Heart and Neck Vessels

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James 1:5-6

Philippians 4:13
HEART AND NECK VESSELS • The purpose of the cardiovascular
health history is to provide
• Cardiovascular disease is the every information about your patient’s
State’s leading killer for both men cardiovascular symptoms and how
and women among all racial and they developed. A complete
ethnic groups. cardiovascular history will give you
• A thorough cardiovascular indications to potential or
assessment will help to identify underlying cardiovascular illnesses
significant factors that can influence or disease states.
cardiovascular health such as high
blood cholesterol, cigarette use, PAST HEALTH HISTORY:
diabetes, or hypertension. - hypertension,
- elevated blood cholesterol or
CENTRAL VENOUS PRESSURE (CVP) triglycerides,
- It is the venous pressure as - heart murmurs,
measured at the right atrium, done - congenital heart disease,
by means of a catheter introduced - rheumatic fever or unexplained joint
through the median cubital vein to pains
the superior vena cava.
LIFESTYLE AND PSYCHOSOCIAL STATUS
BLOOD PRESSURE • Nutrition (usual food intake) – more
• It is the amount of force (pressure) detrimental than smoking and
that blood exerts on the walls of the alcohol
blood vessels as it passes through • Smoking (how many packs / day;
them. how long)
• ARTERIES RECEIVE MORE PRESSURE, • Alcohol
HUS, THE TUNICA MEDIA IS • Exercise
GREATER THAN • Drugs
• Family History
SYSTOLIC PRESSURE
• The blood pressure measured during ASSESSMENT ARTICLES:
the period of ventricular • A Double-Headed, Double-Lumen
contraction (systole). In blood Stethoscope
pressure readings, it is the higher of • A Blood Pressure Cuff
the two measurements • A Moveable Light Source or Pen
Light
DIASTOLE • Sphygmomanometer
• The period between contractions of • Measure tap
the atria or the ventricles during • Wrist watch and pen
which blood enters the relaxed
chambers from the systemic GENERAL INSPECTION
circulation and the lungs. EYES:
• The blood pressure (as measured by • The presence of yellowish plaques
a sphygmomanometer) after the on the eyelids (xanthelasma) could
contraction of the heart while the indicate hyperlipoproteinemia, a risk
chambers of the heart refill with factor for hypertension as well as
blood. arteriolosclerosis.
CHEST:
HEAD TO SHOULDER – Superior Vena Cava • Observe the chest for overall torso
SHOULDER GOING DOWN – inferior vena contour.
cava • Do you see pectus excavatum
(caved-in chest)?
Sinoatrial node – natural pacemaker of the • Do you see pectus carinatum
heart (pigeon chest)?
Atrioventricular Node – SKIN:
*should be fast, if slow, pt. is bradycardic • Clubbing - The presence of clubbing
(broadening of the extremities of
HISTORY: the digits, accompanied by nails
James 1:5-6
Philippians 4:13
which are abnormally curved and • S1, the “lub” of the “lub-dub,” is
shiny) indicates chronic poor oxygen produced by the closure of tricuspid
perfusion to the distal tissues of the and mitral valves.
hand and feet. • S1 is accentuated in exercise,
• Cyanosis - The presence of cyanosis anemia, hyperthyroidism, and mitral
(bluish colour) also denotes chronic stenosis.
poor oxygen delivery to the • S1 is diminished in first degree heart
peripheral tissues of the hands and block. (Arrythmia)
feet. • S1 split is most audible in tricuspid
• Xanthomas - The presence of area (T-lub-dub)
yellowish plaques under the skin
(non-eruptive) excoriated through S2
the skin (eruptive) could indicate • S2, the “dub” of the “lub-dub,” is
hyperlipoproteinemia, a risk factor produced by the closure of aortic &
for hypertension as well as pulmonic valves.
arteriolosclerosis. • Normal physiological splitting of S2
• Edema – (Bipedal E. – both feet) - is best heard at pulmonic area. It
The presence of edema (tissue occurs on inspiration(“lub-T-dub,
swelling) can be caused by several lub-dub”).
factors, although most commonly is • Splitting of S2 can indicate pulmonic
associated with decreased cardiac stenosis, atrial septal defect, right
function leading to decreased ventricular failure, and left bundle
capillary flow.  branch block

• Palpation S3
- Use the palm of your hand to feel • S3 is also known as a ventricular
the chest wall for the "Point of gallop (“lub-DUB-ta”). S3 is heard in
Maximal Impulse" (PMI), which is early diastole.
usually found at the apex of the • It is normal in pregnancy, children,
heart. This apical pulse is generally adults less than thirty years old,
located in the 5th intercostal space, during exercise, anxiety, or anemia.
about 7-9 cm (the width of your • It is heard best at the apex in the
palm) to the left of the midline. left lateral decubitus position, using
the bell. Pathologic S3 occurs in
Rate, rhythm, elasticity – should be noted people over the age of 40, usually
in the pulses due to myocardial failure.
*brachial, radial, femoral, popliteal,
dorsalis pedis, and posterior tibial. Note S4
the contour and amplitude of each • S4 is also known as an atrial gallop
pulsation. These should feel similar (“ta-lub-DUB”). It is typically heard
bilaterally. in late diastole before S1. It results
when ventricular resistance to atrial
Chest percussion: filling is increased from either
• Normally only the left border of decreased ventricular compliance or
heart can be detected by percussion. increased ventricular volume
It extends from the sternum to mid • HEARD BEST IN PT. WITH
clavicular line in the third to fifth HYPERTENSION
inter costal space.
• The right border lies under the right Summation Gallop
margin of the sternum and is not • A summation gallop is produced
detectable. Enlargement of the when S3 & S4 merge into one
heart too either the left or right sound. It often occurs at rates
usually can be noted. greater than 100 beats per minute.
It may occur in heart failure and
HEART SOUNDS: pericarditis.
S1 • Summation gallops occur in 15% of
all myocardial infractions
• ON TACHYCARDIC PATIENTS
James 1:5-6
Philippians 4:13
Opening Snap
• At the end of ventricular systole, NECK VESSELS:
when the aortic and pulmonic valves  Keep neck in a neutral position
close, S2 is produced Immediately  Locate and Palpate Carotid Arteries
after S2, the heart relaxes, and – One at a time
ventricular pressure falls below that – Rate amplitude 0 to 3+
of atrial pressure. This allows the
atrioventricular valves to open. This
is the start of diastole.

Ejection Click
• Similar to an opening snap, an
ejection click is caused by stenotic
valve leaflets. This sound is
produced when the aortic or
pulmonic valves open at the  Auscultate the Carotid-normally no
beginning of systole. sound
• It is a brief high frequency sound
best heard with the diaphragm over – Bruit- blowing or swishing
the aortic or pulmonary artery or sound indicating turbulent blood
Erb’s point, or near the apex over flow
the mitral area
 Use bell of
stethoscope
Mid-systolic Click
 Auscultate at 3
• A mid-systolic click occurs when the positions (down, middle, up)
mitral valve’s leaflets and cordae
tendenae (responsible for opening – Thrill – Palpable vibration
of the valves) tense. The anterior or accompanying bruit
posterior or both leaflets can
prolapse. Every once in a while  A loud aortic heart murmur may
multiple clicks occur. radiate to neck
• They are heard in mid to late systole.
They are best heard over the Jugular Venous Assessment
tricuspid area and towards the  Position client supine at 30 to 45
mitral area. degree angle (Semi-Fowler)
– Always be aware of client
Pericardial Friction Rub comfort
• A pericardial friction rub is usually – Turn client’s head slightly
heard best and is sometimes away
palpable over the tricuspid and  Note the external and internal
xyphoid areas. It occurs when Jugular vein distention (if any) and
inflamed pericardial surfaces rub record the level in relation to the
together. clavicle (normal less than 2 cm)
– Observe for pulsations (if
Murmurs any)
• A murmur is an abnormal heart – Unilateral distension
sound caused by turbulent blood (embossed) = kinking or
flow. The sound may indicate that aneurysm (ballooning of the
blood is flowing through a damaged blood vessel)
or overworked heart valve (usually – Bilateral distension =
needs to have an open heart increased CVP
operation), that there may be a hole
in one of the heart's walls, or that
there is a narrowing in one of the
heart's vessels

ASSESSMENT:
HISTORY:
James 1:5-6
Philippians 4:13
– Normal phenomenon
The Precordium (chest wall) – Occurs at end of inspiration
 Inspect
– Heave(lift) = sustained – Semilunar valves don’t close
forceful thrust of ventricle at the same time
against chest wall = – Heard best at left 2nd ICS
ventricular hypertrophy – Can be fixed or paradoxical
– Apical pulse (palm) may be  S3 – Ventricular Gallop
visible in thin adults and – Early in diastole during rapid
children filling
– Heard best at apex using bell
 Palpate the Apical Pulse of stethoscope
– Sometimes called PMI (point – Doesn’t vary /c resp like Split
of maximum impulse) S2
Note: – Indicates decreased
 Location ventricular compliance
– should be at – In children and young adult
th
or near 5 intercostals space may be innocent and
– LV dilatation disappear when pt sits
(fluid overload) displaces down – May be earliest sign of heart
and to the left and increases size failure
 Size – Heard /c increased CO i.e .
 Amplitude- 0 to 3+ hyperthyroidism
– Increased in  S4- Atrial Gallop
LV hypertrophy (pressure – Ventricular filling sound
overload) – Occurs late in diastole
 Rate and Rhythm immediately before S1
– Regular – Heard when atria contract
irregularity or irregular – Very soft, low pitched sound
irregularity – Heard best at apex /c pt in
– Compare left lateral position /c bell
irregular apical pulse to radial – Can occur /p exercise at 40 yr
– Sinus old
arrhythmia common in children – Occurs /c systolic overload,
and young adults related to the hypertension and aortic
respiratory cycle stenosis
– Premature  Friction Rub
Beat - more common in elderly – Occurs r/t inflammation of
 Palpate the Precordium the pericardial membranes
– Use palm – Occur in both systole and
– Normally thrill or mass not diastole
felt – Hear best at apex
– Common in 1st week
AUSCULTATION following Myocardial
 Use Diaphragm of Stethoscope Infarction and pericarditis
 Identify S1 and S2
– S1 is loudest at apex
 closure of AV valves
 Beginning of systole
– S2 is loudest at base
 Closure of semilunar
valves
 Beginning of diastole
– S1 coincides /c carotid pulse
and R wave on ECG

EXTRA HEART SOUNDS:


 Split S2
James 1:5-6
Philippians 4:13

Murmurs
 Blowing, swooshing sound
 Indicates abnormal turbulent blood
flow
 A murmur outside the heart is called
a bruit
 Are either systolic or diastolic
 Systolic murmur may occur
innocently in children and young
adults r/t increased force of
contraction

Assessment of Murmurs
 Timing- systolic or diastolic
 Loudness-Grade I thru VI
 Pitch- high or low
 Pattern- Crescendo, decrescendo,
plateau, diamond
 Quality- Musical, blowing, harsh or
rumbling
 Location- Where is it loudest?
 Radiation- Is it audible in other parts
of precordium
 Posture- Is it present or louder only
in certain position

Age Specific Considerations


 Infants
– Use appropriate size
stethoscope
– May be irregular
– Murmurs may be present r/t
congenital fetal circulation
remnants
 Children
– May have visible apical
pulses r/t thin chest wall
– May have innocent
murmurs-always note
presence of murmur
 Pregnancy
– Increased pulse rate
– Exaggerated S2 splitting
– Easily heard S3 (normal)
– Systolic murmur may be
present-should disappear /p
delivery
 Elderly
– S4 even /s Hx of CAD
– Irregular pulse more
common

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