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Elbow Trauma Gone Bad 2017 Aaos
Elbow Trauma Gone Bad 2017 Aaos
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ICL 430: Elbow Trauma Gone Bad
Description:
Complications ensuing from elbow trauma can be challenging to manage. We will discuss
current understanding on recognizing and treating instability patterns. Critical concepts
regarding soft tissue complications such as stiffness, wound healing, nerve injury, revision ORIF,
arthroplasty, with an emphasis on case-based discussions.
Outline
II. Missed instability patterns in the elbow, how to recognize and treat—
Robert Tashjian (20 minutes)
a. Posterolateral rotatory instability- the most common pattern of elbow instability
b. Posteromedial instability- associated with anteromedial facet of coronoid
c. Missed instability
i. Salvage: bony reconstruction, ligamentous, external fixation
Q&A 10 minutes
V. Arthroplasty options
Emilie Cheung (20 minutes)
a. Total elbow arthroplasty
i. Track record
b. Radial capitellar arthroplasty
c. Distal humeral hemiarthroplasty
1
Things to consider when dealing with a stiff elbow joint after trauma:
The motion
• Measure with a goniometer
• Pain at rest is concerning for infection
• Motion through the arc is typically not painful, if it is painful
then consider articular joint involvement and the patient may
not respond as well to the surgical release if they continue to
have pain and may need to consider an interposition (provided
ligaments are intact)
• May have pain at the extremes of motion where there is bony
impingement
• Elbow flexion contracture less than 100/110 degrees – need to deal
with the ulnar nerve
• Is it a hard or soft endfeel ie, kissing bone lesions or capsular
contracture
• Do they have motor function to move the elbow after it is released?
Any spasticity issues or pathologic increased muscle tone?
• Functional arc of motion is 30 – 130 degrees, 50 degrees
pronation/supination but can be individualized based on the patients
needs
The joint
• It is reduced/congruent?
• Is there any articular malunion?
• Bone likes to sit in 3 fossae
1. radial fossa
2. coronoid fossa
3. olecranon fossa
• The coronoid and olecranon tips have osteophytes that extend
medially and laterally and can impinge along the columns of the
coronoid and olecranon fossae
2
Treatment options
• Arthroscopy
• If soft tissue contracture only, minimal bone
involvement, flexion ROM >110 degrees
• Open release
• Medial, lateral, combined
• Arthroplasty
• Interposition, joint replacement
Arthroscopic Release or No
Lateral incision with
lateral column approach
Open Release
Soft tissue contracture only Soft tissue and bone involved Soft tissue and bone involved
Flexion < 110 ° Flexion > 110° Flexion < 110 °
Posterior midline incision Lateral or posterior midline incision Posterior midline incision
Medial approach Lateral column approach Medial approach
Ulnar nerve transposition Ulnar nerve transposition
Release of posterior MCL Release of Posterior MCL
Medial approach
if needed
Lateral column
approach if needed
4
Radioulnar Synostosis
• Need advanced imaging to understand reason
• Rehabilitation is more challenging
Rehabilitation
• Anterior splint with arm in extension
– Hang overnight
– Edema control
– Protects posterior wound
– Extension is hardest to keep
• Continuous regional block
– Full AAROM POD #1 – patient driven every hour on the hour, others
have used CPM but I have not been doing this
– Wean off block during POD#1
• Splints
– Night time extension splint
– Cuff and collar or simple sling during day if needed or nothing
Other pearls
References
1. Streubel PN, Cohen MS.Open surgical release for contractures of the elbow.J Am
Acad Orthop Surg. 2015 Jun;23(6):328-38.
2. Koh KH, Lim TK, Lee HI, Park MJ.Surgical release of elbow stiffness after internal
fixation of intercondylar fracture of the distal humerus.J Shoulder Elbow Surg. 2013
Feb;22(2):268-74.
3. Charalambous CP, Morrey BF.Posttraumatic elbow stiffness.
J Bone Joint Surg Am. 2012 Aug 1;94(15):1428-37.
4. Williams BG, Sotereanos DG, Baratz ME, Jarrett CD, Venouziou AI, Miller MC.The
contracted elbow: is ulnar nerve release necessary?J Shoulder Elbow Surg. 2012
Dec;21(12):1632-6.
5. Lindenhovius AL, van de Luijtgaarden K, Ring D, Jupiter J.
Open elbow contracture release: postoperative management with and without
continuous passive motion.J Hand Surg Am. 2009 May-Jun;34(5):858-65.
6. Lindenhovius AL, Linzel DS, Doornberg JN, Ring DC, Jupiter JB.
Comparison of elbow contracture release in elbows with and without heterotopic
ossification restricting motion.J Shoulder Elbow Surg. 2007 Sep-Oct;16(5):621-5.
7. Ring D, Adey L, Zurakowski D, Jupiter JB.Elbow capsulectomy for posttraumatic
elbow stiffness. J Hand Surg Am. 2006 Oct;31(8):1264-71.
8. Tan V, Daluiski A, Simic P, Hotchkiss RN.Outcome of open release for post-
traumatic elbow stiffness.J Trauma. 2006 Sep;61(3):673-8.
10. Dávila SA, Johnston-Jones K.Mana ging the stiff elbow: operative, nonoperative,
and postoperative techniques.J Hand Ther. 2006 Apr-Jun;19(2):268-81. Review.
11. Jupiter JB, O'Driscoll SW, Cohen MS.The assessment and management of the stiff
elbow.Instr Course Lect. 2003;52:93-111. Review.
Missed instability patterns in the elbow, how to recognize and treat: PLRI, PMRI,
Missed dislocations with ligament and bone defects
ICL – Elbow Trauma Gone Bad
Robert Z. Tashjian, MD
Associate Professor, Department of Orthopaedics
University of Utah School of Medicine
I. Background
o Condition where radial head AND ulna rotate externally on the distal humerus leading to
posterior displacement and subluxation of the radial head relative to the capitellum
o Radius and ulna move as a unit since proximal radioulnar joint remains intact
o First described by O’Driscoll in 1991 (O’Driscoll et al. JBJS 1991)
o Overall incidence is rare yet considered most common form of chronic elbow instability
o Instability typically occurs from an elbow dislocation with failure to heal of the lateral
ligamentous structures. May be secondary to other mechanisms of LCL injury.
o Typically complaints of clicking, mechanical symptoms, instability
o Lateral ulnar collateral ligament (LUCL) insufficiency considered primary etiology
although now most recognize that secondary stabilizer insufficiency is also required to
result in PLRI
o Treatment includes nonoperative (avoidance, bracing) although with limited success;
Surgical reconstruction of LUCL preferred with multiple techniques described
o Outcomes are reproducible and durable to prevent instability
II. Anatomy
-Primary restraints to PLRI are the lateral ligament complex, the radial head and coronoid
process and the lateral extensor musculature
a. Lateral ligament complex
1. Primary restraint of PLRI – prevents external rotation of radius and ulna relative to
humerus (O’Driscoll JBJS Am 1991).
2. Components –
• Annular ligament – anterior margin of sigmoid notch to supinator crest
• Radial collateral ligament – lateral epicondyle to annaular ligament
• Lateral Ulnar Collateral Ligament – lateral epicondyle to supinator crest
(originally described as primary ligament component to restrain PLRI).
Original anatomic description by Morrey et al. Clin Orthop Rel Res 1985.
3. Anatomically appears to be a continuous ligamentous sheet (Imantani et al J Shoulder
Elbow Surg 1999)
• Isolated sectioning of LUCL ligament alone will not lead to PLRI –
requires 2 of 3 components of LCL complex to be injured to result in
PLRI (Dunning et al JBJS Am 2001)
• Arthroscopic sectioning study showed that both the radial collateral and
LUCL need to be cut to cause significant PLRI; full PLRI instability only
when muscle cut as well (McAdams J Shoulder Elbow Surg 2005)
4. Isometry
Radial collateral ligament is isometric; LUCL is not; LUCL is loose in extension
and tight in flexion; LUCL is most isometric 2 mm anterior to lateral epicondyle.
(Moritomo et al JBJS 2007)
5. Injury may occur due to trauma (dislocation), chronic attenuation or iatrogenic causes
• Trauma – dislocations cause disruption of the LUCL in all cases
(Josefsson 1987); typically will heal – failure to heal may lead to chronic
instability
o Most common injury of LCL complex is avulsion off the lateral
epicondyle (52%) followed by midsubstance rupture (29%) in
series of patients with simple or complex dislocations. (McKee
et al. J Shoulder Elbow Surg 2003)
o May occur with radial head excision in cases of trauma (Hall et
al JBJS Am 2005)
• Chronic attenuation –
o Long-standing cubitus varus (O’Driscoll JBJS AM 2001, Beuerlin
JBJS Am 2004); longstanding overuse; generalized hyperlaxity
• Iatrogenic -
o Lateral epicondylitis release (open or scope)(Kalainov JBJS
2005); open approaches to lateral elbow and radial head; serial
steroid injections
b. Radial head, capitellum and coronoid –
1. Radial Head – Provides osseous congruency with capitellum and tightens LCL complex
Improving stability
• Jensen et al J Shoulder Elbow 2005 – Biomechanically showed increased
varus and external rotatory laxity after isolated excision of radial head
• Deutch et al. J Shoulder Elbow Surg 2003 – radial head excision resulted
in 45% increase in pathologic forearm external rotation with intact
ligaments
• Schneeberger AG et al. JBJS 2004 – Excision of radial head with intact
ligaments increased posterolateral rotatory laxity which was restored
with radial replacement
• Hall et al JBJS 2005 – Clinical series of PLRI in patients after radial head
resection
2. Capitellum –
• Loss of height would lead to slackening of LCL and possible PLRI
• Sabo et al. J Shoulder Elbow Surg 2011 – Biomechanical study showing
capitellum excision leads to increased ulnar external rotation which is
corrected with replacement
3. Coronoid process –
• May play additive role with radial head in PLRI stabilization
• Schneeberger AG et al. JBJS 2004 – 30% coronoid resection plus radial
head resection led to dislocation of elbow where radial head resection
alone led to increased posterolateral rotatory laxity alone without
dislocation; Elbow stability restored with radial head replacement
although elbows lacking > 50% of coronoid could not restore elbow
stability with radial head replacement alone despite intact ligaments
•Deutch et al J Shoulder Elbow Surg 2003 – 50% coronoid resection
increased pathologic forearm rotation 28%
• Okazaki et al. J Hand Surg Am 2007 – 3 patient case report of
insufficient coronoid process after fx leading to PLRI
c. Common extensor origin
1. Important static secondary stabilizers crossing the lateral elbow
2. Possible dynamic stabilizers
3. Extensor disruption commonly occurs with dislocations or fracture-dislocations;
Occurred in 66% of cases. (McKee et al. J Shoulder Elbow Surg 2003)
4. May be violated as part of surgical approach and may lead to instability if not fully
repaired or reattached
5. Cohen et al. JBJS 1997 – sectioning studies provide evidence that extensor muscle
origins provide stability through fascial bands and intermuscular septa
Varus Posteromedial Rotatory Instability (PMRI) – Anteromedial Facet Fractures of the Coronoid
I. Background
o The coronoid process plays a critical role in maintaining stability of the elbow joint.
o Historically, fractures of the coronoid process were defined by their overall size based
on the classification system described by Regan and Morrey.
o Several recent classification systems have been created to define fractures of the
coronoid process by their location and their associated injury patterns
o O’Driscoll et al created the coronoid fracture classification system
Type II fractures, which involve the anteromedial facet, include three subtypes.
• Subtype 1 fractures involve the rim.
• Subtype 2 fractures involve the rim and tip.
• Subtype 3 fractures involve the rim and sublime tubercle.
o Adams et al classified coronoid fractures based on CT scans.
The authors reported that oblique anteromedial fractures, which are equivalent
to O’Driscoll type II fractures, accounted for 17% of coronoid process injuries.
The authors also reported on a newly described oblique anterolateral fracture
pattern, which accounted for 7% of coronoid process injuries and did not often
require surgical fixation.
Conversely, anteromedial facet fractures often require surgical treatment to
avoid the development of early rapid arthritis.
o Aggressive surgical fixation for most coronoid process injuries has been supported
biomechanically and suggested clinically
o Recent studies have recognized that not all anteromedial facet fractures require surgical
repair
II. Pathophysiology
- Varus PMRI of the elbow results from a fall backward onto both hands, in which there was
axial loading of the arm with pronating and varus forces.
- Varus PMRI will result in a rupture of the LCL complex, and the trochlea will fracture the
anteromedial facet of the coronoid.
- Doornberg et al reported that 58% of the anteromedial facet is unsupported by the proximal
ulna, which places it at risk for fracture in the mechanism of injury already mentioned.
- The radial head is typically preserved without injury.
- The MCL can be injured as well with this injury pattern.
- Rhyou et al reported concomitant repair of the MCL in 3 of 18 cases while Park et al
reported injury in 6 of 11 cases of which 3 cases required surgical repair due to residual
instability after LCL repair and coronoid fracture fixation.
- The treatment strategy for anteromedial facet injuries potentially includes nonsurgical
treatment, LCL repair only, anteromedial facet repair only or both LCL and anteromedial
facet repair.
- The surgical indications for anteromedial facet injuries continue to evolve as clinical
information on their outcomes becomes available.
III. Evaluation
- The history should query patients on the dislocation as well as the position of the hands and
arms at the time of the injury.
- Ap and lateral radiographs of the elbow and wrist should be performed
- Radiographs must be carefully reviewed because anteromedial facet injuries often may be
very subtle.
- The medial joint space often is narrowed or incongruent on AP radiographs.
- The fracture may be difficult to see on lateral radiographs
o Sanchez-Sotelo et al described a double-crescent sign in the setting of a
depressed fracture.
- Three-dimensional CT reconstructions with the radius and humerus subtracted are
critical to evaluate coronoid process injuries
o The ulna can be rotated to view the articular surface from proximal to distal,
which allows for classification of the injury based on the O’Driscoll
classification system
o The fracture should be evaluated for displacement as well as the subtype
classification.
- Varus stress radiographs also may be obtained to evaluate for medial joint space
collapse and lateral joint space widening.
- On physical examination, the patient should be placed in the supine position and
brought through a full range of elbow flexion and extension to offload the LCL.
o The patient should be queried about symptoms of instability or grinding, both
of which are indications for surgical repair.
IV. Treatment - Indications
- Because this injury pattern has been recognized only in the past 10 years and it is, overall,
much less common than other forms of complex elbow instability, studies on the outcomes
of clinical care for patients who underwent treatment for cornoid process injuries are
limited.
- Because of these limited data, the indications for nonsurgical and surgical treatment of
coronoid process injuries are still evolving.
o Initially, all injuries were believed to require surgical fixation of both the LCL and
the anteromedial facet fracture.
o As knowledge of cornoid process injuries improved, the treatment algorithm, which
is based on fracture pattern, size, and location; stress radiographs; and clinical
symptoms, has been refined
- All patients should initially be treated with closed reduction and splinting, after which the
elbow should be reexamined.
- Varus stress radiographs with the forearm placed in pronation should be obtained in the
setting of an anteromedial facet fracture that has minimal displacement with no static
collapse of the medial ulnohumeral joint space seen on AP radiographs.
o If no medial collapse of the ulnohumeral joint space, no lateral gapping, and a firm
end point are seen, the patient can be considered for nonsurgical treatment.
o Passive elbow flexion and extension should be evaluated with the patient placed in
the supine position.
o If no crepitus or feelings of instability exist and stress radiographs are negative,
nonsurgical treatment can be considered.
o If nonsurgical treatment is selected, flexion and extension overhead exercises with
the patient in the supine position as well as passive supination and pronation with
the elbow positioned 90° of elbow flexion should begin 1 week after injury.
o A sling should be worn for 6 weeks to protect the elbow between physical therapy
sessions.
o Radiographs should be obtained every week for the first 3 weeks as well as 6 weeks
after injury to confirm maintenance of alignment and no collapse.
o Six weeks after injury, the sling should be discontinued, and progression stretching
may be performed, with strengthening initiated 3 months after injury.
o Patients may return to activities as tolerated 4 to 5 months after injury.
- Surgery is recommended if substantial displacement with subluxation is seen on radiographs
or if positive varus stress or clinical symptoms of grinding or instability with supine overhead
elbow flexion and extension are apparent.
- Surgery may include LCL repair only, anteromedial facet repair only, or both.
o Park et al treated patients who had O’Driscoll type II subtype 1 fractures with LCL
repair only and patients who had O’Driscoll type II subtype 2 and subtype 3
fractures with LCL repair and buttress plating.
o Rhyou et al recommended treatment based on fracture fragment size (articular
height as defined by Pollack et al.)
If the fragment was smaller than or equal to 5 mm, the authors did not
repair the fracture.
If the fragment was larger than 6 mm and not comminuted, the authors
repaired the facture via a medial approach.
If varus stress testing under fluoroscopy with the forearm positioned in
pronation revealed a firm end point with a congruent medial ulnohumeral
joint, the authors did not repair the LCL.
If loss of congruency occurred, the authors repaired the LCL.
This treatment algorithm allowed some patients who had very small
fractures and a stable elbow to undergo nonsurgical treatment.
o Nonsurgical treatment can be considered for slightly larger fragments if no grinding
or instability exists during overhead passive flexion and extension with the patient
placed in the supine position
IV. Outcomes
- Overall, limited data exist on the outcomes for patients who undergo surgical
treatment for anteromedial facet fractures in the setting of varus PMRI
- Doornberg and Ring reported on a series of 18 patients who had anteromedial facet
fractures, 15 of whom were treated surgically, and 3 of whom were treated
nonsurgically.
o The coronoid was fixed with a plate in nine patients, a screw in one patient,
and sutures in one patient. The coronoid was not repaired in the remaining
seven patients.
o At mean follow-up of 26 months, the authors reported that six patients had
malalignment of the fracture and varus subluxation, which was caused by a
lack of fixation in four patients and a loss of fixation in two patients.
o All six of the patients with malalignment and varus subluxation had fair or
poor results.
o The remaining 12 patients had good or excellent outcomes. There was a 17%
incidence of ulnar neuropathy in patients who underwent repair via the FCU-
splitting approach. The three patients who were treated nonsurgically had
maintenance of alignment of the fracture and had excellent results based on
the rating system described by Broberg and Morrey. The authors
recommended that surgical fixation of anteromedial facet fractures to prevent
malalignment, except in patients who have very small fractures with no
subluxation and no radiocapitellar joint opening with varus stress.
Missed Instability
I. Background
a. Missed or neglected elbow dislocations are fortunately uncommon. Can be a result of
an incomplete examination or lack of obtaining radiographs
b. Chronic elbow dislocations can also result of surgically repaired simple or complex
dislocations with failure of fixation or ligament repair leading to persistent subluxation
or dislocations
i. Causes of failure
1. Bony stabilizers insufficient
a. Inadequate bony fixation due to poor methods of internal
fixation
b. Poor bone quality due to size, comminution or osteoporosis
leading to early failure of appropriate methods of fixation
c. Failure of healing fracture fixation
i. Age
ii. Smoking
iii. Comorbidities
iv. Osteoporosis
2. Ligament stabilizers insufficient
a. Failure to repair ligament injuries
b. Failure of ligament repair techniques due to fixation failure
c. Failure to heal of ligament repairs
i. Lack of postoperative protect
1. Splint, brace, sling
2. Protected therapy
3. External fixation
ii. Lack of fixation of other stabilizers (i.e bony)
d. Failure to reconstruct chronic ligament injuries
3. Cartilage loss
a. Acute – Large osteochondral injuries requiring repair or
reconstruction
i. Trochlear – Consider large allograft reconstruction vs
arthroplasty
ii. Posterior capitellar – Osborne-Cottrell lesion requiring
fixation or grafting
b. Chronic
i. Loss of articular congruity resulting in persistent
instability despite treatment of bony and ligamentous
stabilizers - Salvage options – interposition, fusion,
arthroplasty
b. Coronoid
i. The coronoid process has been biomechanically shown to be an important
stabilizer to varus, posteromedial rotatory, posterolateral rotatory and axial
forces
ii. Poor results have been reported in cases of complex dislocations where
coronoid process fractures have not been repaired
iii. Consequently, repair should be attempted for most fractures that are larger
than 10% of coronoid process after a fracture dislocation
iv. Irreparable coronoid fractures due to severe comminution or cases where the
coronoid fragments have been excised pose a difficult problem
v. Treatment options for irreparable fractures
1. Hinged external fixation has been reported as a salvage procedure for
unreconstructable coronoid fractures in order to maintain elbow
stability
2. Other authors have reported reconstruction of the coronoid process
with a variety of grafts including both autograft (radial head, iliac crest,
proximal olecranon) and allograft
a. Van Riet et al reported on 6 patients who underwent allograft (3
cases) and autograft (3 cases using radial head) reconstruction
i. At a mean of 64 months postoperative, the authors
reported 1 excellent, 2 good, 1 fair and 1 poor result
using the Mayo Elbow Performance Score and that
results were unpredictable using this technique
b. Esser et al and Chen et al also reported on graft reconstructions
utilizing autograft radial head
c. Moritomo et al reported 2 cases of using the ipsilateral
olecranon tip as a graft source for coronoid reconstruction using
an anterior approach
i. Both elbows were stable at minimum of 1 year follow-
up with a pain free flexion arc from 30° to 120°.
d. Kohls-Gatzoulis et al and Chung et al each reported on using a
iliac crest autograft to reconstruct a coronoid with good results
at a minimum of 1 year
3. Results of graft reconstructions vary significantly. Despite this variability
from a very limited group of case reports, the technique is a reasonable
alternative for a difficult clinical problem
vi. Variety of fixation techniques for large coronoid graft reconstructions
1. AP screws, PA Screws, Suture
III. Ligamentous Insufficiency
a. Consider ligament repair laterally and/or medially – Reconstruction likely needed if
longer than 6 weeks post injury; may consider repair in any case as based on Daluiski et
al although most would perform reconstruction if over 6 weeks from injury
i. Daluiski et al – Reported primary repair of ligaments with success in 34 patients
even in chronic setting > 30 days after injury;
b. Combined medial and lateral ligament insufficiency
i. Single ligament graft reconstruction
1. Van Riet et al - drilling 2 ulnar tunnels; gracilis autograft; posterior
midline approach; reported single and double loop techniques
2. Finkbone PR, O’Sriscoll SW – “Box loop” reconstruction
a. Donar tendon is passed through the humerus and ulnar an dtied
back to itself
b. Fourteen cases with mean follow-up of 64 months. Nine
patients returned to the clinic and were evaluated. 5 patients
phone follow-up
c. Average follow-up time 64 months. 7 elbows were normal and
4 greatly improved, 2 improved and 1 worse.
ii. Separate reconstructions – medial and lateral
b. DJD II
i. Panpandrea, Morrey, O’Driscoll JSES 2007;16:68-77
1. 21 patients with chronic instability after
coronoid fracture dislocation assessed at a
minimum of 2 years after reconstruction
2. 13/21 patients had a successful outcome – only
1 of 7 with delay greater than 7 weeks was
considered a success
3. 19 fractured RH, 11 of which were originally
treated with RH excision
4. 16 patients with coronoid fx dislocation treated
with hinge; 12/16 considered successful; only
50% remained reduced with use of fixator
5. 71% complication rate
6. Delay beyond 7 – 8 weeks led to uniformly
unsuccessful results
7. Conclusions – persistent instability after
fracture dislocation of the elbow is a situation
to be prevented because reconstruction is
unpredictable.
c. DJD II and Orthofix
i. Sorensen et al JSES 2011;20:1300-9.
1. 20 patients with persistent instability after fx
dislocation treated at a mean of 11 weeks after
injury. Evaluated 17 elbows at a mean of 44
months postop; all had hinge fixator and
reconstruction of static stabilizers
2. 10/17 elbows had a good or excellent result; 96
degree ROM; MEPS 74; Patients treated within
6 weeks after trauma had better scores (MEPS
81 vs 62); no recurrent dislocations;
3. 41% complication rate
d. Orthofix–
i. Ruch et al Injury 2001-
1. 5 patients treated with hinge ex fix at 6 weeks
or greater post injury
a. repair or reconstruct all bony and
ligamentous injuries
b. flexion arc 84, congruent joint in all,
DASH 23
c.
e. Unilateral self centering fixator (not available yet)–
i. Bigazzi et al – New external fixator designed with a gear
to freely align itself withteh center of the elbow
rotation during passive flexion extension
1. 7 patients; no cases of misalignment, loss of
fixation, pin loosening or instability
3. Static
a. Place 2 humeral pins and 2 ulnar pins; humeral pins can be
placed directly posteriorly through the triceps to avoid the
radial nerve - Radial nerve is directly posterior at level of the
deltoid tuberosity; 11 to 14 cm proximal to the lateral
epicondyle the nerve moves from posterior to anterior
therefore direct posterior placement is safe 10 cm to the lateral
epicondyle; ulnar pins placed on dorsal subcutaneous border
b. Leave fixator in place for 4 to 6 weeks; remove and initiate
therapy protected by a hinged brace
4. Severe bone deficiency not reconstructable utilizing coronoid grafting
techniques; radial head insufficiency not allowing replacement due to
persistent subluxation of the proximal radial shaft; tenuous bony or
ligamentous fixation
VIII. Algorithm for Missed instability with combined injuries (authors preferred techinique)
a. Posterior midline approach
b. Ulnar nerve transposition
c. Lateral approaches – Kaplan and Kocher intervals extended proximally
d. Medial approach – through bed of the ulnar nerve
e. Coronoid reconstruction – if radial head absent – approach laterally through lateral
window; repair or reconstruct with olecranon tip or allograft radial head; if RH intact –
approach medially with Taylor Schamm approach
f. Radial head replacement or repair
g. Box-loop ligament reconstruction
h. IF stability not restored due to failure to restore RH, Coronoid or ligament
reconstruction – static ex fix; if self centering hinge becomes available would consider in
this situation
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