Path

DIAGNOSTIC REPORT
CLIENT CODE : C000082254 Cert. No. M-0255
CLIENT'S NAME AND ADDRESS : SRL LIMITED

CALLHEALTH SERVICES PVT LTD LEGEND CRYSTAL,SHOP NO-6,GROUND & 1ST FLOOR,PLOT NO-1-7-
10th FLOOR,RAMKY TOWERS, SURVEY No:136/2 & 4GA CHIBOWLI, 79/A B:,PRENDERGHAST ROAD
MIND SPACE ROAD, DIST:-RANAGREDDY, SECUNDERABAD, 500003
HYDERABAD 500032 TELANGANA, INDIA
TELANGANA INDIA Tel : 040-61176117/48560048 HOME COLLECT NO-040-48542566,
40-33557799 CIN - U74899PB1995PLC045956
Email : customercare.hyderabad@srl.in
PATIENT NAME : BOOBALAN B PATIENT ID : CH.8931182
ACCESSION NO : 0042TE000975 AGE : 29 Years SEX : Male DATE OF BIRTH : 23/08/1990
DRAWN : 14/05/2020 10:44 RECEIVED : 14/05/2020 15:18 REPORTED : 14/05/2020 17:19
REFERRING DOCTOR : DR. CALL HEALTH CLIENT PATIENT ID : 8931182
CLINICAL INFORMATION :
UHID: 8931182 REQNO-122301529

OPD-HYD
Test Report Status Final Results Biological Reference Interval Units
HDFC BIOCHEMISTRY
LIVER FUNCTION PROFILE, SERUM

BILIRUBIN, TOTAL 2.64 High 0.2 - 1.0 mg/dL
METHOD : SPECTROPHOTOMETRY, JENDRASSIK & GROFF
BILIRUBIN, DIRECT 0.33 High 0.0 - 0.2 mg/dL

METHOD : SPECTROPHOTOMETRY, JENDRASSIK & GROFF
BILIRUBIN, INDIRECT 2.31 High 0.1 - 1.0 mg/dL

METHOD : SPECTROPHOTOMETRY,CALCULATED
TOTAL PROTEIN 7.6 6.4 - 8.2 g/dL

METHOD : SPECTROPHOTOMETRY, MODIFIED BIURET
ALBUMIN 4.2 3.4 - 5.0 g/dL

METHOD : SPECTROPHOTOMETRY, BCP - DYE BINDING
GLOBULIN 3.4 2.0 - 4.1 g/dL

ALBUMIN/GLOBULIN RATIO 1.2 1.0 - 2.1 RATIO

ASPARTATE AMINOTRANSFERASE (AST/SGOT) 30 15 - 37 U/L

METHOD : SPECTROPHOTOMETRY, UV WITH PYRIDOXAL -5-PHOSPHATE
ALANINE AMINOTRANSFERASE (ALT/SGPT) 62 High < 45.0 U/L

METHOD : SPECTROPHOTOMETRY, UV WITH PYRIDOXAL -5-PHOSPHATE
ALKALINE PHOSPHATASE 71 30 - 120 U/L

METHOD : SPECTROPHOTOMETRY, P-NPP (AMP BUFFER)
GAMMA GLUTAMYL TRANSFERASE (GGT) 42 15 - 85 U/L

METHOD : SPECTROPHOTOMETRY, G-GLUTAMYL-CARBOXY-NITRONILIDE
LACTATE DEHYDROGENASE 206 High 100 - 190 U/L

METHOD : SPECTROPHOTOMETRY, MODIFIED ENZYMATIC LACTATE - PYRUVATE
CREATININE, SERUM
CREATININE 0.81 Low 0.90 - 1.30 mg/dL
METHOD : SPECTROPHOTOMETRY, ALKALINE PICRATE KINETIC JAFFE'S
SERUM BLOOD UREA NITROGEN

BLOOD UREA NITROGEN 9 6 - 20 mg/dL
METHOD : SPECTROPHOTOMETRY, UREASE UV
GAMMA GLUTAMYL TRANSFERASE, SERUM
Page 1 Of 9
DIAGNOSTIC REPORT
CLIENT CODE : C000082254 Cert. No.

40-33557799 CIN - U74899PB1995PLC045956
UHID: 8931182 REQNO-122301529

OPD-HYD
GAMMA GLUTAMYL TRANSFERASE (GGT) 42 15 - 85 U/L

METHOD : SPECTROPHOTOMETRY, G-GLUTAMYL-CARBOXY-NITRONILIDE
Interpretation(s)
LIVER FUNCTION PROFILE, SERUM-
LIVER FUNCTION PROFILE
Bilirubin is a yellowish pigment found in bile and is a breakdown product of normal heme catabolism. Bilirubin is excreted in bile and urine, and elevated levels may give
yellow discoloration in jaundice.Elevated levels results from increased bilirubin production (eg, hemolysis and ineffective erythropoiesis), decreased bilirubin excretion (eg,
obstruction and hepatitis), and abnormal bilirubin metabolism (eg, hereditary and neonatal jaundice). Conjugated (direct) bilirubin is elevated more than unconjugated
(indirect) bilirubin in Viral hepatitis, Drug reactions, Alcoholic liver disease Conjugated (direct) bilirubin is also elevated more than unconjugated (indirect) bilirubin when
there is some kind of blockage of the bile ducts like in Gallstones getting into the bile ducts, tumors &Scarring of the bile ducts. Increased unconjugated (indirect) bilirubin
may be a result of Hemolytic or pernicious anemia, Transfusion reaction & a common metabolic condition termed Gilbert syndrome, due to low levels of the enzyme that
attaches sugar molecules to bilirubin.
AST is an enzyme found in various parts of the body. AST is found in the liver, heart, skeletal muscle, kidneys, brain, and red blood cells, and it is commonly measured
clinically as a marker for liver health. AST levels increase during chronic viral hepatitis, blockage of the bile duct, cirrhosis of the liver,liver cancer,kidney failure,hemolytic
anemia,pancreatitis,hemochromatosis. AST levels may also increase after a heart attack or strenuous activity.ALT test measures the amount of this enzyme in the blood.ALT
is found mainly in the liver, but also in smaller amounts in the kidneys,heart,muscles, and pancreas.It is commonly measured as a part of a diagnostic evaluation of
hepatocellular injury, to determine liver health.AST levels increase during acute hepatitis,sometimes due to a viral infection,ischemia to the liver,chronic hepatitis,obstruction
of bile ducts,cirrhosis.
ALP is a protein found in almost all body tissues.Tissues with higher amounts of ALP include the liver,bile ducts and bone.Elevated ALP levels are seen in Biliary obstruction,
Osteoblastic bone tumors, osteomalacia, hepatitis, Hyperparathyroidism, Leukemia, Lymphoma, Paget's disease,Rickets,Sarcoidosis etc. Lower-than-normal ALP levels seen
in Hypophosphatasia,Malnutrition,Protein deficiency,Wilson's disease.GGT is an enzyme found in cell membranes of many tissues mainly in the liver,kidney and pancreas.It is
also found in other tissues including intestine,spleen,heart, brain and seminal vesicles.The highest concentration is in the kidney,but the liver is considered the source of
normal enzyme activity.Serum GGT has been widely used as an index of liver dysfunction.Elevated serum GGT activity can be found in diseases of the liver,biliary system and
pancreas.Conditions that increase serum GGT are obstructive liver disease,high alcohol consumption and use of enzyme-inducing drugs etc.Serum total protein,also known as
total protein,is a biochemical test for measuring the total amount of protein in serum.Protein in the plasma is made up of albumin and globulin.Higher-than-normal levels may
be due to:Chronic inflammation or infection,including HIV and hepatitis B or C,Multiple myeloma,Waldenstrom's disease.Lower-than-normal levels may be due to:
Agammaglobulinemia,Bleeding (hemorrhage),Burns,Glomerulonephritis,Liver disease, Malabsorption,Malnutrition,Nephrotic syndrome,Protein-losing enteropathy etc.Human
serum albumin is the most abundant protein in human blood plasma.It is produced in the liver.Albumin constitutes about half of the blood serum protein.Low blood albumin
levels (hypoalbuminemia) can be caused by:Liver disease like cirrhosis of the liver, nephrotic syndrome,protein-losing enteropathy,Burns,hemodilution,increased vascular
permeability or decreased lymphatic clearance,malnutrition and wasting etc
CREATININE, SERUM-
Higher than normal level may be due to:
•Blockage in the urinary tract
•Kidney problems, such as kidney damage or failure, infection, or reduced blood flow
•Loss of body fluid (dehydration)
•Muscle problems, such as breakdown of muscle fibers
•Problems during pregnancy, such as seizures (eclampsia)), or high blood pressure caused by pregnancy (preeclampsia)
Lower than normal level may be due to:

•Myasthenia Gravis
•Muscular dystrophy
SERUM BLOOD UREA NITROGEN-
Causes of Increased levels
Pre renal
•High protein diet, Increased protein catabolism, GI haemorrhage, Cortisol, Dehydration, CHF Renal
•Renal Failure
Post Renal
•Malignancy, Nephrolithiasis, Prostatism
Causes of decreased levels

•Liver disease
• SIADH.
GAMMA GLUTAMYL TRANSFERASE, SERUM-
Gamma glutamyl transferase (GGT) is an enzyme found in cell membranes of many tissues mainly in the liver, kidney, and pancreas. It is also found in other tissues including
intestine, spleen, heart, brain, and seminal vesicles. The highest concentration is in the kidney, but the liver is considered the source of normal enzyme activity. Serum
gamma-glutamyl transferase (GGT) has been widely used as an index of liver dysfunction. Elevated serum GGT activity can be found in diseases of the liver, biliary system,
and pancreas .Conditions that increase serum GGT are obstructive liver disease, high alcohol consumption, and use of enzyme-inducing drugs etc.
Page 2 Of 9
DIAGNOSTIC REPORT

40-33557799 CIN - U74899PB1995PLC045956
UHID: 8931182 REQNO-122301529

OPD-HYD
HAEMATOLOGY
COMPLETE BLOOD COUNT, EDTA WHOLE

BLOOD/SMEAR
BLOOD COUNTS
HEMOGLOBIN 15.3 13.0 - 17.0 g/dL
METHOD : CYANMETHEMOGLOBIN METHOD
RED BLOOD CELL COUNT 5.04 4.5 - 5.5 mil/µL

METHOD : ELECTRICAL IMPEDANCE
WHITE BLOOD CELL COUNT 7.50 4.0 - 10.0 thou/µL

PLATELET COUNT 222 150 - 410 thou/µL

RBC AND PLATELET INDICES

HEMATOCRIT 43.9 40 - 50 %
METHOD : CALCULATED PARAMETER
MEAN CORPUSCULAR VOL 87.0 83 - 101 fL

MEAN CORPUSCULAR HGB. 30.3 27.0 - 32.0 pg

MEAN CORPUSCULAR HEMOGLOBIN 34.8 High 31.5 - 34.5 g/dL

CONCENTRATION
RED CELL DISTRIBUTION WIDTH 12.3 11.6 - 14.0 %

MEAN PLATELET VOLUME 9.3 6.8 - 10.9 fL

WBC DIFFERENTIAL COUNT

SEGMENTED NEUTROPHILS 45 40 - 80 %
METHOD : ACV TECHNOLOGY
ABSOLUTE NEUTROPHIL COUNT 3.38 2.0 - 7.0 thou/µL

EOSINOPHILS 5 1-6 %
ABSOLUTE EOSINOPHIL COUNT 0.38 0.02 - 0.50 thou/µL
Page 3 Of 9
DIAGNOSTIC REPORT

40-33557799 CIN - U74899PB1995PLC045956
UHID: 8931182 REQNO-122301529

OPD-HYD
LYMPHOCYTES 48 High 20 - 40 %
ABSOLUTE LYMPHOCYTE COUNT 3.60 High 1.0 - 3.0 thou/µL

MONOCYTES 2 2 - 10 %
ABSOLUTE MONOCYTE COUNT 0.15 Low 0.2 - 1.0 thou/µL

BASOPHILS 0 0-2 %
ABSOLUTE BASOPHIL COUNT 0 Low 0.02 - 0.10 thou/µL

DIFFERENTIAL COUNT PERFORMED ON: EDTA SMEAR

MORPHOLOGY
RBC NORMOCYTIC NORMOCHROMIC.
METHOD : MICROSCOPIC EXAMINATION
WBC
WITHIN NORMAL LIMITS.
PLATELETS ADEQUATE ON SMEAR.
IMPRESSION NORMAL PERIPHERAL SMEAR.

NO HAEMOPARASITES SEEN.
Interpretation(s)
BLOOD COUNTS-
The cell morphology is well preserved for 24hrs. However after 24-48 hrs a progressive increase in MCV and HCT is observed leading to a decrease in MCHC. A direct smear is
recommended for an accurate differential count and for examination of RBC morphology.
RBC AND PLATELET INDICES-
The cell morphology is well preserved for 24hrs. However after 24-48 hrs a progressive increase in MCV and HCT is observed leading to a decrease in MCHC. A direct smear is
recommended for an accurate differential count and for examination of RBC morphology.
BIO CHEMISTRY
GLUCOSE, FASTING, PLASMA
Page 4 Of 9
DIAGNOSTIC REPORT

40-33557799 CIN - U74899PB1995PLC045956
UHID: 8931182 REQNO-122301529

OPD-HYD
GLUCOSE, FASTING, PLASMA 93 74 - 99 mg/dL

METHOD : SPECTROPHOTOMETRY HEXOKINASE
CHOLESTEROL, SERUM
CHOLESTEROL 143 < 200 Desirable mg/dL

200 - 239 Borderline High
>/= 240 High
METHOD : SPECTROPHOTOMETRY,CHOLESTEROL OXIDASE ESTERASE PEROXIDASE
GLYCOSYLATED HEMOGLOBIN, BLOOD
GLYCOSYLATED HEMOGLOBIN (HBA1C) 5.9 High Non-diabetic: < 5.7 %

Pre-diabetics: 5.7 - 6.4
Diabetics: > or = 6.5
ADA Target: 7.0
Action suggested: > 8.0
METHOD : ION- EXCHANGE HPLC
MEAN PLASMA GLUCOSE 123 High < 116.0 mg/dL

METHOD : ION- EXCHANGE HPLC
Interpretation(s)
GLUCOSE, FASTING, PLASMA-
ADA 2012 guidelines for adults as follows:
Pre-diabetics: 100 - 125 mg/dL
Diabetic: > or = 126 mg/dL
(Ref: Tietz 4th Edition & ADA 2012 Guidelines)

CHOLESTEROL, SERUM-
Serum cholesterol is a blood test that can provide valuable information for the risk of coronary artery disease This test can help determine your risk of the build up of plaques
in your arteries that can lead to narrowed or blocked arteries throughout your body (atherosclerosis). High cholesterol levels usually don't cause any signs or symptoms, so a
cholesterol test is an important tool. High cholesterol levels often are a significant risk factor for heart disease and important for diagnosis of hyperlipoproteinemia,
atherosclerosis, hepatic and thyroid diseases.
GLYCOSYLATED HEMOGLOBIN, BLOOD-
GLYCOSYLATED HEMOGLOBIN, BLOOD
Glycation is nonenzymatic addition of sugar residue to amino groups of proteins. HbA1C is formed by the condensation of glucose with n-terminal valine
residue of each beta chain of hb a to form an unstable schif base. It is the major fraction, constituting approximately 80% of HbA1.
Formation of glycated hemoglobin (GHb) is essentially irreversible and the concentration in the blood depends on both the lifespan of the red blood cells (RBC)
(120 days) and the blood glucose concentration. The GHB concentration represents the integrated values for glucose aver the period of 6 to 8 weeks. GHb
values are free of day to day glucose fluctuations and are unaffected by recent exercise or food ingestion. Concentration of plasma glucose concentration in
GHb depends on the time interval, with more recent values providing a larger contribution than earlier values.
The interpretation of GHb depends on RBC having a normal life span. Patients with hemolytic disease or other conditions with shortened RBC survival exhibit a
substantial reduction of GHb. High GHb have been reported in iron deficiency anemia .
GHb has been firmly established as an index of long term blood glucose concentrations and as a measure of the risk for the development of complications in
patients with diabetes mellitus. The absolute risk of retinopathy and nephropathy are directly proportional to the mean of HbA1C.
EIA - INFECTIOUS SECTION
Page 5 Of 9
DIAGNOSTIC REPORT

40-33557799 CIN - U74899PB1995PLC045956
UHID: 8931182 REQNO-122301529

OPD-HYD
HEPATITIS B SURFACE ANTIGEN, SERUM
HEPATITIS B SURFACE ANTIGEN NON REACTIVE NON REACTIVE

METHOD : IMMUNOBLOT METHOD
* HIV 4TH GEN ASSAY (P24AG + HIV AB),

SERUM
HIV 4TH GEN ASSAY (P24AG + HIV AB) NON REACTIVE NON REACTIVE
METHOD : IMMUNOBLOT METHOD
Interpretation(s)
HEPATITIS B SURFACE ANTIGEN, SERUM-
Hepatitis B is caused by infection with HBV, a enveloped DNA agent that is classified as hepadnavirus.This test detects the presence of viral surface antigen (HbsAg) in serum
sample and is indicative of an active HBV infection, either acute or chronic.
Test Utility:
HbsAg is the first serologic marker appearing in the serum 6-16 weeks following hepatitis B viral infection. In typical HBV infection, HBsAg will be detected 2-4 weeks before
the liver enzyme levels (ALT) become abnormal and 3-5 weeks before patient develops jaundice.In acute cases HbsAg usually disappears 1-2 months after the onset of
symptoms.Persistence of HbsAg for more than 6 months indicates development of either a chronic carrier state or chronic liver disease.The presence of HbsAg is frequently
associated with infectivity. HbsAg when accompanied by Hepatitis Be antigen and/or hepatitis B viral DNA almost always indicates infectivity.
Limitations:
- For diagnostic purposes, results should be used in conjunction with patient history and other hepatitis markers for diagnosis of acute or chronic infection. If the antibody
results are inconsistent with clinical evidence, additional testing is suggested to confirm the result.
- HBsAg detection will only indicate the presence of surface antigens in the serum and should not be used as the sole criteria for diagnosis, staging or monitoring of HBV
infection This test may be negative during ""window period"" i.e. after disappearance of anti-HBs.
- The current assay being a highly sensitive test , may yield a small percentage of false positive reports. Hence all HbsAg positive specimens should be confirmed with an
assay based upon Neutralisation of Human anti Hepatitis B Surface antibody.
HIV 4TH GEN ASSAY (P24AG + HIV AB), SERUM-
Acquired immunodeficiency syndrome (AIDS) is caused by 2 types of human immunodeficiency viruses, collectively designated HIV. HIV is transmitted by sexual contact,
exposure to blood or blood products, and prenatal infection of a fetus or perinatal infection of a newborn.
Phylogenetic analysis classifies HIV-1 into groups M (major), N (non-M, non-O), and O (outlier).HIV-2 is similar to HIV-1 in its structural morphology, genomic organization,
cell tropism, in vitro cytopathogenicity, transmission routes, and ability to cause AIDS. However, HIV-2 is less pathogenic than HIV-1.HIV-2 infections have a longer latency
period with slower progression to disease, lower viral titers, and lower rates of vertical and horizontal transmission. HIV-2 is endemic to West Africa but HIV-2 infections, at a
low frequency compared to HIV-1, have been identified in the USA, Europe, Asia, and other regions of Africa. India predominantly has HIV-1M subtype C.
Test Util
ity;
The test used is the 4th generation assay that detects HIV 1 P24 antigen and HIV 1/ 2 antibodies. This leads to improved sensitivity and, therefore, a shorter diagnostic
window as compared to 3rd generation anti-HIV assays.
If HIV reactive result is obtained, confirmation of HIV antibody status is done using 2 more antibody tests (as per NACO guidelines-Strategy III algorithm). If indicated HIV
serostatus may be confirmed by repeating antibody test on fresh specimen or HIV-1 Western Blot (Immunoblot) Assay (SRL test code #3012) and HIV DNA detector (SRL
test code #9885B).
In case of HIV reactive by one method and non reactive by other two methods, results need to be correlated with the clinical history of the patient. As per NACO guidelines,
result may be interpreted as “ Negat i
ve”for low risk individuals and “
Indet ermi nate”in case of high risk individuals.
For tests reported/interpreted as ‘ Indet er
mi nate,testing should be repeated on a second fresh specimen after 14-28 days. In case the serological results continue to be
indeterminate, HIV Western blot /PCR is recommended.
Limitations:
•Antibody tests may give false negative results during the window period, an interval of 3 weeks to 6 months between the time of HIV infection and the production of
measurable antibodies to HIV seroconversion. Most people develop detectable antibodies approximately 30 days after infection, although some seroconvert later. The vast
majority of people (97%) have detectable antibodies by three months after HIV i nf
ect i
on;a 6-month window is extremely rare with modern antibody testing
•False negative results can occur in patients with X-linked agammaglobulinemia. False positive results can occur in autoimmune disorders, alcoholic hepatitis , multiple
pregnancies.
- Early antiretroviral therapy during the window period may alter antibody responses. This does not apply to individuals undergoing treatment with post-exposure prophylaxis
(PEP).
Page 6 Of 9
DIAGNOSTIC REPORT

40-33557799 CIN - U74899PB1995PLC045956
UHID: 8931182 REQNO-122301529

OPD-HYD
•A positive HIV result in an infant <18 months of age may not reflect the infant's HIV infection status.HIV antibodies persist in the sera of infants upto 18 months of age, due
to transplacentally acquired maternal antibodies. HIV PCR testing is recommended in this age group for diagnosis.
•The presence of HIV antigen or antibodies to HIV is not a diagnosis of AIDS.
•For diagnostic purposes, the results should always be assessed in conjunction with the pat ient’smedical history, clinical examination and other findings.
CLINICAL PATH
URINALYSIS
COLOR PALE YELLOW

METHOD : MANUAL
APPEARANCE CLEAR
METHOD : MANUAL
PH 5.5 4.7 - 7.5

METHOD : REFLECTANCE SPECTROPHOTOMETRY
SPECIFIC GRAVITY 1.025 1.003 - 1.035

GLUCOSE NOT DETECTED NOT DETECTED

PROTEIN NOT DETECTED NOT DETECTED

KETONES NOT DETECTED NOT DETECTED

BLOOD NOT DETECTED NOT DETECTED

BILIRUBIN NOT DETECTED NOT DETECTED

UROBILINOGEN NORMAL NORMAL

NITRITE NOT DETECTED NOT DETECTED

WBC 1-2 0-5 /HPF

EPITHELIAL CELLS 1-2 0-5 /HPF

RED BLOOD CELLS NOT DETECTED NOT DETECTED /HPF

CASTS NOT DETECTED
Page 7 Of 9
DIAGNOSTIC REPORT

40-33557799 CIN - U74899PB1995PLC045956
UHID: 8931182 REQNO-122301529

OPD-HYD
CRYSTALS NOT DETECTED

BACTERIA NOT DETECTED NOT DETECTED

Comments
NOTE : URINE MICROSCOPIC EXAMINATION IS CARRIED OUT ON CENTRIFUGED URINE SEDIMENT.
Interpretation(s)
URINALYSIS-Routine urine analysis assists in screening and diagnosis of various metabolic, urological, kidney and liver disorders
Protein: Elevated proteins can be an early sign of kidney disease. Urinary protein excretion can also be temporarily elevated by strenuous exercise, orthostatic proteinuria,
dehydration, urinary tract infections and acute illness with fever
Glucose: Uncontrolled diabetes mellitus can lead to presence of glucose in urine. Other causes include pregnancy, hormonal disturbances, liver disease and certain
medications.
Ketones: Uncontrolled diabetes mellitus can lead to presence of ketones in urine. Ketones can also be seen in starvation, frequent vomiting, pregnancy and strenuous
exercise.
Blood: Occult blood can occur in urine as intact erythrocytes or haemoglobin, which can occur in various urological, nephrological and bleeding disorders.
Leukocytes: An increase in leukocytes is an indication of inflammation in urinary tract or kidneys. Most common cause is bacterial urinary tract infection.
Nitrite: Many bacteria give positive results when their number is high. Nitrite concentration during infection increases with length of time the urine specimen is retained in
bladder prior to collection.
pH: The kidneys play an important role in maintaining acid base balance of the body. Conditions of the body producing acidosis/ alkalosis or ingestion of certain type of food
can affect the pH of urine.
Specific gravity: Specific gravity gives an indication of how concentrated the urine is. Increased specific gravity is seen in conditions like dehydration, glycosuria and
proteinuria while decreased specific gravity is seen in excessive fluid intake, renal failure and diabetes insipidus.
Bilirubin: In certain liver diseases such as biliary obstruction or hepatitis, bilirubin gets excreted in urine.
Urobilinogen: Positive results are seen in liver diseases like hepatitis and cirrhosis and in cases of hemolytic anemia
**End Of Report**
Dr. Gaurav Rastogi, MD

Sr.Pathologist
Page 8 Of 9
DIAGNOSTIC REPORT

40-33557799 CIN - U74899PB1995PLC045956
UHID: 8931182 REQNO-122301529

OPD-HYD
CONDITIONS OF LABORATORY TESTING & REPORTING

1. It is presumed that the test sample belongs to the patient 5. The results of a laboratory test are dependent on the
named or identified in the test requisition form. quality of the sample as well as the assay technology.
2. All Tests are performed and reported as per the 6. Result delays could be because of uncontrolled
turnaround time stated in the SRL Directory of services circumstances. e.g. assay run failure.
(DOS). 7. Tests parameters marked by asterisks are excluded from
3. SRL confirms that all tests have been performed or the “scope"of NABL accredited tests. (If laboratory is
assayed with highest quality standards, clinical safety & accredited).
technical integrity. 8. Laboratory results should be correlated with clinical
4. A requested test might not be performed if: information to determine Final diagnosis.
a. Specimen received is insufficient or inappropriate 9. Test results are not valid for Medico- legal purposes.
specimen quality is unsatisfactory 10. In case of queries or unexpected test results please call
b. Incorrect specimen type at SRL customer care (Toll free: 1800-222-000). Post proper
c. Request for testing is withdrawn by the ordering doctor investigation repeat analysis may be carried out.
or patient
d. There is a discrepancy between the label on the
specimen container and the name on the test requisition
form
SRL Limited
Fortis Hospital, Sector 62, Phase VIII,
Mohali 160062
Page 9 Of 9
Gall eal*h3
Everything about health
Name : B BOOBALAN- Age: 29 Years Sex: Male
MRN :8931182 Report Dater 14-05 2020
lnvestigation: Urine Cotinine Test
Result: NEGATIVE
Reference Limits of Detection: < 200n&/ml- Negauve
>200n9/ml Pos tive
Note:
l.Detection limited cut offfor Urine cotinine is > 200n9/fiL.

The above test is for Qualitative analysis
Signa
i,EnS
I r 'i :i7

Path

Uploaded by

Document Information

Original Description:

Original Title

Copyright

Available Formats

Share this document

Share or Embed Document

Sharing Options

Did you find this document useful?

Is this content inappropriate?

Copyright:

Available Formats

Path

Uploaded by

Copyright:

Available Formats

DIAGNOSTIC REPORT

CLIENT CODE : C000082254 Cert. No. M-0255

CLIENT'S NAME AND ADDRESS : SRL LIMITED

PATIENT NAME : BOOBALAN B PATIENT ID : CH.8931182

ACCESSION NO : 0042TE000975 AGE : 29 Years SEX : Male DATE OF BIRTH : 23/08/1990

DRAWN : 14/05/2020 10:44 RECEIVED : 14/05/2020 15:18 REPORTED : 14/05/2020 17:19

REFERRING DOCTOR : DR. CALL HEALTH CLIENT PATIENT ID : 8931182

UHID: 8931182 REQNO-122301529

Test Report Status Final Results Biological Reference Interval Units

LIVER FUNCTION PROFILE, SERUM

BILIRUBIN, DIRECT 0.33 High 0.0 - 0.2 mg/dL

BILIRUBIN, INDIRECT 2.31 High 0.1 - 1.0 mg/dL

TOTAL PROTEIN 7.6 6.4 - 8.2 g/dL

ALBUMIN 4.2 3.4 - 5.0 g/dL

GLOBULIN 3.4 2.0 - 4.1 g/dL

ALBUMIN/GLOBULIN RATIO 1.2 1.0 - 2.1 RATIO

ASPARTATE AMINOTRANSFERASE (AST/SGOT) 30 15 - 37 U/L

ALANINE AMINOTRANSFERASE (ALT/SGPT) 62 High < 45.0 U/L

ALKALINE PHOSPHATASE 71 30 - 120 U/L

GAMMA GLUTAMYL TRANSFERASE (GGT) 42 15 - 85 U/L

LACTATE DEHYDROGENASE 206 High 100 - 190 U/L

SERUM BLOOD UREA NITROGEN

GAMMA GLUTAMYL TRANSFERASE, SERUM

CLIENT CODE : C000082254 Cert. No.

CLIENT'S NAME AND ADDRESS : SRL LIMITED

PATIENT NAME : BOOBALAN B PATIENT ID : CH.8931182

ACCESSION NO : 0042TE000975 AGE : 29 Years SEX : Male DATE OF BIRTH : 23/08/1990

DRAWN : 14/05/2020 10:44 RECEIVED : 14/05/2020 15:18 REPORTED : 14/05/2020 17:19

REFERRING DOCTOR : DR. CALL HEALTH CLIENT PATIENT ID : 8931182

UHID: 8931182 REQNO-122301529

Test Report Status Final Results Biological Reference Interval Units

GAMMA GLUTAMYL TRANSFERASE (GGT) 42 15 - 85 U/L

Lower than normal level may be due to:

Causes of decreased levels

CLIENT CODE : C000082254 Cert. No.

CLIENT'S NAME AND ADDRESS : SRL LIMITED

PATIENT NAME : BOOBALAN B PATIENT ID : CH.8931182

ACCESSION NO : 0042TE000975 AGE : 29 Years SEX : Male DATE OF BIRTH : 23/08/1990

DRAWN : 14/05/2020 10:44 RECEIVED : 14/05/2020 15:18 REPORTED : 14/05/2020 17:19

REFERRING DOCTOR : DR. CALL HEALTH CLIENT PATIENT ID : 8931182

UHID: 8931182 REQNO-122301529

Test Report Status Final Results Biological Reference Interval Units

COMPLETE BLOOD COUNT, EDTA WHOLE

RED BLOOD CELL COUNT 5.04 4.5 - 5.5 mil/µL

WHITE BLOOD CELL COUNT 7.50 4.0 - 10.0 thou/µL

PLATELET COUNT 222 150 - 410 thou/µL

RBC AND PLATELET INDICES

MEAN CORPUSCULAR VOL 87.0 83 - 101 fL

MEAN CORPUSCULAR HGB. 30.3 27.0 - 32.0 pg

MEAN CORPUSCULAR HEMOGLOBIN 34.8 High 31.5 - 34.5 g/dL

RED CELL DISTRIBUTION WIDTH 12.3 11.6 - 14.0 %

MEAN PLATELET VOLUME 9.3 6.8 - 10.9 fL

WBC DIFFERENTIAL COUNT

ABSOLUTE NEUTROPHIL COUNT 3.38 2.0 - 7.0 thou/µL

ABSOLUTE EOSINOPHIL COUNT 0.38 0.02 - 0.50 thou/µL

CLIENT CODE : C000082254 Cert. No.

CLIENT'S NAME AND ADDRESS : SRL LIMITED

PATIENT NAME : BOOBALAN B PATIENT ID : CH.8931182

ACCESSION NO : 0042TE000975 AGE : 29 Years SEX : Male DATE OF BIRTH : 23/08/1990

DRAWN : 14/05/2020 10:44 RECEIVED : 14/05/2020 15:18 REPORTED : 14/05/2020 17:19

REFERRING DOCTOR : DR. CALL HEALTH CLIENT PATIENT ID : 8931182

UHID: 8931182 REQNO-122301529