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TABIJE, NCP (L07)
TABIJE, NCP (L07)
TABIJE, NCP (L07)
OF THE INTERVENTION
PROBLEM
Subjective: ‘’lagi Deficient fluid STG: after 3hrs of -Assess precipitating -These will provide STG: after 3 hrs of NI,
akong nauuhaw at volume is the nursing intervention, factors such as other baseline data for patient will have
nagtatae din’’ decreased the patient have illnesses, new onset education once with verbalized
intravascular, verbalized diabetes, or poor resolved understanding of
Objective: interstitial, and understanding of compliance with hyperglycemia. causative factors and
-elevated intracellular fluid. causative factors and treatment regimen. Urinary tract infection purpose of individual
temperature of 38.4 C This refers to purpose of individual and pneumonia are therapeutic
-sweating of the skin dehydration, water therapeutic the most common interventions and
-thirst loss alone without interventions and infections causing medications.
-exhaustion change in sodium. medications. -Assess skin turgor, DKA and HHNS
-weight loss mucous membranes, among older clients. LTG: After 2 days of NI,
-dry skin or mucous LTG: after 2 days of and thirst. -To provide baseline the patient will have
membrane nursing interventions, data for further maintained fluid volume
the patient shall have comparison. Skin at a functional level as
maintained fluid turgor will decrease evidenced by individual
volume at a functional and tenting may good skin turgor, moist
level as evidenced by occur. The oral mucous membrane and
Nursing diagnosis: individual good skin -Monitor hourly intake mucous membranes stable vital signs.
Risk for Deficient turgor, moist mucous and output. will dry, and the client
Fluid Volume r/t membrane and stable may experience
inadequate fluid intake vital signs. extreme thirst.
as evidence by poor -Monitor BP especially -Oliguria and anuria
skin turgor. for orthostatic results from reduced
hypotension. glomerular filtration
and renal blood flow.
-Decreased blood
may be manifested
-Monitor respirations, by a drop in systolic
e.g acetone breath, blood pressure and
kussmaul’s orthostatic
respirations. hypotension.
-Acetone breath is
due to the
breakdown of
acetoacetic acid.
Kussmaul’s
espiration (rapid and
shallow breathing)
represent a
-Monitor laboratory compensatory
studies like; mechanism by the
blood glucose respiratory buffering
levels, system to raise
serum ketones, arterial pH by
potassium, exhaling more
sodium, carbon dioxide.
blood urea nitrogen -Elevated ketones is
and creatinine. associated with DKA.
-Monitor ABG for
metabolic acidosis.
-Insert dwelling
urinary catheter as -Clients with DKA
indicated have metabolic
acidosis with arterial
bicarbonate level
less than 18 mEq/L,
-Administer fluid as and a pH less than
indicated: Isotonic 7.30.
solution (0.9% NaCl) -To provide accurate
as ordered by the measurement or
doctor. urinary output
-Administer anti- especially for clients
pyretic as prescribed with neurologic
by the doctor. bladder.
-To replace
electrolytes, fluid loss
and prevent
dehydration.