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Case Study - HTN Uncontrolled
Case Study - HTN Uncontrolled
College of Nursing
Dasmariñas, Cavite
Case Study of
Hypertension
In Partial Fulfillment of the Requirement in NCM 102 – RLE
Morbidity Rate:
5th Causes of Morbidity Rate in the Philippines Hypertension
Number and Rate /100,000 Population, Philippines
CAUSE 5 Year Average (2000- 2005*
2004)
No. Rate No. Rate
5. Hypertension 314,175 400.5 382,662 448.8
II. Patient Profile.
Name: Mr. N.R.H.
Age: 70 years old
Gender: Male
Address: Salawag
Date of Birth: March 22, 1939
Place of Birth: Salawag
Nationality: Filipino
Religion: Roman Catholic
Civil Status: Married
Occupation: Resigned/Retired
Date of Admission: November 19, 2009
Time of Admission: 5:00 pm
Physician: Dr. Sembrano
Place of Admission: Metro South Medical Center
Admitting Diagnosis: t/c Transient Ischemic Attack, Hypertension Uncontrolled
Chief Complaint:
• The patient complains of Stiffening of Extremities and Dizziness.
Renin-Angiotensin-Aldosterone system. When blood volume falls or blood flow to the kidneys decreases,
juxtaglomerular cells in the kidneys secrete renin into the bloodstream. In sequence, renin and angiotensin
converting enzyme (ACE) act on their substrates to produce the active hormone angiotensin II, which raises
blood pressure in two ways. First, angiotensin II is a potent vasoconstrictor; it raises blood pressure by
increasing systemic vascular resistance. Second, it stimulates secretion of aldosterone, which increases
reabsorption of sodium ions and water by the kidneys. The water reabsorption increases total blood
volume, which increases blood pressure.
Antidiuretic hormone. ADH is produced by the hypothalamus and released from the posterior pituitary in
response to dehydration or decreased blood volume. Among other actions, ADH causes vasoconstriction,
which increases blood pressure.
Atrial Natriuretic Peptide. Released by cells in the atria of the heart, ANP lowers blood pressure by causing
vasodilation and by promoting the loss of salt and water in the urine, which reduces blood volume.
VII. Pathophysiology.
Family
History
Prolongs vasoconstriction
Inflammation
Increase sodium,
calcium, water, proteins, humoral substances
Increase vasoconstriction
DEPENDENT:
Give due medications
Assessment Diagnosis Planning Intervention Evaluation
SUBJECTIVE: INDEPENDENT:
“Bakit kaya Fatigue related After 8hours of • Teach name, dose, After 8 hours of
madalas ako to effects of nursing action, and side effects of nursing
mahilo?” hypertension intervention her antihypertensive interventions,
as verbalized by the and stresses of blood pressure medication. the patient was
patient. daily life. readings will able to manage
reduce to less • Discuss stress-reducing his stress.
OBJECTIVE: than 150 techniques, helping
¨ Request for systolic and 90 identify possible • Goal Partially
information. diastolic choices. Met
¨ Agitated
behavior DEPENDENT:
¨ Inaccurate •Give due medications
follow through
of instructions.
¨ V/S taken as
follows:
T: 37.2
P: 84
R: 18
BP: 180/110
X. Discharge Planning.
Medication:
• Take home medication as prescribed by the physician.
Exercise:
• Instruct to walk for at least 15mins a day as an exercise.
Treatment:
• Take home medication and follow dietary plans
Health Teaching:
• Discourage patient from stressful and heavy activities
Out Patient Follow-Up:
• Instruct patient to visit physician if signs and symptoms of HTN shows.
Diet:
• Instruct patient with Low Salt, Low Fat, No Sweats and Tea diet.