This document summarizes various esophageal diseases including their location, causes, symptoms, diagnosis, and treatment. It covers hiatal hernias, achalasia, esophageal diverticula, and esophageal neoplasms. The main points are:
1) Type I hiatal hernias are the most common, often in overweight women, and cause reflux that can lead to erosion, ulcers, scarring, and strictures. Treatment focuses on lifestyle changes and medications to reduce reflux.
2) Achalasia is a failure of the lower esophageal sphincter to relax, causing painless dysphagia and regurgitation. It is diagnosed via manometry and treated with
This document summarizes various esophageal diseases including their location, causes, symptoms, diagnosis, and treatment. It covers hiatal hernias, achalasia, esophageal diverticula, and esophageal neoplasms. The main points are:
1) Type I hiatal hernias are the most common, often in overweight women, and cause reflux that can lead to erosion, ulcers, scarring, and strictures. Treatment focuses on lifestyle changes and medications to reduce reflux.
2) Achalasia is a failure of the lower esophageal sphincter to relax, causing painless dysphagia and regurgitation. It is diagnosed via manometry and treated with
This document summarizes various esophageal diseases including their location, causes, symptoms, diagnosis, and treatment. It covers hiatal hernias, achalasia, esophageal diverticula, and esophageal neoplasms. The main points are:
1) Type I hiatal hernias are the most common, often in overweight women, and cause reflux that can lead to erosion, ulcers, scarring, and strictures. Treatment focuses on lifestyle changes and medications to reduce reflux.
2) Achalasia is a failure of the lower esophageal sphincter to relax, causing painless dysphagia and regurgitation. It is diagnosed via manometry and treated with
Type/Location Cause Symptoms Diagnosis Treatment & Recurrence
HIATAL HERNIA Type I- Sliding -Type I are more common -LES becomes incompetent and -many pts have no symptoms -suspected based on pt hx -avoid gastric stimulants than type to by 100:1 gastric juice reflux produces a -can cause a burning epigastric or -weight loss is a feature die to -eliminate tight garments that -predominantly in women chemical burn in the esophagus substernal pain/tightness distal esophageal stricture) ↑ intra-abdominal P who have been pregnant -degree of mucosal injury is due to -can be confused with MI, but -hh’s and reflux esophagitis can be -do not eat/drink within -men and women due to ↑ duration of acid contact and NOT usually pain does not radiate confirmed by fluoroscopy during a several hrs of sleeping intra-abdominal P a disease of hyperacidity -continued inflammation of the barium swallow -sleep with the head of the bed -Type I hiatal hernias are distal esophagus may lead to: -esophagogastric endoscopy and elevated at least 6in often in overweight pts 1. mucosal erosion biopsy of inflamed esophagus -weight loss in obese pts 2. unlceration -manometry may show a loss of the -regular use of antacids to 3. scarring lower esophageal high pressure provide steady protection 4. stricutre area -H blockers (increase the pH -other presentations: of the gastric juice reflux) 3. lump or feeling that food is -metoclopramide (may be stuck beneath the xiphoid helpful when poor gastric 2. worse when the patient is emptying is a symptom) supine or leaning over *1/3 of pts don’t respond to 3. antacid therapy frequently medical tx and ½ of those improves the symptoms who respond will relapse and 4. alcohol, aspirin, tobacco, require surgery and caffeine exacerbate sxs -surgery: correct anatomic 5. late dysphagia & vomiting defect and prevent reflux usually suggest strictures (reconstruction of a valve) Type II- -generally show no symptoms until *complications of surgery: Para-Esophageal they incarcerate and become 1. dysphagia ischemic 2. gas-bloat syndrome: -symptoms present include: inability to belch/vomit 1. dysphagia 3. disruption of the repair 2. bleeding (recurrent symptoms) 3. occasional resp. distress 4. intra-abd infection 5. esophageal perforation 6. splenic injury MOTILITY DISORDER Achalasia -failure of high-P zone -painless dysphagia -1st confirmed -medical tx has generally not (LES) sphincter to relax -progressive dilation of the roentgeno-graphically by contrast been helpful -not due to spasm proximal esophagus studies (“bird beak” appearance) -invasive endoscopy (forceful -regurg. of undigested food and -dilation of proximal esophagus & dilation) spitting up foul smelling poss. diverticula at any level -esophageal myotomy secretions on leaning forward -endoscopy (must be careful to (surgical transection of m) -weight loss avoid diverticular perforations) -aspiration pneumonia -esophageal manometry GI- Surgical Aspects of Esophageal Disease Diverticulum -2nd most common of the -distal 1/3 of esophagus: -pulsion diverticula complain of: -pulsion: excision of the -Pulsion esoph. motility disorders esophagogastric jxn problem due 1. regurgitation of recently diverticula and myotomy of (Cervical/Zenker’s) -either pulsion or traction to chronic stricture from: swallowed food or pills the cricopharyngeal m. -Traction depending on the mech that 1. acid reflux 2. choking *excision should always be causes development 2. anti-reflux surgery 3. putrid breath odor accompanied by correction of -pulsion diverticula are 3. achalasia underlying path process related to dysfunction of the cricopharyngeal m. -middle 1/3: almost always traction -traction: asymptomatic -traction: don’t need tx (not related to an intrinsic abnormality in esoph. motility), caused by: 1. mediastinal inflammation (inflam. nodal dis. from TB) 2. histoplasmosis (results in scar formationcontracture traction on esophagus) NEOPLASMS Benign -exceedingly rare -leiomyoma: grow progressively -leiomyoma: indent the lumen of -Leiomyoma -leiomyomas are the most and cause dysphagia the esoph. on contrast study common intramural tumors (potential for malignant degeneration is low) Malignant -usually arises from -commonly occurs in black men in -symptoms have insidious onset, -barium contrast of the esoph. -very poor prognosis (both) -Squamous cell c squamous epithelium association with alcohol and/or precluding early diagnosis and the *apple core lesion is -upper esophageal cancer: -Adenocarcinoma tobacco abuse opportunity for effective tx patho-gnomonic of esoph radiotherapy is the 1˚ tx -in other parts of the world the -as the tumor enlarges progressive carcinoma (usually requires extirpation etiology has been related to: dysphagia becomes the -endoscopy and biopsy of lesion of esoph. en bloc w/the 1. diet predominant symptom -CT of chest and upper abdomen is larynx, permanent 2. vitamin deficiency -other symptoms: used to assess the extent of tumor tracheostomy, and restoration 3. poor oral hygiene 1. pain involvement (important to stage of swallowing- palliation) 4. surgical procedures 2. weight loss the disease) -middle 1/3: tx by a staged 5. # of premalignancies 3. difficulty swallowing procedure w/total thoracic (burns, barrett esophagitis, 4. acquired tracheaesophageal esophagectomy & bypass radiation, P-V syndrome, fistula (due to erosion of the -lower 1/3: esophagogastric and esophageal diverticula) tumor in trachea/bronchus) resection & end-to-end ~10% of pts with Barrett’s develop 5. frequent pneumonia (due to anastomosis in mid chest adenocarcinoma recurrent aspiration) DIAPHRAGMATIC HERNIA Bochdalek Larrey TRAUMATIC DISORDERS Traumatic Rupture of GI- Surgical Aspects of Esophageal Disease the Diaphragm Esophageal -instrumentation by endoscopic -sxs of Boerhaave’s syndrome: -tx requires aggressive surgical Perforation and/or biopsy 1. draumatic or occult intervention -blind nasogastric tubes 2. profound shock -instuments designed for dilation of 3. mediastinal sepsis strictures 4. severe chest or abd pain -Sengstaken-Blakemore tubes 5. hypotension (balloon dilation for achalasia) 6. diaphoresis -Boerhaave’s syndrome 7. nausea/vomiting (spontaneous perforation secondary to forceful vomiting) Ingestion of Casutic *medical emergency -alkaline containing products -inspect mouth to assess injury -neutralization and induced Materials (drano, liquid plummer, etc.) emesis not recommended -corrosive gastritis: due acetic acid -perform endoscopy, airway or hydrochloric acid maintenance, and patency of the esophagus -NO steroids