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Home Page CON - DRUG SCIENTIST CON - FAKE PHYSICIAN

THE CON

DR. MICHAEL H. PFEIFFER


THE CON- BECOMING A
FAKE PHYSICIAN
Playing Dangerous Games
with Lives

CON - DRUG SCIENTIST

RESEARCH DEGREE (PHD) IN NEUROPHYSIOLOGY /


HUMAN EXPERIMENTAL DRUG RESEARCHER / SCIENTIST

Mr. Michael H. Pfeiffer conducted a full schedule of research for 6 years at the institute of physiology and
neuroscience laboratory at Charite Hospital at Humboldt University in Berlin at the same time that he
claims to have been a full time medical student at the same university.

1994-1999

PhD (Research Degree), May 1999, from Laboratory of Professor Dr. Uwe Heinemann Institute of
Physiology at the Medical Faculty of Charité, Humboldt University, Berlin, Germany

"Neuronal and gilal GABA-transporter and and their junction in the transition form
pharmacosensitive to pharmacoresistant forms of epileptiform activity in the
temporal lobe of the rat in vitro."

Pfeiffer, M., Draguhn, A., Meierkord, H. & Heinemann, U. (1996). Effects of gamma-aminobutyric acid (GABA) agonists and GABA
uptake inhibitors on pharmacosensitive and pharmacoresistant epileptiform activity in vitro. British Journal of Pharmacology
119(3):569-77.

Source Abt. Neurophysiologie, Humboldt-Universität zu. Berlin, Germany; British Journal of Pharmacology 1996
Oct;119(3):569-77. (http://www.ncbi.nlm.nih.gov/pmc/articles/PMC1915701/?tool=pubmed)
PhD Research Source of funds: Deutche Forschungsgemeinschaft (DFG) The German Research Council - Research
Collaboration Center - no. SBF 506

Abstract: Lowering of the extracellular Mg(2+)-concentration induces various patterns of epileptiform activity in
combined rat entorhinal cortex-hippocampal brain slices. After a prolonged period of exposure to Mg(2+)-free
medium seizure-like events in the entorhinal cortex change to a state of late recurrent discharges which cannot
be blocked by clinically available antiepileptic drugs. This late epileptiform activity thus represents a useful
model to test the effects of new anticonvulsant substances. 2. A mechanism possibly underlying the
development of sustained seizure-like activity is the loss of synaptically released gamma-aminobutyric acid
(GABA). Drugs which increase the amount of GABA available in presynaptic endings might thus be useful in the
treatment of these therapeutically complicated forms of epilepsy. 3. Therefore, we studied the effects of various
substances increasing GABA-mediated inhibition on early and late forms of epileptiform activity. GABA and the
GABAA receptor agonist muscimol blocked both the pharmacosensitive discharges in the hippocampus and
entorhinal cortex as well as the late recurrent discharges in the medial entorhinal cortex. The GABAB receptor
agonist baclofen blocked the recurrent short discharges very potently, but did not consistently block seizure-like
events and late recurrent discharges in the entorhinal cortex. 4. GABA uptake blockers showed a differential
potency to block the various discharge patterns. Whereas nipecotic acid and beta-alanine suppressed all forms
of epileptiform activity albeit at high concentrations (1-5 mM), tiagabine was much more potent in blocking the
hippocampal recurrent short discharges and the seizure-like events in the medial entorhinal cortex, but could not
block the late recurrent discharges. 5. Our data support the idea that prolonged neuronal overactivity might result
in a loss of synaptically available GABA. Selective block of uptake into glia cells or substitution of the transmitter
may therefore be an efficient strategy for the treatment of severe prolonged epileptic discharges whereas block
of neuronal GABA uptake fails to counteract synchronized discharges in this situation.

Note: Michael H Pfeiffer Research - German Research Collaboration Grant 506 (1997-2003) given to Prof. Dr. med Peter
T. Daniel Director Molecular Hematology and Oncology, Charité - Humboldt University Campus Berlin, Germany
Publication: Cell Death and Differentiation (2003) 10, 461–467. Combined p53/Bax mutation results in extremely poor
prognosis in gastric carcinoma with low microsatellite instability Edited by V De LaurenziA Mrózek1, H Petrowsky 3,
I Sturm1, J Kraus2, S Hermann1, S Hauptmann2, M Lorenz3, B Dörken1 and Peter T Daniel, German Research Grant
SBF 506

Research Student Scholarship 1996


Student Scholarship - October 01,1995 to February 28,1996
Medical Faculty, Humboldt University Berlin Graduate Program
March 01 to December 31,1996

Mechanisms of Damage in CNS Disease -


Application of Imaging Techniques"
Medical Faculty, Humboldt University Berlin

Research Presentations 1996

Congress of the Society of Neuroscience in Berlin, Germany,1996: 'Effects of GABA uptake inhibitors on
different patterns of low magnesium induced epileptiform activity in rat entorhinal cortex-hippocampal
slices" in Symposium: "Entoriiinal-hippocampal interactions: From cellular mechanisms to clinical
aspects"

Meeting of the Graduate Program "Mechanisms of Damage in CNS Disease - Application of Imaging
Techniques" in Eggsdorf, Germany,1996:

'Effects of GABA-receptor agonists and of GABA-uptake inhibitors on different forms of epileptiform


discharges in combined brain slices of rats in the low-magnesium model of temporal lobe epilepsy"

Mr. Pfeiffer’s Thesis records from the German National Library show he did receive a PhD.

Strong evidence indicates that Michael Herbert Pfeiffer is trained as a human experimental drug
researcher/ scientist (PhD in medical neurophysiology) from the Institute of Physiology
(Neurophysiology) in the Medical School at Charite, Humboldt University Hospital. He who has used
people in the USA misunderstanding of the German educational and medical systen background to falsely
receive a medical license for practicing medicine without proper medical training credentials.
PhD RESEARCH PUBLICATIONS
1996-1999 THE CON– REINVENTING – FROM
SCIENTIST TO FAKE PHYSICIAN
“Look for Publications of M. Pfeiffer in PubMed”
Born 18 February 1963 in Nuremberg,
Journals with Michael Pfeiffer's Research
(Full publications below to download) Germany.

European Journal of Pharmacology (1999) Home base: Pettenkoffer str. 5,


Bayreuth, Germany
British Journal of Pharmacology
(1996,1999,2000) Father: Dr. rer pol. Johann Michael
Pfeiffer, President E-On International
Nervenartz- "Drug Research" (1999) Division (retired).

Naunyn-Schmiedeber's Archives of Mother: Margarite Wilhemine Pfeiffer


Phamracology (1999) (Garruth)

Journal of Neuroscience Methods (1997) Brother: Detlef Georg Pfeiffer Child


_______________________ Diagnosis: "Autism"
The PhD Research leading up to the PhD in
1982 Graduated High School Gymnasium
1999 and the publication of its research paper
Christian-Ernestinum Bayreuth, Germany
from 1996-1999.
1983-1984 Military Service
Pfeiffer, M., Draguhn, A., Meierkord, H. &
Heinemann, U. (1996). Effects of gamma- 1985/1985 Entered University for
aminobutyric acid (GABA) agonists and GABA Engineering - did not finish; studies
uptake inhibitors on phamacosensitive and interrupted for 1-2 years. Must move
phamacoresistant epileptiform activity in vitro. back home with parents in Bayreuth,
British Journal of Pharmacology 119 (3): Germany.
569-77.
1990-1991 Semmelweis University Medical
School, Budapest Hungary (September
Draguhn, A., Pfeiffer, M., Heinemann, U. 1990 to April 1991 - (2 years of 6 year
&Polder, R. (1997). A simple hardware model program) "left, no progress"
for the direct observation of voltage-clamp
performance under realistic conditions. Journal
of Neuroscience Methods 78 (1-2):105-13. 1992-1994 Students of the French
Language, 1st Degree Study; Ministry of
National Education of France. Diploma for
Meierkord, H., Griinig, P., Gutschmidt, U., Students of the French Language, 1st Degree,
Gutienez, R., Pfeiffer, M., Draguhn, A., Ministry of National Education of France, June
Briickner, C. & Heinemann, U. (1999). Sodium 1994, Berlin
bromide: effects on different patterns of
epileptiform activity, extracellular pH changes 1994-1999 P hD in Neurophysiology
and GABAergic inhibition. Naunyn granted from The Johannes-Müller-Institut für
Schmiedebergs Archives of Pharmacology Physiologie der Charité, Humboldt-Universität
361 (1):25-32. zu Berlin (The Institute of Physiology at the
Medical Faculty of Charite, Humboldt University,
Berlin) , Germany
Holtkamp, H., Pfeiffer, M., Buchheim, K. &
Meierkord, H. (1999). Tiagabin und
nontonvulsiver Status epilepticus (Tiagabine 1994 September: Start as PhD Student – Year
and nonconvulsive tatus epilepticus). 1 Programme at Johannes-Müller-Institut
Newenarzt (Drug Research) 70 (12). 1104- für Physiologie der Charité, Humboldt-
1106. Universität zu Berlin Charite uner Uwe
Heinmann, Physiology Institute,
Sabau. A., Frahm, C., Pfeiffer, M., Breustedt, 1994 - Year 1 - PhD Student
1.-M., Piechotta, A., Numberger, M., Engel, D.,
Heinemann, U. & Draguhn , A. (1999). Age
Institute for Physiology
Dependence of the anticonvulsant effects of the
GABA uptake inhibitor tiagabiie in vitro.
European Journal of Pharmacology, 383 (3), 1995 - Year 2- PhD Research Charite,
259-266. Experimental Physiology Institute, under Dr.
Uwe Heinmann

Sleziki KI, Cho YW, Yi SD, Brock MS, Pfeiffer 01 October 1995 to December 1995
MH, Mcvearry KM, Tractenberg RE,
Motamedi, GK (2009). Department of Graduate Student Scholarship
Neurology, Georgetown University Medical
Center, Washington, DC, USA. Incident of
atypical handedness in epilepsy and its 1996 - Year 3 - The Active Student
association with clinical factors. Epilepsy Researcher Year
Behaviour, 2009 Oct. 16(2):330-4. Epub 2009
Aug 28. PMID: 19716770.
PhD Student – Year 3- Programme at Charite
uner Uwe Heinmann, Physiology Institute,

1 January to 28 February 1996; Student Scholarship,


Medical Faculty, Humboldt University Berlin

1 March 1996 to 31 December 1996;


Graduate Scholarship, Graduate Program
“Mechanisms of Damage in CNS Disease -
Application of Imaging Techniques” (see Note:
NeuroCure)

Meeting of the Graduate Program Presentation:


“Mechanisms of Damage in CNS Disease –
Application of Imaging Techiques“ in Eggsdorf,
Germany, 1996: „Effects of GABA-receptor agonists
and of GABA-uptake inhibitors on different forms of
epileptiform discharges in combined brain slices of rats
in the low-magnesium model of temporal lobe
epilepsy“.

Congress of the Society of Neuroscience in


Berlin, 1996: Presentation: “Effects of GABA
uptake inhibitors on different patterns of low
magnesium induced epileptiform activity in rat
entorhinal cortex-hippocampal slices” in
Symposium: Entorhinal-hippocampal
interactions: From cellular mechanisms to
clinical aspects

Pfeiffer, M., Draguhn, A., Meierkord, H. &


Heinemann, U. (1996). Effects of gamma-
aminobutyric acid (GABA) agonists and GABA
uptake inhibitors on pharmacosensitive and
pharmacoresistant epileptiform activity in vitro.
British Journal of Pharmacology 119(3):569-
77.

1996-1999 PhD Research publications from


Humboldt University, Berlin, Germany

2000 Germany to the USA- CV Claims he


was award of MD/PhD degree in 1998, but
no joint MD/PhD program until 2002.

2001-2003 Research Fellow, Baylor College


of Medicine, Neuroscience Laboratory,
Houston, Texas

2003-2004 Internal Medicine Resident, St.


Francis Medical Center, Trenton, NJ

2004-2007 Neurology Resident, Georgetown


University Medical Center, Washington, DC
20007

Children’s National Medical Center,


Washington, DC

Washington Hospital Center


Washington, DC

Veterans Administration Medical Center,


Washington, DC

National Institute of Health Stroke Unit,


Washington, DC

2007-2008 “Neurology Fellowship”

Mr. Pfeiffer practices of medicine at


Georgetown University Medical Center
Department of Neurology without a medical
license.

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