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    Gonorrhea and Syphylis Pa Tho Physiology

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    japz yeah
    The document discusses the pathophysiology of gonorrhea and syphilis. For gonorrhea, the bacteria Neisseria gonorrhoeae adheres to mucous membranes and penetrates tissue, causing inflammation and abscess formation that can lead to complications like infertility. The infection can spread through direct contact with secretions or indirectly through fomites, and from mother to child. For syphilis, the spirochete bacteria adheres to epithelium and causes a chancre lesion during the primary stage. In later stages, it may cause widespread rash, mucous patches, or gummas and damage organs like the heart, brain, and bones in the tertiary stage if left untreated.

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    Gonorrhea and Syphylis Pa Tho Physiology

    Uploaded by

    japz yeah
    2K views3 pages
    The document discusses the pathophysiology of gonorrhea and syphilis. For gonorrhea, the bacteria Neisseria gonorrhoeae adheres to mucous membranes and penetrates tissue, causing inflammation and abscess formation that can lead to complications like infertility. The infection can spread through direct contact with secretions or indirectly through fomites, and from mother to child. For syphilis, the spirochete bacteria adheres to epithelium and causes a chancre lesion during the primary stage. In later stages, it may cause widespread rash, mucous patches, or gummas and damage organs like the heart, brain, and bones in the tertiary stage if left untreated.

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    The document discusses the pathophysiology of gonorrhea and syphilis. For gonorrhea, the bacteria Neisseria gonorrhoeae adheres to mucous membranes and penetrates tissue, causing inflammation and abscess formation that can lead to complications like infertility. The infection can spread through direct contact with secretions or indirectly through fomites, and from mother to child. For syphilis, the spirochete bacteria adheres to epithelium and causes a chancre lesion during the primary stage. In later stages, it may cause widespread rash, mucous patches, or gummas and damage organs like the heart, brain, and bones in the tertiary stage if left untreated.

    Copyright:

    Attribution Non-Commercial (BY-NC)
    Download as DOC, PDF, TXT or read online from Scribd
    Flag for inappropriate content
    Download as doc, pdf, or txt
    2K views3 pages

    Gonorrhea and Syphylis Pa Tho Physiology

    Uploaded by

    japz yeah
    The document discusses the pathophysiology of gonorrhea and syphilis. For gonorrhea, the bacteria Neisseria gonorrhoeae adheres to mucous membranes and penetrates tissue, causing inflammation and abscess formation that can lead to complications like infertility. The infection can spread through direct contact with secretions or indirectly through fomites, and from mother to child. For syphilis, the spirochete bacteria adheres to epithelium and causes a chancre lesion during the primary stage. In later stages, it may cause widespread rash, mucous patches, or gummas and damage organs like the heart, brain, and bones in the tertiary stage if left untreated.

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    PATHOPHYSIOLOGY OF GONORRHEA Direct Contact Transmission through exudates or secretions of mucous membranes Indirect Contact Transmission through Fomites

    Mother-Child Contaminated vaginal secretions

    Gonococcus / N. Gonorrhoeae adheres to the epithelium

    Redness Pain Heat/Burning Sensation Swelling

    Bacteria penetrates mucosa

    Inflammatory response; Formation of small abscesses

    May obstruct drainage of small abscesses Small abscesses becomes large abscesses Scarring formation Epidydymal Involvement Sterility Involvement of Fallopian Tubes Complications Ectopic Pregnancy Infection spreads along mucosal surfaces

    Spread/Travel through the blood stream

    PATHOPHYSIOLOGY OF SYPHILIS Contact with body fluids (i.e. semen, blood, tears), mucous discharges (i.e. form the eyes, nose, genital tract and bowels), surface lesions Indirect contact with Fomites Placental transmission from Syphilitic mother

    Congenital Syphilis Spirochete adheres in the epithelium

    PRIMARY STAGE Chancre appears in contact skin or mucosa

    Organisms reproduce in the Chancre; Initiates immune response

    Lymphadenopathy

    Lesion heals Organisms enter the general circulation SECOND STAGE General Signs of Infection

    Mucous Patches (Loose, white, tongue)

    Widespread Rash (Macular, Reddish, Skin/Mucous Membranes)

    Lesions Disappear spontaneously LATENT STAGE (years) Asymptomatic, but with serologic evidence TERTIARY STAGE/LATE Recurrence of Lesions may occur

    TERTIARY STAGE/LATE Gumma appears (Nervous Fibrosis)

    Bone

    Skin

    Liver

    Heart

    Brain

    Pathologic Fractures

    Cirrhosis

    Damage the arterial walls

    Neurosyphilis CNS Affectation

    Aortic Aneurysm

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