This document provides a pathophysiology diagram of acute glomerulonephritis. It shows that modifiable risk factors include gender, age 5-11 years old, and family history of kidney disease. Non-modifiable factors include streptococcal and skin infections and poor hygiene. The diagram then illustrates how an antigen-antibody reaction leads to immune complex formation and entrapment in the glomerular tissue, causing destruction, inflammation, and damage to the glomerular basement membrane. This increased permeability and leakage of protein results in proteinuria and fluid shifting from blood vessels to tissues, causing edema.
This document provides a pathophysiology diagram of acute glomerulonephritis. It shows that modifiable risk factors include gender, age 5-11 years old, and family history of kidney disease. Non-modifiable factors include streptococcal and skin infections and poor hygiene. The diagram then illustrates how an antigen-antibody reaction leads to immune complex formation and entrapment in the glomerular tissue, causing destruction, inflammation, and damage to the glomerular basement membrane. This increased permeability and leakage of protein results in proteinuria and fluid shifting from blood vessels to tissues, causing edema.
This document provides a pathophysiology diagram of acute glomerulonephritis. It shows that modifiable risk factors include gender, age 5-11 years old, and family history of kidney disease. Non-modifiable factors include streptococcal and skin infections and poor hygiene. The diagram then illustrates how an antigen-antibody reaction leads to immune complex formation and entrapment in the glomerular tissue, causing destruction, inflammation, and damage to the glomerular basement membrane. This increased permeability and leakage of protein results in proteinuria and fluid shifting from blood vessels to tissues, causing edema.