Cardiogenic pulmonary edema (CPE) is pulmonary edema caused by increased fluid buildup in the lungs due to high pressure in the pulmonary veins from cardiac dysfunction. CPE results when fluid with low protein accumulates in the lung tissue and air sacs as a consequence of the heart not pumping effectively, as seen on x-rays showing fluid in the lungs of a patient admitted for a heart attack.
Cardiogenic pulmonary edema (CPE) is pulmonary edema caused by increased fluid buildup in the lungs due to high pressure in the pulmonary veins from cardiac dysfunction. CPE results when fluid with low protein accumulates in the lung tissue and air sacs as a consequence of the heart not pumping effectively, as seen on x-rays showing fluid in the lungs of a patient admitted for a heart attack.
Cardiogenic pulmonary edema (CPE) is pulmonary edema caused by increased fluid buildup in the lungs due to high pressure in the pulmonary veins from cardiac dysfunction. CPE results when fluid with low protein accumulates in the lung tissue and air sacs as a consequence of the heart not pumping effectively, as seen on x-rays showing fluid in the lungs of a patient admitted for a heart attack.
Cardiogenic pulmonary edema (CPE) is pulmonary edema caused by increased fluid buildup in the lungs due to high pressure in the pulmonary veins from cardiac dysfunction. CPE results when fluid with low protein accumulates in the lung tissue and air sacs as a consequence of the heart not pumping effectively, as seen on x-rays showing fluid in the lungs of a patient admitted for a heart attack.
Cardiogenic pulmonary edema (CPE) is defined as pulmonary edema due to
increased capillary hydrostatic pressure secondary to elevated pulmonary venous pressure. CPE reflects the accumulation of fluid with a low-protein content in the lung interstitium and alveoli as a result of cardiac dysfunction (see the image below). (See Etiology.)
Radiograph shows acute
pulmonary edema in a patient who was admitted with acute anterior myocardial infarction. Findings are vascular redistribution, indistinct hila, and alveolar infiltrates. View Media Gallery