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Monocyte ·Dt: % (01-08) Creatinlne . . mg/di · (0.7-1.4)
Basophils % (<01)
LIPID PROFILE
ESR mm in lhour (M:<10,F:<20) Total CholesteroJ mg/di (<200)
AEC (<450) Triglyceride mg/di (<?,60)
Platelets / •.2~ -. (1.5-4.SLac) HDL . mg/di (40-60)
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MH BHUJ LAB REPORTS
HAEMATOLOGY BIOCHEMISTRY Wida!
SUGAR (F) .................... (" ... mg/dL
ESR .................... Cinm fall 1st wintrobe's
SUGAR (PP) ......... ... ';J ...... mg/dL
Hb ........... u.~.~···· ·
gm/dl
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SUGAR (R) ......n ..lJ..~······· mg/dL
TO .................... TH .................... .
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TLC .. i.JQ.Q .... cell/cumm UREA ............... k:1 ............ mg/dL AH .................... BH .....................
CREATININE .....0 .~.~ ... mg/dL
DLC BILIRUBIN ..... rly.. .. 7
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Sample Profile
STP pHo..< Ultra
' Printed 'J9/01 /2020 02 26 20 PM
Analyzed . 29/01/2020 02 24·11 PM
Analyzer ID z1 ·1A16340N
D· I < ~ .
B r 752 6 mmHg
Sample Type Venous
Operator 123456
Releaser auto
Patient ID 1cu
Patient Name
Qther F@g~
Comments
,:
'
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Investigotion View Pag.c 1 of 2
~ Urea 12
PCV 29 .9
Packed Cell Volume
MCV 85.3
MCV
MCH 29.3
MCH
MCHC 34 .3
MCHC
31-01-202(
https://pwsapp/NHIMS/OPD/Casesheet_InvestigationResultView.aspx?VisitID=356470 ...
· Jm esti!?ation View Page 2 01 z
ULTRASONOGRAPHYREPORT
ABDOMEN ,,aol,,JP,Po~ ~ - (...o C - y ~ -
1._Jo L,..7V.
Gall Bladder
Both Kidnevs Right Kidney : II c.....,__, in size. CMD is maintained. Nonnal echo.
No Ascitis / RPLNE.
p uv, ~ J-...'..,
Comments:
1es
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r ~ v,n,
p F,8579 /2 .it> D c.,,.:.,....,, t.. , : , ~ . . I ) ~t " pu~ c-f ~ p .,_J ~ " " ' c,.., ' .,
IMPRESSION : J c..J~
.
Ll,7 • PP-ASHWINI HOSPITAL, COLABA
Near RC Church, Colaba Mumbai,
Maharashtra ,00005
1/2/2020 6 41 OOPM
CollQC:tod
Name SGT. O.K.JHA 904177S 11212020 o o.,oPM
Rf!colvod
M:alu 3/2/2020 5.48 02PM
Lab No. 276490500 Ago: 32 Ve3ra Gondor: Hoportod
Report Statua Final
Ale Status p RofBy: AMW
OrN=~
l,!!}~liSlr)
~ ~-Oneal Cher=!.,y &
-
o, PINI C"<>Pt•
!IJ) Labotlto,y MIJdleina
Consul.ant
a.oc..- a.a Gencl::s llRl. • Or Lal Patnu,b1 Ltd
NRL-OrutlFi~W
- - - - - - - - - E n d of r e p o r t - - - - - - - - -
IMPORTANT INSTRUCTIONS
•r~ ~ r ~ per.ail'l to the tpeetmen subm,ned "AH test results are dependent
•~ ~ a•e c,riy a toot lo tacirtate 1n amvang at a diagnosis and ghould be dmtca!ly CQf'l'ela!e,
are ~ oo rei.;.iest o1 Refemng Phys10an within 7 days post repo111ng •Repon de!.'.'1ery
~...161JQ';! r&Qrded "Certa n tests may require further testing at add tlorul cost fur
72 h:>url ~ repotf!!IQ "Tit$! rosults may &how antortaborntory variations "The
related w te I ro~ults
U7 • PP.ASHWINI HOSPITAL, COLABA
~ RC Church, Colaba Mumbai,
lbharut\trll '00005
ANTI NUCLEAR ANTIBODY/ FACTOR ( ANA/ANF) IFA (HEP-2) ENO POINT TITRE @
' =A
Negative
1:100
Note
1. lmrr:mc;ffucrescence microscopy using human cellular extracts like HEp-2 cells is a sensitive test for
ce~e::ton of serum antibodies that react specifically with various cellular proteins and nucleic acids
2 Test conducted on Serum.
3. Pattems are ieported as per International consensus on ANA Patterns (ICAP).
Comments
An• Nu...'iear ant.body ( flcNA I A..NF) Is a group of autoantibodies directed against constituents of cell n ~
ru:t.td:tg DNA, RttlA &. various nuclear proteins. These autoant1bodles are found with high froquc~, in pa• ~
2h connect.ve tissue di&erders specially SLE. Since positive ANA rosults have boan rtiportoo ,n he.at~,
ma idua1s these reactivitie, aro not by thornselvoa diagnostic but must bt1 corrolatod with otht1~ lJboratZlf')
:"11Ca1f~ngs
L •7. PP-ASHWINI HOSPITAL, COLABA
Near RC Church, Col1b1 Mumbll,
Maharashtl"I 400005
I1-----------------1-----------------------
'=1.00 I Reactive/ Acute/
I I Resol\ing / Chronic
I I HBV infection
Note
1 Discrepant results may be observed in patients receiving mouse monoclonal
antibodies for d1agnos1s or therapy
2. For hepannized patients. draw specimen pnor to heparin therapy as presence
of tibnn leads to erroneous results
Comments
Anti- 1--iBc Total s the first antibody to appear usually 4-10 weeks after appearance of HBsAg a• -., sa,~
time as dinical i 1ness and persists for years or maybe hretime. It is almost always presen1 cu...:,g chrm'=t
1
...:gv infection. It detects virtually all individuals who have been previously infected with HBV. Oete:::t.:r. of Ar.
i.;Bc Total positi\-e donors reduces incidence of post transmission Hepatitis and poss o ty 0
1
er.---:-·
nfections like HIV due to frequency of dual infections. This antibody may be seen 1n 2% o1 ro..t. ne ckr::lrs
without any other serological marker and with normal liver enzyme levels. This 1nd1cates rec~ =TY Ml--
suxl:mcal r.BV ,r,fect,ons. Anti HBc Total is not protective and cannot be used to dlStingu.s.,i. :..O.....e ~
Chron c mfection.
Uses
• A.s a marker for HBV infection
• f..s a screening test for blood donors
8.00 ugldl
Iron
fut ral'on
Comments
Iron an rrt I lt min r I I m,mt wht h form lli
L27. PP-ASHWINI HOSPITAL. COLABA
,-.~ar RC Church, Colaba Mumbai,
Maharashtra 400005
: 1/2/2020 6 41 :00PM
Collected
. 1/2/2020 6:43 :40PM
Name SGT. D.K.JHA 904177S Received
3/2/2020 5:48:02PM
Gender: Male Reported
Lab No. 276490500 Age : 32 Years
Final
Report Status
A/c Status p Ref By : AMW
Comments
Serum ferritin appears to be in equilibrium with tissue ferritin and is a good indicator of storage iron in normal
subjects and in most disorders. In patients with some hepatocellular diseases malignancies and
inflammatory diseases, serum ferritin is a disproportionately high estimate of storage iron because serum
ferritin is an acute phase reactant. In such disorders iron deficiency anemia may exist with a nonnal serum
ferritin concentration. In the presence of inflammation, persons with low serum ferritin are likely to respond to
iron therapy.
Increased Levels
• Iron overload - Hemochromatosis, Thalassemia & Sideroblastic anemia
• Malignant conditions -Acute myeloblastic & _Lyrnphoblastic leukemia, Hodgkin's disease & Breast
carcinoma
• Inflammatory diseases - Pulmonary infections, Osteomyelitis, Chronic UTI, Rheumatoid arthlitis.
SLE, burns
• Acute & Chronic hepatocellular disease
Decreased Levels
Iron deficiency anemia
HEPATITIS B CORE ANTIBODY (Anti- HBc), TOTAL, 0.24 Index <1 .00
SERUM
(ChemiluminesE~t Microparticle lmmu!'oassay)
Interpretation
t:ltf $2 ■ 1.
......._
. . AII . . . NQIPl1'N..CGI.W
-•CIMllh.o...--.
AA:Slalua
l?MlolOO
..
At• uv..... Gender ..... ...,.,...
NtOllvtd ,1112no t;41...,..
~•M~
,,,..
AtlW lll1po,111Mtu•
ftttf ·~
,...........
~ ( A ~ f"E"°"'°mN), TUM°" MAltKll!lt,
----
Nole
Ct."
•
Useful for determining prognosis and tnOnf10fing thWepy for Hepatocellular carcinoma ~ of
AFP rs a prognostic mdicator of IUMvaf Elevated AFP and serum bllirubin levels in . _ . .
patients rs assoc:iated with shorter survrva1 time
•
An aid ,n the management of Gem, cell (Non.Sem nomatous) tumors. Measurement of ~P
le\,els m oomblnabon with HCG levels are useful n dassdying and staging Germ cell tumors
• To pt8dic:t tumor recurrence/presence of residual tumor
Inc, l ■ dLewals
.•
•
-----~
Gl■I cw t • 1111 tr.a cancers with or without liver metastas,s
. . . , , hip ■tc CDnditiona fb Acute Vtral Hepatitis, Chronic active hepatrua and ~
·--►~-.,.
34 10
""'91N 0 ..
Wtt, IUJI'
~, ,,on
f\lllllNd~ l) ~kT._._.,_,,Nll•ot
re,.,_
Mil I0:9MOA1Ncw»py IIPOU
bOJB@IA8US
Of~~ Gr.ae Ihe: l, Gtide I JC 2 no RCS, no IU9mat•
D-l: Normal
D-2: Nom'laf
I • C t - • -, '· • • •
._ilAL1Rra:r4ao..
IIUFl&I PhL "••rca
. . ...... . .
ECHOCABDIOGRAPHY REPORT
DEPARTMENT OF CARDIOLOGY
INHS AS~INI, COLABA, MUMBAI - 4 OOS oo
Name : D{: ;:)o..... s~f/Wife,'SeA/Daughte,/Metl:ler/Fa t her-ef - - - - - - - - -
Ejection Fraction
/ Dilated/Hypertrophied
E/A fl.. )/1
Left Atrium ~
--
rma~ Dilated
RWMA - LA ex/RCA (),_,ii{ WAA/J-
Norma Mild/ Moderate/ Severe.
Right Ventricles Dilated Right Atriumy / Dilated
Tricuspid Valve ~
c;7~ (d70
~{d/4~
/Abnormal
0
Plum Valve a Abnormal
lAS/NS
<:vu,! /MA-
Ase Aorta IVC: SVC:
Pericardium
CONCLUSION
CARO OlO
.............
ECHOCARDIOGRAPIIY REPORT :/
DEPARTMENT OF CARDIOLOGY
INHS AStJINI, COLABA, MUMBAI • 400 005
Name :
Rank ·.
-
.[\
-
{!
--- •
.:io.,_
------l.-J.....A,....::::~- -
p No .
• ---Ll~ :.....L-QF-
Age :
~ e
s:ff/Wife/SeA/Daughter/M 9t t:ler/Fat heH1f - - - - - - - - -
Yr - M F Date
(23~ l'?OZZ) ECHO No:;___ _
~
~
Norma Mild / Moderate/ Severe.
Right Ventricles Dilated Right Atrium / Dilate
~
IAS/IVS
Pericardium
CONCLUSION
(S~ ' l
- C
Department of Radiodiagnosis & Imaging
INHS ASVINJ Colaba, Mumbai - 400 005 20 0 -...
Tel.: 2214 3705 / 2214 3500-3705 r
ULTRASONOGRAPHY 'REPOT{T
c~~ ~ ~~/ ~
A ti...Jj CMAL, 1. ,) f.µti1w.. i.)ui. k
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~JPRESSJON :
- CONFIDENTIAL
INHS Asvlni
Near RC Church, Colaba, Mumbai
AG NETIC RESONANCE I MAGI NG
;P/ GASTROENTROLOGY
Cateuory : Service Person W •rd / OPD AND RESPIRATORY
WARD
O rdered By
: CAPT ( MNS) SARU CHAUHAN NS23313L Requisition No : 579511
Auth o rized B y
: SURG. CDR HASHIM P I MR072 57N Requisition Date : 04 Feb 2020
Auth o rized O n
: 08 Feb 2020 Accession No : 200204189
Rel e v a nt Clinica l
D e t a ils
Sca n D e t a ils
Acut e on chronic liver failure
Hematemesis & malena
Findin gs/ Observations Gross ascitis seen- perihepatic, perisplenic, interbowel & pelvic.
Liver has surface nodularity . It is normal in size, signal intensity .Caudate lobe
h y pertrophy seen . There are no focal areas of altered signal intensit y on any
sequ ence .
....
Interpretation iii ;.-
FVC FEV1 FEV1%
6 ~ ~
Normal Spirometry
D1agnost1cs
Interpretation(s)
CYTOMEGALOVIRUS !GM, SERUM -
CYTOMEGAlOVIRUS !GM, SERUM
Cytomegalovrrus (CMV) rs a member of the Herpesvirldae family and Is classified as Human herpesvrrus Type S.CMV causes a number of protean disease synoror.,es m mfants
as well as adults. lnfect,on ,s common and reaches most or the population, whereas associated disease Is relat,vely rare. CMV rs a recognized cause of monoeuClec;s s a-~
hepat,os amongst normal Immunocompetent rndivlduals .But ,tis among the immunosuppressed (Immature neonates, organ transplant reop,ents. AIDS pat>ents) t.-:at CMV
causes most s1gnrficant disease, manifesting as hepatitis, retinitis, pneumonitis, encephal1t1s, colitis etc.
The nsk of an infected pregnant woman transmrttrng CMV to the foetus is highest ,n the 3rd trimester and during the brrth process (Perinatal infeet,on).
Jntrautenne/Congenrtal CMV infections, though less frequently seen than perinatal infections, are responsible for causing severe CMV diseases that may be fatal. Such
rntrautenne/congenrtal CMV ,nrect,ons are usually seen in infants born to mothers suffering from a primary infection during pregnancy
Test Utility:
CMV speofic lgM develops a few weeks after acute infection followed by development of lgG about a week later. lgM levels usually increase for some wtt..s an<! then
decrease slowly rn four to srx months. Occas10Aally, lgM may circulate for years.
A pos,t,ve CHV lgM result may not always indicate a pnmary acute Infection, as lgM has a tendency to persist, even at high levels, after pnmary inlect,on. false-l)<ls t,ve \;M
results may ocwr due to rheumatoid factor and antinuclear antibodies. Hence, lgG avidity testing Is recommended to d1Herent1ate between pnmary 1nlectt0n, \gM ~ s \ e ~
and reactJvat,on.
Avidity ,s defined as the functional brndrng strength of antibodies to multiple binding sites (epltopes) on the antigen . The test 1s based on the pnnctl)le tl'\at ant bac1~s IOl'meG
rn response to promary infection have relatively low avidity to the corresponding antigen With time, a broader antibody response develops "41th ant1t)od1t'> bemg tanned to
more ep1topes on the ant,gen and with a corresponding Increase In the antibody antigen avidity . Therefore, when a secondary ant,boJy response occurs wtth re1nl~n
stimulates donal expansion of memory B cells to a much wider spectrum or epltopes, producing antibodies of conslderably greater avidity.
A pos,t1ve IgM accompanied by low-av1d1ty JgG Is suggestive or a primary Inrect1on, whereas a high avidity lgG Ind1cates either lgM persistence or reat\"at,on ._'°"'av.
,ndex may also be seen In a proportion of Infected persons for months Hence rt Is adv,sed to perform lgM testlnq InItI,1lly to l'lllnt to the neN for ll)G G'IIC ty \o a.
m1smterpretat,on of results.
Um1ta1,ons:
Samples which ar~ s1ron9ly pos1t111e for the prl"~enti• uf anti V,1rln·ll,1 70~1, r (VIV) Mid (lJ~l rln O,,rr lqM ,111tIlto,lles till\ qMi l,11,e posih~e results ~se
compllcat,ons of SProlo91c d1;,9nos1s of uin9l'n1tal lnfi•rllon, vIru•, 1~01,111011rro1111111m 111 thr llr ,l wr1·I. ot 1111'! rr111nm~ lh" b< ,t w11y to d111g~ 111\r!lule
A~ence c,f CMV si,euf1r JgM does not f )(((udt thr pc,<•ll11llty c,f (MV "''"' 11011 11110111 l tl 10"/u ul 1111,111\ • lll~Y 1011 to (\t"velc,p (Ml/ lg!o\ llflhbody ~Mtt
1nfect1on with ow.
• .tend Of Rrpo,t• •
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CONFIDENTIAL
JNHS Asvinl
Near RC Church, Colaba, Mumbai
NG COMPUTED TOMOGRAPHY
OEPARTMENT OF R.ADlOLOGV AND lMAGl •
UH10 : ASVl.00003-40151
Name : 0 Y. JHA
Relation : Self
Authorbed By • SURG. LT COR NlHARIKA GUPTA 76008w Roqulsltlon Date : 06 Fe!> 2020
Procedure : Chest CT
Scan Details 5 mm sections through chest an a cranlo-caudal direction in spiral mode were
obtained from the level of root of the neck till the adrenals and images
reconstructed in both lung and soft tissue windows.
Findings/ Observations • Small loculated pleural effusion noted along right lateral chest wall adJace~: to
lateral basal segment of Rl.l for ~3 x 0.8 cm in max dimensions .
• Subsegmental patchy consolidation is seen involving the antenor segment c1 tt--.e
Rl.l. Rest of the lung fields are clear .
• Few discrete lymph nodes without any obvious necrosis or calc1ficaticn n~t~ .,.
paratracheal, pretracheal & precarinal stations with largest measunng S m-n
MSAO at right precarinal station.
• The trachea and main bronchi are central and normal.
• The heart and mediastinal structures including the oesophagus are norm.a
• The i11sualised spine appears normal.
• No left pleural effusion seen.
In the FOV,
Hypodense collection is noted In perihepatic & pensplenic location & ngh!
subdia phragmatlc location.
Swfac.e nodularlty of liver noted.
Name
: 0 K JHA. Rank Unit : 22 AU
: SGT
Age
: 32 Gender : 904177S
: MALE P.No
NOK Informed
NOK Address : JHA, S, SMQ-144/6 AF STATION NALIYA, JHAKHAR, Samastipur, Bihar, INDIA, 9140936807,
997839242
V\Shvv~CS
f1.":1i A';IJ,,._
\\ll l'\~► RESIDEN i
\Nr\) A~'v\N\
CONFIDENTIAL A.F.M.S.F - 9
SPECIAL INVESTIGATION FORM
INHS ASVINI Colaba, Mumbai- 400 005
PART-1
WARD - f\V.J..- I[.
NAME: RANK: i!, P. NO.: ~co Lr, ~s
UNIT: L."2-- A--u AGE/SEX: Q'.l.-'171'1\i NATURE OF SPECIMEN: Lu .VO~
EXAMINATION REQUIRED:
VISHWANAt-fl-
DATE: 0 f\ -~IGN OF MOI/C
r
- p We_ N~e c hv.,_,-..-:i f:/ b ~--wl ~ 1~✓ fJ- 12, J,,(.. •