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Identify Non-Modifiable and Modifiable Risk Factors For Hypertension
Identify Non-Modifiable and Modifiable Risk Factors For Hypertension
Identify Non-Modifiable and Modifiable Risk Factors For Hypertension
3. How will you decrease the workload of the heart of the patient with heart disease?
Important lifestyle changes may include:
Quitting smoking
Maintaining or losing weight
Tracking your daily fluid intake
Avoiding or limiting alcohol
Avoiding or limiting caffeine
Eating a heart-healthy diet
Being physically active
Managing stress
Medications will include:
o Ace inhibitors (usually end in “pril”) – widen your blood vessels and reduce your heart’s
workload
o Angiotensin II receptor blocker (usually end in “sartan”) – widen your blood vessels
and reduce your heart’s workload
o Beta blockers (usually end in “lol”) – lower your blood pressure and slow your heart rate
to reduce the workload on your heart
o Anticoagulants or “blood thinners” – thin your blood, preventing blood clots
o Vasodilators – widen the blood vessels and reduce your heart’s workload
o Calcium channel blockers – lower blood pressure and slow your heart rate, helping to
reduce your heart’s workload
Diuretics or “water pills”– remove extra fluid from your body which helps to improve
o
symptoms like swollen legs
o Digoxin – helps your heart to pump more blood and slow your heart rate down
o Potassium or magnesium – if you are taking fluid tablets, your levels of potassium or
magnesium may decrease, so you may need to take supplements
o Statins (cholesterol-lowering medications) – statins control the level of cholesterol in
your blood, and very large studies have demonstrated that they reduce the risk of blood
vessel diseases, heart attacks and stroke.
4. Make a drug study of common medication use for heart disease.
NAME OF MECHANISM OF INDICATION CONTRAINDICATION ADVERSE NURSING INTERVENTION
MEDICAT ACTION REACTIONS
ION
Digoxin Inhibits sodium- Heart failure, CNS: Drug-induced arrhythmiasmay increase the severity of
Classificat potassium- paroxysmal Contraindicated fatigue,generalizedm heart failure andhypotension.
ion: activated supraventricu inpatients uscleweakness,agitati
Inotropic adenosine lar hypersensitive to drug on,hallucinations,
Antiarrhyt triphosphatase, tachycardia, and in those with CV:
hmic promoting atrial digitalis-induced arrhythmias,heart Before giving loading dose,obtain baseline data
Cardiacgl movement of fibrillation toxicity, ventricular block. (heartrate and rhythm, bloodpressure, and
ycoside calcium from and flutter fibrillation, or EENT: electrolytes)and ask patient about use of cardiac
extracellular to ventricular tachycardia yellow-green glycosides within theprevious 2 to 3 weeks.
intracellular unless caused by heart halosaround
cytoplasm and failure. visualimages,blurred
strengthening vision,light Loading dose is usuallydivided over the first 24hours
myocardial Patients with Wolff- flashes,photophobia, with approximatelyhalf the loading dose given inthe
contraction. Also Parkinson-White diplopia. first dose.
acts on CNS to syndrome unless the GI:
enhance vagal conduction accessory anorexia,nause
tone, slowing pathway has been
conduction pharmacologically or Before giving drug, takeapical-radial pulse for
through the SA surgically disabled. 1minute. Record and notifyprescriber of
and AV nodes significantchanges (sudden increase ordecrease in
Elderly patients and in pulse rate, pulsedeficit, irregular beats and,particularly,
those with acute MI, regularization of a previously irregularrhythm). If these
incomplete AV block, occur, checkblood pressure and obtain a12-lead ECG
sinus bradycardia,
PVCs, chronic
constrictive
pericarditis