Professional Documents
Culture Documents
Hyperemesis Gravidarum (Case Study)
Hyperemesis Gravidarum (Case Study)
AJA19-0226
Certificate No.
Republic of the Philippines
EASTERN SAMAR STATE UNIVERSITY
College of Nursing Allied Sciences
Submitted by:
Joan Pedrosa
Ma. Fraila Cabato
BSN2
Submitted to:
Ammy Evilynda C. Cesista, RN, MAN
Instructor
Certificate No. AJA19-0226
Certificate No.
TABLE OF CONTENTS:
A, NURSING ABSTRACT
B, DEMOGRAPHIC PROFILE
C. NURSING HISTORY
D. GORDON’S TYPOLOGY OF 11 FUNCTIONAL HEALTH PATTERNS
E. PHYSICAL ASSESSMENT
F. VITAL SIGNS
G. LABORATORY RESULT
H. PATHOPHYSIOLOGY
I. DRUG STUDY
J. NURSING CARE PLAN
K. TEACHING PLAN
L. LEARNING FEEDBACK
M. REFERENCES
Certificate No. AJA19-0226
Certificate No.
A. CLINICAL ABSTRACT
Mrs. X, a 24 year old female was admitted in the OB ward last February 26, 2020, at
Eastern Samar Provincial Hospital with a diagnosis of PU 13 weeks AOG G1P0
Hyperemesis Gravidarum with mild Dehydration. She has been experiencing a severe
vomiting since the month of January and not able to focus on her work and decided to
resign due to her illness. She’s also been experiencing chest pain and weight loss.
B. DEMOGRAPHIC PROFILE
Name: Mrs. X
Age: 24
Sex: Female
Birthday: May 19, 1995
Nationality: Filipino
Religion: Roman Catholic
Address: Brgy. Bato Borongan City E. S
Father’s Name: xxx
Mother’s Name: xxx
Spouse Name: xxx
Occupation: None
Educational Attainment: High School level
Chief Complaint: Vomiting
Admitting Diagnosis: PU 13 weeks AOG G1P0 Hyperemesis
Gravidarum with mild Dehydration
Attending Physician: Leah Golda M. Domingo M.D.
Certificate No. AJA19-0226
Certificate No.
C. NURSING HISTORY
1. History of Present Illness
The patient’s chief complaint is uncontrolled vomiting for almost 2 months
already, either eating or drinking small amount of food and water triggers her to
vomit at any time of the day. She doesn’t know her last menstruation due to her
irregular period and based on ultrasound, November 25, 2019 revealed a positive
result.
During the 1st trimester, she had her 1st prenatal check-up at Bato
Borongan City but doesn’t have a baby book yet.
2. Past Health History
As stated by the patient, she had no history of any hospitalizations. She
has experience cough and colds during her early years.
3. Immunization
As stated by the patient, she doesn’t know if she had a complete
vaccination during her childhood years.
4. Family Health History
4.1 Father Side
According to the patient her father is hypertensive and a cigarette
smoker too.
4.2 Mother Side
According to the patient her mother’s side has a history of
hypertension.
5. Allergies
According to the patient she had neither allergy to food nor beverages and
has not taken food supplements in the past.
Certificate No. AJA19-0226
Certificate No.
3. Elimination Patterns
Before Hospitalization Mrs. X usually had a bowel elimination once a day
and did not specified how many times a day she urinates but she made
mentioned that she urinates frequently a day due to pregnancy and
according to the urinalysis her urine is yellow and slightly turbid.
Legends:
0 = complete independent
1 = requires use of equipment or device
2 = requires help from another person for assistance, supervision, or
teaching
3 = requires help from another person and equipment or device
4 = complete dependence
5. Sleep-Rest Pattern
Prior to Hospitalization, the patient stated that she’s having a difficulty of
sleeping because of severe vomiting and restlessness and during
hospitalization, the patient stated that she usually sleeps 3 hours a day
she doesn’t have a consistent hour of sleep because she’s staying in the
hospital.
Certificate No. AJA19-0226
Certificate No.
6. Cognitive Perceptual
Based on observation, Patient’s senses are all functioning, she is able to
read and write. She is aware to the environment, has a good perception,
responses very well and very cooperative.
8. Role-Relationship Patterns
According to the patient she lives in the house of her parents with his
husband and this is her first pregnancy and she has a good relationship to
her family and friends. She is not involved to any organizations in her
community.
E. PHYSICAL ASSESSMENT
Assessment Findings Actual Findings
Integumentary
Cornea
Tongue and floor of the Central position, pink but with Normal
mouth whitish
coating which is normal, with
veins
prominent in the floor of the
Certificate No. AJA19-0226
Certificate No.
mouth.
Mental Status
Motor Function
F. VITAL SIGNS
Area Assessed Technique Used Normal Findings Actual Findings
Temperature Measured using 36.5 - 37.5 36.6 C
Thermometer
Pulse Rate Palpation 60- 100 bpm 92 bpm
G. LABORATORY FINDINGS:
A. HEMATOLOGY
TEST NAME RESULT NORMAL ANALYSIS
VALUES
Hgb 150 g/L 135-180g/L
Hct 0.64 0.40-0.54
WBC 6.7x10 3 /mm 3 4.5-11.0x10 3 /mm Normal
3
Diffeential
Count
Neutrophils .66 0.55-.70 Normal
Lymhocytes 0.20-0.40 Normal
Basophils
Monocytes
Eosinophil 0.01-0.04 Normal
Stab
CT
BT
Platelet
Count
B. URINALYSIS
Certificate No. AJA19-0226
Certificate No.
H. PATHOPHYSIOLOGY
TEST NAME RESULT NORMAL VALUES ANALYSIS
Bacteria Rare
Mucus
threads
Cast
Hyperemesis gravidarum refers to intractable vomiting during pregnancy that leads to
weight loss and volume depletion, resulting ketonuria and/or ketonemia. There is no
consensus on specific diagnostic criteria, but it generally refers to the severe end of the
spectrum regarding nausea and vomiting in pregnancy.
Pathophysiology
The exact cause of hyperemesis gravidarum remains unclear. However, there are
several theories for what may contribute to the development of this disease process.
Hormone Changes
Levels of human chorionic gonadotropin (hCG) have been implicated. hCG levels
peak during the first trimester, corresponding to the typical onset of hyperemesis
symptoms. Some studies show a correlation between higher hCG concentrations and
hyperemesis. However, this data has not been consistent.
Estrogen is also thought to contribute to nausea and vomiting in pregnancy. Estradiol
levels increase early in pregnancy and decrease later, mirroring the typical course of
Certificate No. AJA19-0226
Certificate No.
nausea and vomiting in pregnancy. Additionally, nausea and vomiting are the known
side effects of estrogen-containing medications. As the level of estrogen increases, so
does the incidence of vomiting.
Changes in the Gastrointestinal System
It is well-known that the lower esophageal sphincter relaxes during pregnancy due to
the elevations in estrogen and progesterone. This leads to an increased incidence of
gastroesophageal reflux disease (GERD) symptoms in pregnancy, and one symptom of
GERD is nausea. Studies examining the relationship between GERD and emesis in
pregnancy report conflicting results.
Genetics
An increased risk of hyperemesis gravidarum has been demonstrated among women
with family members who also experienced hyperemesis gravidarum.
Two genes, GDF15 and IGFBP7, have been potentially linked to the development of
hyperemesis gravidarum.
Evaluation
There is no single accepted definition for hyperemesis gravidarum. However, it
generally refers to the extreme cases of nausea and vomiting during pregnancy. It is a
clinical diagnosis. The criteria for diagnosis include vomiting that causes significant
dehydration (as evidenced by ketonuria or electrolyte abnormalities) and weight loss
(the most commonly cited marker for this is the loss of at least five percent of the
patient’s pre-pregnancy weight) in the setting of pregnancy without any other underlying
pathological cause for vomiting. Significant abdominal tenderness, pelvic tenderness, or
vaginal bleeding should prompt workup for alternative diagnoses.
Certificate No. AJA19-0226
Certificate No.
DRUG ACTION INDICATION CONTRAINDICATION ADVERSE EFFECTS NURSING RESPONSIBILITY
Binds to Treatment of History of Antibiotic-associated Baseline assessment
bacterial cell susceptible hypersensitivity/anaphylacti colitis, other Obtain CBC, renal function tests.
Cephalexin membranes, infections due c reaction to superinfections Question for history of allergies,
500 mg Oral inhibits cell to cephalexin, cephalosporins. (abdominal cramps, particularly
wall staphylococci, Cautions: Renal severe cephalosporins, penicillins.
PHARMACOTHERAPEUTIC: synthesis. group A impairment, history of GI watery diarrhea, Intervention/evaluation
First-generation Therapeutic streptococcus, disease fever) may result Assess oral cavity for white patches on
cephalosporin. Effect: K. pneumoniae, (esp. ulcerative colitis, from altered bacterial mucous membranes, tongue (thrush).
CLINICAL: Antibiotic. Bactericidal. E. coli, P. antibiotic-associated colitis), balance in GI tract. Monitor daily pattern of bowel activity,
mirabilis, H. history of penicillin allergy. Nephrotoxicity may stool consistency. Mild GI effects may
influenzae, M. occur, esp. in pts be tolerable (increasing severity may
catarrhalis, with preexisting renal indicate onset of antibiotic-associated
including disease. Pts with colitis). Monitor I&O, renal function tests
respiratory tract, history of penicillin for nephrotoxicity. Be alert for
genitourinary allergy is at superinfection: fever, vomiting, diarrhea,
tract, skin, soft increased risk for anal/genital pruritus, oral mucosal
tissue, bone developing a severe changes (ulceration, pain, erythema).
infections; otitis hypersensitivity With prolonged therapy, monitor
media; reaction (severe renal/hepatic function tests.
rheumatic fever pruritus, Patient/family teaching
prophylaxis; angioedema, • Doses should be evenly spaced.
follow-up to bronchospasm, • Continue therapy for full length of
parenteral anaphylaxis). treatment.
therapy. • May cause GI upset (may take with
OFFLABEL: food, milk).
Suppression of • Refrigerate oral suspension.
prosthetic joint • Report persistent diarrhea.
infection.
DRUG ACTION INDICATION CONTRAINDICATI ADVERSE NURSING RESPONSIBILITY
ON EFFECTS
Ampicillin inhibits Treatment of susceptible Hypersensitivity to Antibiotic-associated Baseline assessment
bacterial cell wall infections due to ampicillin, any colitis, other Question for history of allergies,
Ampicillin synthesis. streptococci, S. penicillins, or superinfections esp. penicillins, cephalosporins;
1 g q8h Sulbactam inhibits pneumoniae, sulbactam. (abdominal cramps; renal
bacterial staphylococci (non– Hx of cholestatic severe, impairment.
beta-lactamase. penicillinase-producing), jaundice, hepatic watery diarrhea; Intervention/evaluation
CLASSIFICATION Therapeutic meningococci, Listeria, impairment fever) may result Promptly report rash (although
PHARMACOTHERAPE Effect: Ampicillin is some associated with from altered bacterial common with ampicillin, may
UTIC: Penicillin. bactericidal in Klebsiella, E. coli, H. ampicillin/sulbactam balance in GI tract. indicate
CLINICAL: Antibiotic. susceptible influenzae, Salmonella, . Cautions: History Severe hypersensitivity) or diarrhea (fever,
microorganisms. Shigella, including GI, of allergies, esp. hypersensitivity abdominal pain, mucus and blood
Sulbactam protects GU, cephalosporins; reactions, including in stool
ampicillin from respiratory infections, renal anaphylaxis, acute may indicate antibiotic-associated
enzymatic meningitis, endocarditis impairment; interstitial colitis). Evaluate IV site for
degradation. prophylaxis. OFFLABEL: infectious nephritis, blood phlebitis. Check
Surgical prophylaxis for mononucleosis; dyscrasias may IM injection site for pain, induration.
liver transplantation. asthmatic pts. occur. High dosage Monitor I&O, urinalysis, renal
may produce function
seizures. tests. Be alert for superinfection:
fever, vomiting, diarrhea,
anal/genital pruritus,
oral mucosal changes (ulceration,
pain, erythema).
Patient/family teaching
• Take antibiotic for full length of
treatment.
• Space doses evenly.
• Discomfort may occur with IM
injection.
• Report rash, diarrhea, or other
new symptoms.
DRUG ACTION INDICATION CONTRAINDICATION ADVERSE NURSING
EFFECTS RESPONSIBILITY
Inhibits histamine Short-term treatment of Hypersensitivity to Reversible Baseline assessment
Apo-RaNITIdine action at active duodenal ulcer. raNITIdine. OTC: Do not hepatitis, Obtain history of
IV histamine H2- Prevention of duodenal use if trouble blood epigastric/abdominal
receptors of ulcer or pain when swallowing dyscrasias pain.
PHARMACOTHERAPEUTIC gastric parietal recurrence. Treatment of food, vomiting with occur rarely. Intervention/evaluation
: Histamine H2-receptor cells. active benign gastric blood, or bloody or black Assess mental status in
antagonist. Therapeutic ulcer, pathologic GI stool is elderly. Question present
CLINICAL: Antiulcer. Effect: Inhibits hypersecretory conditions, present. Do not use 150 abdominal pain, GI
gastric acid acute gastroesophageal mg with kidney disease distress.
secretion. reflux disease (GERD), (unless medically Patient/family teaching
Reduces gastric including erosive advised). • Smoking decreases
volume, esophagitis. Maintenance Cautions: Renal/hepatic effectiveness of
hydrogen ion of healed erosive impairment, elderly pts, medication.
concentration. esophagitis. OTC: history of acute • Do not take medicine
Relief of heartburn, acid porphyria. within 1 hr of
indigestion, sour stomach. magnesium- or
OFF-LABEL: Treatment aluminum-containing
of antacids.
upper GI bleeding. • Transient
Prevention of stress- burning/pruritus may
induced ulcers in ICU. occur with IV
Anaphylaxis administration.
(adjunct therapy). • Report headache.
Premedication to prevent • Avoid alcohol, aspirin.
taxane hypersensitivity.
B. Nursing Observations
During our duty, the ward was kept clean. Every now and then, there
would be new patients being admitted. The patients seemed uncomfortable with
oyur presence but they fe
C. Personal Reaction
We feel very happy for those patients because we know that they are also
happy that they already had the operations and others, finally, will be having their
operation. We love how the doctors and nurses from the Bulig Kablas Medical
Mission treated the patients because we know they are trying their best to
understand patients and assist their health needs. Were so amazed on how they
have their rounds because we can see that they really have the heart to help
patients. Whenever these people smiles, patients also smiles which makes the
ward a very busy yet happy place to us, to the client’s SO and to post-operative
patients.
D. Learning Insights
While on our duty, we learned that we should not just sought to help
patients just for personal experience of handling patients but also we should be
able to understand and acknowledge patients’ stories, feelings and have
empathy for them. It is really important that we establish not only to our individual
patients but also to their significant others, who, just like us give care to our
patients. Being with the patients, taking care of them, and understanding them is
really a great responsibility that we, student nurses, should be very patient with.
We should have a good communication skills not only for our own good but also
for the patients, for them to be open with us and render them the appropriate
service that they need while in the facility.
E. Recommendation
We are hoping that the facility will always give their best of care to patients in
order for them to fully recover from their illnesses. It will also be good to the patients
if the facility’s surrounding will be maintained for patient’s comfort and faster
recovery.
M. REFERENCES:
Barbara Kozier, MN, RN, and et al; Fundamental of Nursing, Concepts, Process,
and Practice, 8th Edition; Pearson Prentice Hall, Upper Saddle River, New Jersey
07458