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  • Giant Cell Arteritis

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    Contributory factors for giant cell arteritis include being over 50 years old, female gender, Caucasian race, family history, and living in Scandinavian countries. The disease process involves an abnormally activated immune system where antibodies attach to the walls of the temporal artery, increasing inflammatory cells and proteins that damage the arterial wall. This can lead to headaches, jaw pain, vision problems, and other symptoms as immune complexes travel throughout the body.

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    Giant cell arthritis

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    Contributory factors for giant cell arteritis include being over 50 years old, female gender, Caucasian race, family history, and living in Scandinavian countries. The disease process involves an abnormally activated immune system where antibodies attach to the walls of the temporal artery, increasing inflammatory cells and proteins that damage the arterial wall. This can lead to headaches, jaw pain, vision problems, and other symptoms as immune complexes travel throughout the body.

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    © All Rights Reserved
    Download as DOCX, PDF, TXT or read online from Scribd
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    Download as docx, pdf, or txt
    24 views1 page

    Giant Cell Arteritis

    Uploaded by

    Aye
    Contributory factors for giant cell arteritis include being over 50 years old, female gender, Caucasian race, family history, and living in Scandinavian countries. The disease process involves an abnormally activated immune system where antibodies attach to the walls of the temporal artery, increasing inflammatory cells and proteins that damage the arterial wall. This can lead to headaches, jaw pain, vision problems, and other symptoms as immune complexes travel throughout the body.

    Copyright:

    © All Rights Reserved
    Download as DOCX, PDF, TXT or read online from Scribd
    Flag for inappropriate content
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    of 1

    Contributory factors:

    GIANT CELL ARTERITIS


    Age (50 years old and above) s LEGEND
    Gender (women) Dark green- Contributory factors
    Race (Caucasians)
    Abnormally activated Immune Yellow- Title
    Hereditary (first degree relatives) system
    Light green- disease process
    Geographical (Scandavian Countries)
    White- Symptoms manifested
    Antibodies attached at the
    walls of the temporal artery

    Headache Release of immune


    Vascularization
    Increased circulating peptides and protein
    of the vessel
    monocytes producing IL-1 and
    IL-6

    Breakage due to Immune peptides and


    Jaw the damage of proteins travel through
    pain the vessel wall Ophthalmic artery Systemic Circulation
    is affected

    Halos

    Release of vessel Blurring of vision


    components Fatigue Fever Myalgia Chills Night
    Stiffness of the Blindness
    neck sweats

    Antibodies
    Immune complexes Immune
    surround the
    travel through complexes clog Arthralgia
    components and
    systemic circulation the joints
    form immune
    complexes

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