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Arch Expansion
Arch Expansion
By
AMR SAMY
MSc student, Orthodontic Department
Faculty of Dental Medicine
Al-Azhar University (Assiut)
Content:
Introduction.
Diagnosis.
Causes For Transverse Maxillary Deficiency.
Types of Expansion:
1) Orthodontic Expansion.
2) Orthopedic Expansion.
3) Passive Expansion.
Slow expansion.
Rapid expansion.
Surgically assisted rapid palatal expansion.
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Introduction
The goals of orthodontic treatment are well established for static and
functional occlusal relationships. In order to achieve Andrews‘ six keys to
normal occlusion for the dentition, 1 the jaws must be optimally proportioned in
three planes of space and positioned in CR. Orthodontists have well-established
methods for diagnosing the skeletal relationship of the maxilla to the mandible
in the sagittal and vertical dimensions. 2,6 Several analyses for the transverse
dimension are also available, 3,6,7 but these analyses are not well accepted as
forming part of traditional orthodontic diagnosis. In the sagittal dimension,
when the jaws do not relate optimally, the dentition will attempt to compensate,
resulting in excessively proclined or retroclined anterior teeth. In the transverse
dimension, when the jaws do not relate optimally, usually due to a deficiency in
the width of the maxilla, 7,8 the teeth will erupt into a crossbite or reconfigure
their inclinations to avoid a crossbite. This compensation typically involves
lingual tipping of the mandibular posterior teeth, which are then described as
being excessively negatively inclined. In addition, the maxillary posterior teeth
are tipped facially. These teeth are then described as being excessively
positively inclined.
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In 1970, L. F. Andrews published his landmark paper describing the six keys
to normal static occlusion. 1 Over the next several decades, he and his son, W.
A. Andrews, worked to develop the six elements philosophy of orthodontic
diagnosis. One of the diagnostic criteria, Element III, is devoted to analyzing
the transverse relationship of the maxilla and mandible and is based on both
bony and dental landmarks. 10 The Element III analysis is based on the
assumption that the WALA (named after Will Andrews and Larry Andrews)
ridge determines the width of the mandible. According to Andrews‘ definition,
the WALA ridge is coincident with the most prominent portion of the buccal
alveolar bone, when viewed from the occlusal surface The WALA ridge is
essentially coincident with the mucogingival junction and approximates the
center of resistance of the mandibular molars. In a mature patient, the WALA
ridge and the width of the mandible cannot be modified with conventional
treatment. Thus the WALA ridge forms a stable basis for the Element III
analysis. 6The Element III analysis is based on the width change, if any, of the
maxilla needed to have upper and lower posterior teeth upright in bone, centered
in bone, and properly intercuspated.
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To determine the discrepancy, the first step is to determine the width of the
mandible, or the horizontal distance from the WALA ridge on the right side to
the WALA ridge on the left side. According to Andrews, optimally positioned
mandibular molars will be upright in the alveolus, and their facial axis (FA)
point, or center of the crown, will be horizontally positioned 2 mm from the
WALA ridge. With this information, the width of the mandible is then defined
asthe WALA-WALA distance minus 4 mm. 6
One then looks at the angulation of the maxillary molars and estimates the
amount of horizontal change that will occur between the FA points of the right
and left molars when they are optimally angulated. The estimated amount of
change is subtracted from the original FA-FA measurement. The result
represents the width of the maxilla. 6
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In order to have optimally positioned and optimally inclined molar teeth that
intercuspate well, Andrews states that the maxillary width must be 5 mm greater
than the mandibular width. 6 In order to determine the amount of transverse
discrepancy, or Element III change, needed to produce an ideal result, one takes
the optimal mandibular width, adds 5 mm, and subtracts the maxillary width.
Before choosing a method for measuring the base of the jaws, we must first
decide what location to use for measurement. In determining the location of the
WALA ridge, Andrews stated that the WALA ridge is an approximation of the
center of resistance of the mandibular teeth. Above the WALA ridge, the
alveolus can be dimensionally molded and altered, depending on the change in
angulation of the teeth. However, the same cannot be said for the portion of the
alveolus below the WALA ridge.
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Thus, in a mature patient, any portion of the alveolus apical to the WALA
ridge can be assumed to be reasonably dimensionally stable during tooth
movement, and,therefore, can define the dimensions of the patient‘s arch. In
Ricketts‘ analysis, Ag-Ag represents the basal portion of the mandible.
However, when one looks at the position of Ag on a three-dimensional image,
one sees that its correlation with the base of the alveolus is relatively weak in all
three planes of space for mature patients Thus, to locate the beginning of the
base of the mandible with a CT scan, it would seem best to find the skeletal
representation of the WALA ridge. This is approximately at the edge of the
cortical bone opposite the furcation of the mandibular first molars. We can also
use this technique to locate the beginning of the base of the maxilla. If we
assume that the maxilla begins at the projection of the center of resistance of the
maxillary teeth onto the buccal surface of the cortical bone, Ricketts‘ use of Mx
to determine maxillary width appears to be at approximately at the same
horizontal position. Additionally, by using Mx point, any exostoses present
along the buccal portion of the alveolus will not interfere with the measurement.
Andrews‘ method, on the other hand, has no directly definable skeletal
landmark for the maxilla; it relies on estimated changes in the angulation of the
molars to determine the skeletal transverse discrepancy.
Therefore, Ricketts‘ method of defining the basal skeletal width of the maxilla
appears to be more appropriate. We begin, then, by defining locations for
measuring maxillary and mandibular skeletal basal width. Next, we explore
concepts for defining these locations on cone-beam CT imaging.The basic
premise for the mandible is to locate the most buccal point on the cortical plate
opposite the mandibular first molars at the level of the center of resistance.
According to Katona, this location is approximately coincident with the
furcation of the roots of the molars. 30 As we explained above, the authors
chose this point due to the relative immutability of the alveolus apical to this
location with orthodontics and because it represents the absolute minimal width
of the basal bone for each jaw.For the purposes of this technique, the authors
used Dolphin 3D, release 11, but the concepts can be applied to any software
with the capability to analyze a cone-beam CT image. After properly orienting
the image, we open the multiplanar view (MPV) screen to see simultaneous
axial, sagittal, and coronal cuts of the image.
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To determine the width of the mandible, we scroll down through the image
until we locate the furcation of the first molar. Then we scroll posteriorly
through the scan until we locate the coronal cross-section through the center of
the mandibular first molars. Now we switch to full-screen axial view. Using the
cut lines as a guide, we measure the width of the mandible from the intersection
of the cut line with the most buccal portion of the cortical plate on both the right
and left sides.
For the maxilla, a similar method is employed. The only difference is that the
axial and coronal cuts must be taken at the position Mx-Mx, and the same
measurement as in the Ricketts‘ analysis is used.
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Habits—thumb sucking.
Syndromes.
Klippel-Feil syndrome.
Cleft lip and palate.
Congenital nasal pyriform aperture stenosis.
Marfan syndrome.
Craniosynostosis (Apert syndrome, Crouzon disease, Carpenter
syndrome)
Osteopathia striata.
Treacher Collins.
Duchenne muscular dystrophy.
Obstructive sleep apnea.
Iatrogenic (cleft repair).
Palatal dimensions and inheritance.
Muscular.
Nonsyndromic palatal synostosis.
Multifactorial.
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Types Of Expansion
1) Orthodontic Expansion
This is produced by conventional fixed appliances and different types of
removable appliances. In orthodontic expansion, there is lateral movement of
the buccal segments, which results in mainly dentoalveolar expansion. There is
buccal tipping of the crowns and lingual tipping of the roots. Aberrant soft-
tissue pressure from cheeks can cause relapse of the achieved expansion.
2) Orthopedic Expansion
In this type of expansion, changes are produced mainly in the skeletal
structures. There is less amount of dentoalveolar expansion. Rapid maxillary
expansion (RME) appliances are classical examples for true orthopedic
expansion. RME causes separation of midpalatal suture,and also affects
circumzygomatic and circumaxillary sutures. After expansion, new bone is
deposited in the midpalatal suture.
3) Passive Expansion
Passive expansion is the result from the intrinsic forces exerted by the tongue.
With the use of buccal shields (e.g. Frankel), the forces from the labial and
buccal musculature are prevented from acting on the dentition. This results in
the widening of the arches, because the forces from tongue exert expansible
forces on the arches. The tongue force is not counteracted by bucci nator
mechanism. Passive expansion is not achieved by mechanical appliances but by
the vestibular or lip shields.
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Expansion Plates
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W Arch Appliance
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2. In the palatal bridge: Derotation and expansion of molar on the same side and
distalization of molar on the opposite side.
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Disadvantages:
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This fixed removable appliance has stainless steel extensions that could be
adjusted and inserted into standard horizontal lingual sheaths which are welded
to the molar bands. An indent on the lingual attachment locks the expander to
the molar band ensuring patient protection. For increased protection, an
elastomer may also be placed.
The appliance has got a central component and a lateral component . The central
component is made of thermally activated NiTi alloy and the lateral arm, which
is bracing the palatal aspect of the maxillary posterior teeth, is made of stainless
steel. At room temperature, the expander is too stiff to be compressed and
inserted into the maxillary arch. The transition temperature of the NiTi alloy
used in the expander is 94°F, which is close to intraoral temperature. For the
appliance to be inserted into the mouth, it is chilled so that the central part
softens and can be easily inserted. Once the appliance is fitted, the expander
warms to the body temperature, becomes stiff and returns back to its original
position. The expander delivers constant force as it deactivates.
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This concept was reported in the dental literature by Emerson Angell in 1860,
which was initially opposed by McQuillen (1860) 2 and Coleman (1865).
Till the early 1900s, rapid palatal expansion was still uncommon as some
clinicians, like Lundstrom (1923), Brodie (1938), questioned the stability of the
rapid maxillary expansion. It was reintroduced by Hass (1961), called Hass
expander, whose clinical studies and animal experiments strongly supported
rapid maxillary expansion. Hass (1961) (1965), 7 (1970), Timms (1971),
(1980) and Wertz (1970) stated that rapid maxillary expansion produces true
orthopedic expansion wherein the changes are produced in the skeletal
structures rather than by the movement of the teeth through the alveolar bone.
5. To gain arch length in patients with moderate tooth size arch length
discrepancy. In a child with decreased maxillary width and dental crowding
expansion can be the correct method of gaining space to align the teeth.
6. Another use of RME is to widen the maxilla to make the smile more
attractive.
7. Other indications for rapid maxillary expansion include poor nasal airway,
septal deformity, recurrentear, nasal or sinus infection, allergic rhinitis, asthma
and before septoplasty.
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Applied Anatomy:
The maxilla together with the palatine bone forms the hard palate, floor and
greater part of the lateral walls of nasal cavity. It is paired bone that articulates
with its opposite. The maxillae articulate with 10 other bones of the face and
cranium including frontal, ethmoid, nasal, lacrimal, vomer, zygomatic and
palatine bones. These bones join the maxilla by suture in posterior and superior
aspects leaving the anterior inferior aspect free. The tenacity of circumaxillary
attachments due to buttressing is strong posterosuperomedially and postero
superolaterally. The midsagittal portion of the anterior and middle part of the
cranial base is formed by the sphenoid bone and lies posterior to maxilla. The
other significant articulation is that between frontal and zygomatic bones at the
lateral aspect of the orbit and the frontomaxillary sutures at their union with the
basal bone.
Though the pterygoid plates of sphenoid are bilateral, midsagittal suture that
will allow for lateral displacement is absent. The pyramidal process of the
palatine bone intertwine with the pterygoid plates. This confinement of
sphenoid ptery goid plates drastically reduces the ability of palatine bones to
segregate at the midsagittal plane.
Another equally important factor is the soft-tissue complex that invests these
skeletal structures. Masticatory muscles, facial muscles and its investing fascia
are relatively elastic so that it stretches on application of expansion force.
However, the ability of permanent adaptation of the stretched muscles,
ligaments and the fascia to the new environment needs further investigation.
During teeth movement, orthodontists are aware of this soft tissue limitation.
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suture
The intermaxillary and interpalatine sutures are called midpalatal sutures. RME
should be initiated prior to the ossification of the midpalatal suture. In infancy,
the sutures in vertical coronal section have a ‗Y‘ shape and bind the vomer with
the palatine processes. In the juvenile period, the junction between three bones
becomes higher and assumes more of a ‗T‘ shape with the interpalatal section
taking a serpentine course. By adolescence, the oronasal course of the suture
may become so interdigitated that mechanical interlocking is as in a jigsaw
puzzle and islets of bone are formed. Melsen 14 reports that transverse growth
of midpalatal suture continued up to 16years in girls and 18 years in boys. Most
studies report a broad range of ossification between 15 and 25 years, 15 but the
optimal period for performing RME procedure was between 8 and 15 years. The
suture starts to ossify posteriorly and always shows a greater degree of
obliteration posteriorly then anteriorly, while ossification comes very late
anterior to incisive foramen.
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Skeletal Effects
The midpalatal suture consists of a fibrous tissue with osteogenic layers on both
surfaces. The suture is nothing but an extension of the periosteal layer of the
bone. At an early age, there is more proliferation of fibrous tissue and with
aging the sutural space is replaced with bone. 16
Microscopic study of the midpalatal suture at different ages reveals that the
suture becomes increasingly tortuous and more interdigitated with bony spicules
as age increases (Melsen 14 ) . Hence, any expansion in late adolescence
becomes very difficult.During expansion, the opening of the suture involves
tissue injury followed by a repair characterized by proliferation of cells for
regeneration of the suture. The space created in the midpalatal area is filled with
fluids and blood, which is later replaced by bone. The tensile force on the suture
initiates bone formation between the sutures.
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This is due to the buttressing effect of the other bones in the posterior maxillary
regions. Thus, the maxillary bone opens like a hinge superiorly at the base of
the nose and the opening is more in the anterior region than in the posterior
region. 13 shows opening of the suture in the superior view. Similar
observations were made by Wertz (1970) 18 who noted that the maxilla opens
in a triangular fashion with the apex at the posterior nasal spine and base in the
anterior region. A word of caution is that as the intermaxillary suture is less
ossified in preschool children, rapid expansion force should not be used, as this
may produce undesirable changes in the nose.
Dentoalveolar Changes
Other Effects
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There are two types of fixed RME, namely, bonded and banded. Banded RME
appliances include Derichs- weiler, Haas, Issacson (Minne expander) and
Hyrax.
Derichsweiler Type
In this, the screw is connected to the bands by means of tags that are welded and
soldered to the palatal aspects of the band on one side and embedded in acrylic
on the palatal aspects of all nonbanded teeth except the incisors. Acrylic adapts
to the palate and is in two halves to permit activation of screw.
Haas type 6
Heavy stainless steel wire 0.045 inch or 1015 mm in diameter is welded and
soldered along the palatal aspects of the band. The free ends are turned back
embedded in acrylic (Fig. 36.6B). Both Derichsweiler and Haas types
incorporated the same type of expansion screw.
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Isaacson 21 Type
Hyrax 12
This type of expander uses the Hyrax (hygienic rapid expansion) screw. It has
heavy wires that are adapted, welded and soldered to the palatal aspects of the
bands .
Acrylic splints involve thick-gauge stainless steel wire being adapted closely to
the premolars and premolars both buccally and palatally and the screw is
soldered to the wire. The acrylic splint covers the occlusal, buccal and palatal
occlusal third of all the posterior teeth.
Advantages:
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Appliance Management :
Rapid palatal expansion appliance is done with two turns daily of the jackscrew
(0.5 mm activation), which produces 10–20 lb of pressure across the suture,
sufficient enough to create separation of suture by causing micro-fractures in the
bony spicules.
The maximum load occurred during turning of the screw, which dissipates soon
after (Zimring and Issacson 22 ). In younger individuals, the load dissipation is
longer in a twice daily activation. Therefore, slower rates of expansion would
allow for physiologic adjustment at the maxillary articulations, and would
prevent the accumulation of large residual loads within the maxillary complex.
22,23
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Activation Schedule :
The expansion schedule advocated by Zimring and Issacson: 22
1. Young patients: Two turns a day for 4–5 days and one turn a day for the rest
of the treatment period.
2. Adult patients: For the first 2 days, 2 turns/day are given. For the next 5–7
days, 1 turn/day is given and during the rest of the treatment period, 1 turn is
given every other day.
Every turn of the screw opens the appliance by 0.25 mm. Each turn involves 90°
activation. With rapid maxillary expansion at a rate of 0.5–1 mm/day, a 10 mm
expansion is achieved in 2–3 weeks.
But there are disadvantages, like bulkiness of the appliance and difficulty
in cleaning the appliance, which limits its use in young child in the mixed
dentition.
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A string or floss is tied to the key to prevent it from being swallowed. The
patient is monitored at weekly intervals and the amount of expansion is
measured as the distance between the two halves of the screw. Usually, as a
matter of overcorrection of the crossbite, expansion is stopped when the
maxillary lingual cusps come into contact with mandibular buccal cusps. After
the expansion is completed, the appliance itself serves as a fixed retainer for 3
months. The expansion screw is immobilized by adding cold-cure acrylic over
it. After 3 months of fixed retention, the RME is removed and replaced by a
removable retainer with acrylic covering the palate.
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Semirapid maxillary expansion involves two turns a day for the first 5–6 days
after appliance insertion and three turns a week for the rest of the treatment
period. Semirapid maxillary expansion is nothing but initial RME followed by
slow expansion. 24
Surgically assisted palatal expansion (SARPE) that cuts the bone for reducing
the resistance without completely releasing the maxillary segments succeeded
by Jackscrew rapid expansion is another possible treatment approach in adults
with narrow maxilla. The original concept of surgically assisted expansion was
that in adults, the cuts in maxillary lateral buttress decreases the resistance to a
level where midpalatal suture is opened forcefully (i.e. microfractured).
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The implication of SARPE is that the problem affects only the transverse plane
of space, and this is when it is most useful. It is difficult to justify the additional
cost and morbidity of surgically assisted expansion as a first stage of surgical
treatment, in a patient who would require another operation later to reposition
the maxilla in the anteroposterior or vertical planes of space. The primary
indication for SARPE is such severe maxillary constriction that segmental
expansion of the maxilla in the LeFort I procedure might compromise the blood
supply to the segments.
Indications:
SARPE is indicated for the treatment of the adults with narrow palatal
arch for the following:
1. To expand the arch for correcting posterior cross bite when no other surgical
jaw movements are considered.
4. To expand the arch for creating space without premolar extractions, if the
space required could be gained reasonably due to maxillary expansion 10 and
if other factors, like maxillary incisor protrusion on the underlying bone, have
been considered.
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References
1. Andrews LF. The six keys to normal occlusion. Am J Orthod. 1972;
62(3):296-309.
6. Andrews LF, Andrews WA. Andrews analysis. In: Syllabus of the Andrews
Orthodontic Philosophy. 9th ed. Six Elements Course Manual;2001.
10. Utt TW, Meyers CE, Wierzbe TF, Hondrum SO. A three-dimension-al
comparison of condylar position changes between centric relation and centric
occlusion using the mandibular position indicator. Am J Orthod Dentofac
Orthop. 1995; (107): 298-308.
11. Wertz RA. Skeletal and dental changes accompanying rapid mid-palatal
suture opening. Am J Orthod 1970;58:41–66.
12. McNamara JA, Brudon WL. Orthodontic and orthopedic treatment during
the mixed dentition. Ann Arbor: Needham Press; 1995.
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16. Graber TM, Vanarsdall RL. Current orthodontic principles and techniques
tissue reactions in orthodontics. 4th ed. 2004. [chapter 4].
19. Adkins MD, Nanda RS, Currier GF. Arch perimeter changes on rapid
palatal expansion. Am J Orthod Dentofacial Orthop 1990;97:194–99.
20. Hartgerink DV, The effect of rapid maxillary expansion on nasal airway
resistance: one year follow up. Unpublished Masters‘ thesis, Department of
Orthodontics, University of Michigan; 1986.
21. Isaacson RJ, Murphy TD. Some effects of rapid maxillary expansion in
cleft lip and palate patients. Angle Orthod 1964;34:143–154.
22. Zimring JF, Isaacson RJ. Forces produced by rapid maxillary expansion.
III. Forces present during retention. Angle Orthod 1965;35:178–86.
23. Isaacson RJ, Wood JL, Ingram AH. Forces produced by rapid maxillary
expansion. Angle Orthod 1964;34:256–70.
25. Adams, C. Philip, W. John S. Kerr, and C. Philip Adams. The Design,
Construction, and Use of Removable Orthodontic Appliances. London:
Butterworth-Heinemann, 1990.
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29. Akkaya, S., Lorenzon, S., Ucem, T.T. Comparison of dental arch and arch
perimeter changes between bonded rapid and slow maxillary expansion
procedures. Eur J Orthod. 1998;20:255–261.
30. Lines PA. Adult rapid maxillary expansion with corticotomy. Am J Orthod
1975;67:44–56.
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