Non- Modifiable Risk Factor: Modifiable Factor: 3.

Fond eating noodles

Impaired cerebral tissue
 Lost consciousness and cannot move his 
 Name: JB perfusion Hypertensive 4. High Salt diet
whole body (2005)
 Male  Takes ani-hypertensive medications
 Underwent therapy (for 1 month)  Poor health
 61 years old (Haemorrhagic)  Diet
 Family history: Parent died with management: No
 Filipino 1. 4 cups of rice
hypertension. Siblings are hypertensive. exercise and stress
 Stroke(2005) 2. 1 lite of water a day
no TB, DM, and mental illness. from work

Intracerebral
TYPE TYPE Subarachnoid
hemorrhage
hemorrhage
160/90
mmHg Lying URINALYSIS CLINDAMYCIN
Build Up of Rupture of a 600mg cap q6h
plaque Blood Vessels
deposits in within abnormal Uncontrolled
BLOOD CHEMISTRY
walls of bleeding into Hypertension
blood vessels area of
small CBC ELEVATED Rupture of
extravascular
arteries brain WBC saccular/ Berry
Impaired Physical Mobility Aneurysm
damaged
Bleeding into CLONIDINE Membrane
CEFTAZIDIME FELODIPINE depolarization LEGENDS:
brain tissue 75mcg tab 5mg tab OD Predisposing &
500 mg IV q8h occurs
OD ETIOLOGY Precipitating
Stages vessel all
Cytotoxic
is disrupted MEDICATIONS
Space- occupying edema
May lead to Result in influx of
and cell Local acidosis
blood clots, put temporary calcium and sodium Disease
dead [2nd occurs
more pressure on neurologic Process SIGN &
neuronal
the brain. deficit SYMPTOMS
injury Drowsy to
LEAKAGE KCl 500mEqs in PNSS Stuporous
1L. to run for 1 hrs
TYPES
MECHANISM
The regulatory mechanisms of the brain attempt to Cells in the centre of the stoke area, or A cascade of
MANNITOL
maintain equilibrium by increasing Blood Pressure the core die almost immediately after biochemical process
150cc IV and Intracellular pressure occurs within minute LABORATORY
the stroke onset [primary neuronal NX DIAGNOSIS
q4h of cerebral ischemia
injury}