Mat was admitted due to symptoms of angina. The nursing diagnosis was decreased cardiac output related to coronary artery disease. The plan was for the patient to report reduced angina episodes after 8 hours of nursing intervention. Interventions included establishing rapport, instructing the patient on medications and symptoms, and monitoring the patient's condition. The goal of reducing angina symptoms was met after 8 hours.
Mat was admitted due to symptoms of angina. The nursing diagnosis was decreased cardiac output related to coronary artery disease. The plan was for the patient to report reduced angina episodes after 8 hours of nursing intervention. Interventions included establishing rapport, instructing the patient on medications and symptoms, and monitoring the patient's condition. The goal of reducing angina symptoms was met after 8 hours.
Mat was admitted due to symptoms of angina. The nursing diagnosis was decreased cardiac output related to coronary artery disease. The plan was for the patient to report reduced angina episodes after 8 hours of nursing intervention. Interventions included establishing rapport, instructing the patient on medications and symptoms, and monitoring the patient's condition. The goal of reducing angina symptoms was met after 8 hours.
Mat was awakened output related to nursing intervention Establish rapport After 8 hours of due to shortness of disease process the patient will be Encourage the patient to take nursing intervention breath, noticed chest of coronary able to: medicine on time. the patient was able to discomfort and artery disease Report Instruct patient to notify nurse weakness in both (CAD) as anginal immediately when chest pain report reduce upper and lower evidenced by episodes occurs. the frequency extremities, and general weakness decreased in Assess and document patient and severity of increase in blood and dizziness frequency, response to medication. angina pressure. duration, and Provides information about The patient severity. disease progression. will be able to O: Demonstrate Identify precipitating event, if verbalize (2/8/11) relief of pain any: frequency, duration, understanding BP 160/120 as evidenced intensity, and location of pain. of the disease Cholesterol: 234 by stable vital Helps differentiate this chest process and Triglyceride: 123 signs, absence pain, and aids in evaluating treatment HDL: 26 of muscle possible progression to regimen. LDL: 83.4 tension and unstable angina. CHEST PAL: restlessness Observe for associated Negative for symptoms: pneumohemothorax. dyspnea, nausea and vomiting, Heart is not enlarged. dizziness, palpitations, desire Atherosclerotic aorta. to micturate. Decreased Bones are cardiac output stimulates unremarkable sympathetic and parasympathetic nervous (2/19/11) system. Troponin T: 0.03-0.1 Evaluate reports of pain in ECG CONCLUSION: jaw, neck, shoulder, arm, or Thickened hand. Pain is often referred to anterior mitral more superficial sites served valve leaflet by the same spinal cord nerve with trivial level. mitral Place patient at complete rest regurgitation. during anginal episodes. Aortic Reduces myocardial oxygen sclerosis with demand to minimize risk of trivial aortic tissue injury. regurgitation. Elevate head of bed if patient Trivial is short of breath. Facilitates tricuspid gas exchange to decrease regurgitation hypoxia and resultant CT SCAN REPORT: shortness of breath. Monitor heart rate and Chronic small vessel rhythm. Patients with ischemic changes unstable angina have an increased risk of acute life- threatening dysrhythmias. Monitor vital signs every 5 min during initial anginal attack. Blood pressure may initially rise because of sympathetic stimulation. Maintain quiet, comfortable environment. Restrict visitors as necessary. Mental/emotional stress increases myocardial workload. Provide light meals. Have patient rest for 1 hr after meals. Decreases myocardial workload associated with work of digestion, reducing risk of anginal attack. Administer Prescribed medications. Aspirin to reduce ability of blood clot so that the blood flows easier; Nitrates to relax blood vessels; Statis to reduce deposit on the arterial walls; Beta blockers to decrease cardiac demand for oxygen Provide supplemental oxygen as indicated. Increases oxygen available for myocardial uptake and reversal of ischemia.