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Acute Gastroenteritis: Our Lady of Fatima University
Acute Gastroenteritis: Our Lady of Fatima University
COLLEGE OF NURSING
Km. 311, Maharlika Highway, 3100 Cabanatuan, Nueva Ecija
ACUTE GASTROENTERITIS
Case Study
Submitted by:
I. Introduction
VII. Pathophysiology
OBJECTIVES
General Objective
This study aims to convey familiarity and to provide an effective nursing care to a patient
diagnosed with Acute Gastroenteritis through understanding the patient history, disease
process and management.
Specific Objectives
I. INTRODUCTION
Acute gastroenteritis is inflammation and/or irritation of the digestive tract that can cause
nausea, vomiting, diarrhea, and/or abdominal pain that lasts less than 14 days. When
symptoms last 14 to 30 days, the condition is considered persistent gastroenteritis. When
symptoms last longer than 30 days, it is considered chronic.
Various pathogens and noninfectious agents cause gastroenteritis, and it is one of the most
common infectious disease syndromes. In the United States, viral gastroenteritis accounts for
about 50% to 70% of acute gastroenteritis cases, with norovirus being the leading cause. Since
the introduction of the rotavirus vaccine, the incidences of rotavirus gastroenteritis and related
hospitalizations have been greatly reduced. On the other hand, norovirus is common and highly
contagious.
After viral gastroenteritis, another 10% to 15% of acute cases have a parasitic cause, while
bacterial gastroenteritis accounts for about 15% to 20% of cases. Infectious diarrhea can also be
classified into 2 categories: inflammatory or non-inflammatory. Non-inflammatory diarrhea is
more common, and although the cause is usually viral, it can be bacterial or parasitic in origin,
as well. Non-inflammatory diarrhea, which is typically less severe than inflammatory diarrhea,
causes large, watery stool with cramping but no blood; fecal leukocytes are absent. Common
causes of non-inflammatory diarrhea are enterotoxigenic Escherichia coli, Clostridium
perfringens, Bacillus cereus, Staphylococcus aureus, rotavirus, norovirus, Giardia,
Cryptosporidium, and Vibrio cholerae. Inflammatory diarrhea is more severe and caused by
toxin-producing bacteria. It disrupts the mucosa, causing bloody diarrhea, abdominal pain, and
fever, and fecal leukocytes are present. Common causes of inflammatory diarrhea are
Salmonella, Shigella, Campylobacter, Shiga toxin-producing E. coli, enteroinvasive E. coli, C.
difficile, Entamoeba histolytica, and Yersinia.
II. PATIENT PROFILE
Name: A.D.C
Age: 2 y/o
Gender: Male
Citizenship: Filipino
Religion: Catholic
BP:
Temperature: 37.7 C
Prior to admission, the client was vomiting and defecating. Her stool was watery and its
color is green. At first, they went to the baranggay health center and the staff gave them
medication. According to the staff, the medication is for LBM, but after drinking the medication,
the client was still defecating and vomiting so the family decided to rush the client at Eduardo L.
Joson Memorial Hospital the next day.
Mrs. D.C Cruz says “ ito first time niya ma-admit buhat nung ipanganak ko siya.” A.D.
gets seasonal cough and colds at times but never serious because it usually last only for a few
days. They always consult their doctor once sick. He is complete in his vaccinations.
Family Medical History
According to the significant others of D.C they have a familial disease of asthma, both on
her father and mother's side. An incident of hypertension on his father's side.
Immunization History
The patient had a complete vaccination as follows:
September 1, 2017 (at birth) Vitamin K, Hepatitis B, BCG January 8,
2018 Pentahib1, OPV1, PCV1, Rotavirus1 February 5, 2018 Pentahib2,
OPV2, PCV2, Rotavirus2 March 4, 2018 Pentahib3, OPV3, PCV3, IPV
August 30, 2018 Measles
Lips, nail beds, soles and palms Lighter colored palms, soles, lips and nail beds
Temperature 37.7
EARS
Ear canal opening Free of lesions, discharge of inflammation
NOSE
Shape, size and skin color Smooth, symmetric with same color as the face
MOUTH
Lips Pink, moist symmetric
Most of the digestive organs (like the stomach and intestines) are tube-like and contain the
food as it makes its way through the body.
The digestive system is essentially a long, twisting tube that runs from the mouth to the anus,
plus a few other organs (like the liver and pancreas) that produce or store digestive chemicals.
The start of the process - the mouth: The digestive process begins in the mouth. Food is partly
broken down by the process of chewing and by the chemical action of salivary enzymes (these
enzymes are produced by the salivary glands and break down starches into smaller molecules).
On the way to the stomach: the esophagus - After being chewed and swallowed, the food
enters the esophagus. The esophagus is a long tube that runs from the mouth to the stomach.
It uses rhythmic, wave-like muscle movements (called peristalsis) to force food from the throat
into the stomach. This muscle movement gives us the ability to eat or drink even when we're
upside-down.
In the stomach - The stomach is a large, sack-like organ that churns the food and bathes it in a
very strong acid (gastric acid). Food in the stomach that is partly digested and mixed with
stomach acids is called chyme.
In the small intestine - After being in the stomach, food enters the duodenum, the first part of
the small intestine. It then enters the jejunum and then the ileum (the final part of the small
intestine). In the small intestine, bile (produced in the liver and stored in the gall bladder),
pancreatic enzymes, and other digestive enzymes produced by the inner wall of the small
intestine help in the breakdown of food.
In the large intestine - After passing through the small intestine, food passes into the large
intestine. In the large intestine, some of the water and electrolytes (chemicals like sodium) are
removed from the food. Many microbes (bacteria like Bacteroides, Lactobacillus acidophilus,
Escherichia coli, and Klebsiella) in the large intestine help in the digestion process. The first part
of the large intestine is called the cecum (the appendix is connected to the cecum). Food then
travels upward in the ascending colon. The food travels across the abdomen in the transverse
colon, goes back down the other side of the body in the descending colon, and then through
the sigmoid colon.
The end of the process - Solid waste is then stored in the rectum until it is excreted via the
anus.
VI. PATHOPHYSIOLOGY
If untreated
Hypovolemic Shock
Death
VII. LABORATORY AND DIAGNOSTIC EXAM
NAME: A.D.C AGE: 2 y/o ROOM: Pedia Ward
URINALYSIS
Transparenc SI turbid Increase urine SI turbid Increase urine Clear Normal Clear Normal
y concentration concentration
Sugar (#) Increase sugar (-) Normal (-) Normal (-) Normal
03/05/20 Interpretation
Color Green Sign of diarrhea
Consistency Soft Sign of diarrhea
GENERIC NAME:
ONDANSETRON
CLASSIFICATION:
5-HT3 antagonist
IX. COURSE IN THE WARD
On the first day, March 9, 2020, IVF was changed to #2 D5 IMB 500 ml x
10cc/hr at 9:50 am. He was seen by his Physician at 11:15 am and given an order
of urinalysis and fecalysis. He was prescribed with Ampicillin 50 mg IV every 6
hours. Also, Zinc Sulfate (E-Zinc) drops (0.6 ml) once daily was ordered. His fever
decreases gradually until there administration of paracetamol every 4 hours for
fever was discontinued. His vital signs were monitored and recorded.
Diarrhea
Dependent:
1. To
decrease
GI
motility
and
minimize
fluid
losses
2. To treat
infectiou
s
process,
decrease
motility
and
absorb
water
XI. DISCHARGE PLAN
M-edication
Upon discharge a client was advised to continue intake of Zinc-Sulfate (E-zinc) drops 0.6
ml once a day.
E-nvironment
T-reatment
A.D.C. was still advised for increase fluid intake, periodic complete emptying of urinary
bladder and keep hands clean.
Follow up checkup after 1 week of discharge.
H-ealth Teaching
Diet – Client was ordered with diet for age, with increase fluid intake.