorpsi cairan


    serebrospinal
    hidrocephalus

    hipoxia serebral
    RR=42x/mnt

    TIK
    herniasi otak

    infark serebral

    kejang

    kematian

    ar. Otak & injuri

    tak (pons)
    N. IX & X
    gangguan
    menelan

    N. XII
    ggn. Gerak lidah

    aphasia

    ggn.komunikasi verbal

    sorpsi cairan

    V aphasia
    bicara pelo

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