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Emergency Delivery and Peripartum Emergencies
Emergency Delivery and Peripartum Emergencies
Emergency Delivery and Peripartum Emergencies
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SECTION XII WOMEN’S HEALTH AND GYNECOLOGIC DISEASES
Even though multiple physical interventions may be neces- shoulder. Once clear, the posterior shoulder is typically deliv-
sary with an abnormal delivery, an uncomplicated one typi- ered spontaneously or with upward traction in a similar
cally only requires measures that support smooth fetal passage. manner as just described. At this point the largest fetal diam-
Resuscitation equipment should be available immediately. eter has passed, and labor is generally smooth. The cord may
Episiotomies are no longer recommended during routine be clamped once it is accessible after passage of the fetus. If
pregnancies. They should be used sparingly and typically only the cord is tightly wound around the fetus, knotted, or abnor-
with complicated deliveries. If shoulder dystocia is encoun- mal in any way, it is clamped (typically at 7 and 10 cm) and
tered, hyperflexion of the maternal hips and knees (McRoberts cut once accessible. When free, the neonate is dried and
maneuver) and suprapubic pressure are first-line interven- warmed and its resuscitation needs evaluated. If available, a
tions that resolve most instances of dystocia. neonatologist should be present at any ED delivery.
Three classic signs indicate delivery of the placenta: sudden
lengthening of the cord, a gush of blood, and a change in the
shape of the uterine fundus. Once the fetus is clear, gentle
For more information on the management of diffi-
cult labor, see www.expertconsult.com traction on the cord should continue until these signs are seen.
After the placenta is delivered, typically within 20 minutes, it
is inspected for irregularities that may suggest retained tissue.
Manual abdominal massage of the uterine fundus will often
After determination of hemodynamic stability, the next pri- assist in uterine contraction. An infusion of oxytocin, typically
orities are to determine whether true labor is occurring and 20 units in a 1000-mL bag of normal saline infused at about
the appropriate disposition to achieve optimal medical care. 200 mL/hr, can be administered to hasten separation and
For any patient with a complaint of passage of clear fluid from assist in contraction of the uterus back into the pelvis.
the vagina without other signs and symptoms of labor (bloody
show and regular, progressing, often painful contractions), a
sterile speculum examination should be performed before a FOLLOW-UP, NEXT STEPS IN CARE,
gloved digital examination to evaluate for PROM. AND PATIENT EDUCATION
Once labor is confirmed, the goal is to evaluate the position-
ing (orientation in space relative to the maternal pelvis) and After the placenta is delivered and the neonate is stabilized,
presentation (body part palpable at the cervix) of the fetus, the mother should be evaluated for bleeding—the causes and
along with the degree of change in the uterine cervix. This treatment of which are covered next. If hemodynamically
includes assessment of station (level of decent into the pelvis stable after the first postpartum hour, the mother may be
relative to the maternal ischial spines), effacement (degree of transferred to the postpartum floor.
cervical thinning), and dilation of the cervical aperture. For
the emergency physician, determination of dilation and efface-
ment is the most important part of the examination—a fetus POSTPARTUM HEMORRHAGE
that is still contained within a closed and minimally effaced
cervix will probably be transferred to obstetrics whether or
not it is vertex (fetal head as the presenting part). EPIDEMIOLOGY
If delivery is imminent, the patient will have to remain in the
ED. A gynecologic bed with lithotomy position capability is Hemorrhage is a significant cause of maternal morbidity and
ideal, and a resuscitation bay with greater accessibility and is the second most common cause of peripartum deaths (fol-
equipment is recommended. A radiant warmer and appropriate lowing amniotic fluid embolism). Hemorrhage was a direct
airway equipment should be available. Positioning of the cause of more than 18% of 3201 pregnancy-related maternal
mother may require an approximate 10-degree tilt to the left to deaths in the United States from 1991 to 1997.3 Worldwide,
prevent uterine pressure on the inferior vena cava and associ- hemorrhage has been identified as the single most important
ated hypotension. When crowning occurs, the mother should cause of maternal death and is responsible for almost half of
be instructed to push along with the contractions, with the all postpartum deaths in developing countries.4
physician positioned in front of the introitus ready to accept Postpartum hemorrhage is the term used to describe exces-
the fetus. As soon as the head is accessible, continuous gentle sive blood loss after delivery. Classically, it is defined as more
countertraction should be administered to maintain it in a than 500 mL of blood loss in a vaginal delivery or more than
flexed position. This technique provides control of an explosive 1000 mL of blood loss in a cesarean delivery; however, careful
delivery, as well as avoidance of the high morbidity associated quantitative measures reveal that blood loss in the range of
with fetal neck hyperextension. Though once recommended, 500 to 1000 mL is actually average for both types of delivery.
the modified Ritgen maneuver has recently been shown to be Of note, practitioners often underestimate blood loss by as
associated with an increased rate of third-degree lacerations much as 50%.5 For the ED physician, the most important
and episiotomy in comparison with a “hands-off ” approach.2 causes of hemorrhage in the postpartum period are birth
Similar rates of perineal tears were found for each modality. trauma, uterine atony, uterine rupture, and uterine inversion.
When the head is clear, the fetus rotates 90 degrees. Suction
of the fetal mouth and nose should be performed as soon as
possible in the setting of meconium staining. Typically, the PATHOPHYSIOLOGY
shoulders will then be delivered, anterior first, without assis-
tance. Mild downward traction on the torso (not lateral flexion Separation and delivery of the placenta constitute the third
of the neck) may be required to assist passage of the anterior stage of labor. With separation of the placenta, there is also
1052
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CHAPTER 122 Emergency Delivery and Peripartum Emergencies
severance of the numerous uterine arteries and veins that carry intravaginal, intraperitoneal, and retroperitoneal accumulation
600 mL/min of blood through the intervillous space. The most of blood can be overlooked.
important factor for hemostasis is contraction and retraction In a reported case series on uterine inversion, the most
of the myometrium to compress and obliterate the open common signs were shock and hemorrhage.6 With a complete
lumens of the vessels. inversion, the prolapsed uterus may be visible as a large, dark
Uterine atony, the most common cause of postpartum hem- red polypoid mass within the vagina or protruding through the
orrhage, is the result of a hypotonic uterus after delivery. introitus. If the fundus remains within the vagina, the diagno-
Factors that lead to uterine overdistention or that interfere with sis may be suspected because of dimpling, indentation, or
uterine contractility can cause uterine atony and are associated absence of the uterine fundus on abdominal examination or
with postpartum hemorrhage. Although it is important to keep because a mass is palpated in the cervix on bimanual exami-
these associations in mind, most cases of postpartum hemor- nation. Establishing the diagnosis of incomplete inversion can
rhage occur without any known predisposing factors. be quite difficult; severe hypotension, postpartum hemor-
Trauma to the genital tract during labor and delivery (lac- rhage, and subtle abnormalities on abdominal examination
erations of the perineum, vagina, vulva, or cervix) is the may be the only clues.
second most common cause of postpartum hemorrhage. Uterine rupture is also a difficult clinical diagnosis and
Abnormal placentation (placenta accreta, increta, or percreta) should be considered in any patient with unexplained peripar-
can contribute to postpartum hemorrhage in different ways: tum hemorrhage or hypotension. The classic findings of
(1) an adherent placenta or large blood clots prevent effective uterine rupture are “ripping” or “tearing,” suprapubic pain and
contraction of the myometrium, thereby impairing hemostasis tenderness, absence of fetal heart sounds, recession of the
at the implantation site, and (2) significant bleeding from the presenting parts, and vaginal hemorrhage. Signs and symp-
implantation site is more likely with placental separation of toms of hypovolemic shock and hemoperitoneum may follow.
abnormally adherent tissue. This classic manifestation is actually rare; 87% of patients
Uterine inversion is prolapse of the uterine corpus to or with uterine rupture have no pain and 89% have no vaginal
through the cervix. Uterine inversion may be incomplete bleeding. Pain is also an unreliable finding because of the
(inverted but does not go through the cervix) or complete (the altered response to noxious intraperitoneal stimuli by a
fundus protrudes through the cervix). In the more extreme stretched abdominal wall. Fetal distress is the most consistent
cases, the entire uterus may prolapse out of the vagina. Most finding (80% to 100%), with fetal bradycardia being the most
cases are acute and occur immediately after delivery and common sign.7 Most reports of uterine rupture describe
before the cervical ring constricts. If inversion occurs (or is patients with normal blood pressure or even elevated blood
noted) after cervical contraction, the inversion is termed sub- pressure without tachycardia. Abnormal maternal vital signs
acute. Chronic inversion takes place weeks after delivery. The are late indicators of severe hemorrhage. The most important
majority of cases of uterine inversion occur as a result of trac- risk factor for uterine rupture is a previous uterine scar; other
tion on the umbilical cord during removal of the placenta. factors are listed in Box 122.1.
Acute, significant hypotension is common.
Uterine rupture is classified by the degree (complete or
incomplete) and cause (spontaneous or traumatic) of the BOX 122.1 Risks for Uterine Rupture
defect. A complete uterine rupture is defined as a full-thickness
tear of the uterine wall and overlying serosa; it is associated Surgery or Procedure
with life-threatening maternal and fetal compromise. After Previous cesarean delivery (most common by far—1 in 125
complete rupture, the uterine contents may escape or partially subsequent pregnancies)
extrude from the uterus and into the peritoneal cavity. An Previous uterine rupture
incomplete uterine rupture is defined as uterine muscle sepa- Previous myomectomy incision (1 in 40 subsequent
ration with an intact visceral peritoneum (often from uterine pregnancies)
scar dehiscence). In an incomplete rupture, hemorrhage fre- External or internal version
quently extends into the broad ligament, which has a tam- Breach extraction
ponading effect. Forceful uterine pressure during delivery
Trauma or Environmental
Trauma
PRESENTING SIGNS AND SYMPTOMS
Cocaine use
Peripartum patients may lose a substantial amount of blood Structural Anomaly
before becoming hypotensive or feeling symptomatic. Imme- Congenital fetal or uterine anomaly
diate postpartum hemorrhage should be recognized as a Previous invasive molar pregnancy
potential complication of a precipitous delivery. The classic Uteroplacental abnormalities: adenomyosis or placenta
clinical manifestation is a woman with sudden massive vaginal increta or percreta
bleeding who is tachycardic, pale, and possibly diaphoretic or Pregnancy Related
hypotensive. However, three factors make this classic mani- Silent rupture in a previous pregnancy
festation flawed. First, an elevated pulse and decreased blood Persistent, intense contractions (spontaneous or iatrogenic)
pressure are insensitive indicators until large amounts of High parity (1 in 100 subsequent pregnancies); grand multi-
blood have been lost. Second, many patients will have steady parity (>7) increases the risk for rupture 20-fold8
bleeding that, although moderate in appearance, may escape In vitro fertilization
notice until serious hypovolemia develops. Third, intrauterine,
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SECTION XII WOMEN’S HEALTH AND GYNECOLOGIC DISEASES
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CHAPTER 122 Emergency Delivery and Peripartum Emergencies
Abdominal pain
Consider uterine
and/or shock
rupture CT if stable (see
ABCs, w/out
Suspected retroperitoneal UR section)
notify OB, significant
postpartum hematoma
bloodbank, vaginal bleed
hemorrhage
IR
Mass protruding Replace uterus
Uterine inversion
from or in vagina (see UI section)
Associated sudden
Consider AFE See AFE section
CV collapse?
No Placenta
Explore
uterus delivered?
Yes
Consider Extract
placenta placenta Fundus Fundus
accreta firm? soft?
Try other
oxytocics:
Bimanual Methergine, OB available
Presumed
compression, Hemabate, to go to
uterine atony
stabilize for OR Prostin E2, or OR/IR?
and probable misoprostol
hysterectomy No
Consider atony
treatment Consider
and rule out temporizing
other causes measures:
balloon,
Inspect for uterine pack,
cervical, factor VII,
vaginal, aortic compression
or vulvar
lesions
Uterine rupture
See UR
suspected or
Vaginal vulvar Laceration No lesions section
confirmed
hematoma found
Retained
≤4 cm and ≥4 cm and Repair if Explore Remove
placenta
stable growing possible uterus
or clot
Consider
coagulopathy,
Ice and Drain, No DIC (bedside
pack, and Still
recheck abnormalities test or empiric
recheck bleeding
found treatment)
Fig. 122.1 Algorithm for postpartum hemorrhage management strategies. ABCs, Airway, breathing, and circulation; AFE, amniotic
fluid embolism; CT, computed tomography; CV, cardiovascular; DIC, disseminated intravascular coagulation; IR, interventional radiology;
OB, obstetrics; OR, operating room; UI, uterine inversion; UR, uterine rupture.
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SECTION XII WOMEN’S HEALTH AND GYNECOLOGIC DISEASES
Uterotonic
Methylergonovine 0.2 mg IM, may repeat q2h (max Ergot Contraindicated in those with
(Methergine, 5 doses) preeclampsia or hypertension
Ergonovine)
Carboprost 0.25 mg (250 mcg) IM, may Prostaglandin (15-methyl May cause transient O2 desaturation;
tromethamine repeat q15-90min (max 8 prostaglandin F2α analogue) contraindicated in those with active
(Hemabate) doses) cardiac, renal, pulmonary, or hepatic
disease
Misoprostol (Cytotec) 600-1000 mcg SL or PR; single Prostaglandin (prostaglandin E1 May cause bronchospasm
dose analogue)
Prostaglandin E2 20-mg uterine suppository, may Prostaglandin May transiently decrease blood
(Dinoprostone or repeat q2h pressure
Prostin E2)
Factor VIIa 35-200 mcg/kg, may repeat q2h Human recombinant factor VIIa
(NovoSeven) until hemostasis achieved
Tocolytic
Magnesium sulfate 4 g IV over 20-min period, then Magnesium enzymes Monitor for side effects; has prolonged
2- to 3-g/hr infusion cofactor effects; calcium gluconate may be
needed to reverse tocolysis when
the uterus in place
Terbutaline 0.25 mg SC, may repeat q20min Terbutaline beta-agonist May cause hypotension; tachycardia
common; hold if heart rate >120
beats/min
Nitroglycerin 50 to 100 mcg IV, may repeat as Nitroglycerin nitrate Rapid onset and half-life; transient
needed hypotension; have ephedrine (10 mg
IV) available as remedy
cupped in the operator’s palm, and firm upward pressure is bleeding has stopped. Antibiotics should be started as
applied to move the uterus up through the cervix along the soon as practical. Uterotonics are continued for at least 24
natural curve of the pelvis toward the umbilicus until it is in hours.
place. Usually when the inverted mass is pushed upward, the If all other efforts have failed to reposition the inverted
uterus automatically reverts, with the fundus returning to its uterus, operative intervention is required. If the uterus was
anatomic position. If the placenta has not separated, it should repositioned with the placenta attached, manual removal can
not be removed. be attempted once the use of relaxants is stopped. Uterotonics
If initial repositioning is unsuccessful, myometrial relax- should be initiated, and if the placenta cannot be removed
ation with pharmacologic agents should be attempted. Mag- easily, it should be left in place.
nesium sulfate, terbutaline, and nitroglycerin (attractive
because of its easy availability and short half-life) are the UTERINE RUPTURE
agents most commonly used (see Table 122.1).13 Attempts at Initial management of uterine rupture differs and is based on
manual repositioning of the uterus should continue. the stability of the patient and fetus. Because the results of
After successful reduction, the uterus should be supported examination are normal in most patients with uterine rupture
for several minutes to allow the ligaments to return to and because fetal distress is the most common finding, initial
their original state while uterotonics are administered. If management will probably be the same as that for other causes
magnesium sulfate was administered as a tocolytic, calcium of acute fetal distress—urgent delivery.
gluconate can be given to reverse the tocolytic effect. Fluid The physician should consult obstetrics on an emergency
and blood replacement and manual uterine massage should basis, ensure adequate IV access, notify the blood bank, and
be maintained until the uterus is well contracted and the alert the neonatal team to be ready for intensive care newborn
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CHAPTER 122 Emergency Delivery and Peripartum Emergencies
Uterine packing Layer sterile gauze within the uterus, with the distal May adhere to the uterine wall and removal required;
end going out through the os does not allow monitoring of ongoing bleeding;
start prophylactic antibiotics
Foley catheter Insert a large bulb catheter (24 French) into the uterus Multiple catheters may be needed (in a sterile
Instill with 80-100 mL of saline overbag), which makes the inner lumen difficult to
Pack the vagina to avoid expulsion of the catheter monitor
SOS Bakri balloon Insert into the uterus Best option if available; allows direct measurement
Instill 300-500 mL of saline through the stopcock of ongoing bleeding via the open inner lumen;
Pack the vagina developed for postpartum hemorrhage; balloon
conforms to the shape of the uterine cavity
Sengstaken-Blakemore Cut off the distal (“stomach”) end of the tube Does not conform to the shape of the uterine cavity;
tube Insert inside the uterine cavity with the end cut off, proximal bleeding can be
Infuse 75-300 mL of saline monitored through the lumen; may be available
Pack the vagina to avoid expulsion of the tube from the gastrointestinal department laboratory if
not available in the emergency department
Rusch catheter Using a 60-mL bladder syringe, inflate the balloon via Urologic catheter used for bladder stretching; may
the drainage port with 150-500 mL of saline be available in the urology department
Pack the vagina to avoid expulsion of the tube
Condom catheter Slide the condom over the end of the Foley catheter A sterile rubber catheter is fitted with a condom
and tie it off with string to close the end
Inflate with 250-500 mL of saline and clamp the end
Pack the vagina to avoid expulsion of the tube
Vaginal packing Pack the vagina with a blood pressure cuff placed Various techniques have been described; concern
inside a sterile glove for bleeding proximal to the vaginal pack
Increase pressure to 10 mm Hg above systolic blood
pressure
Noninflatable antishock Begin application at the ankles and progress Adjust the panels if any discomfort or dyspnea;
garment sequentially up to the abdomen contraindicated in women with heart failure or
mitral stenosis
resuscitation. If these resources are not available in the hos- mother, as well as pediatric or neonatal services for the
pital, arrangements should be made for immediate transfer. newborn. All patients who deliver in the ED should be admit-
For unstable patients, prompt, aggressive resuscitation is an ted to an obstetric floor after delivery or stabilization of the
important temporizing measure until definitive surgical repair acute complication.
is performed. Fetal morbidity almost invariably occurs because
of catastrophic hemorrhage, fetal anoxia, or both. In a study
of 99 cases of uterine rupture, the best fetal outcomes were PERIMORTEM CESAREAN DELIVERY
noted when surgical delivery was accomplished within 17
minutes from the onset of fetal distress.7
If the patient is stable, no signs of fetal distress are present, or EPIDEMIOLOGY
the diagnosis is unclear, ultrasonography can be useful. Ultra-
sound findings include lack of normal orientation, uncertain pla- Maternal arrest is estimated to occur in 1 in 4000 to 6500
cental location, fetal demise, and absence of amniotic fluid. pregnancies in the United States.14-16 In any maternal arrest
that occurs beyond 24 weeks’ gestation, perimortem cesarean
delivery should be considered as a potentially lifesaving inter-
FOLLOW-UP, NEXT STEPS IN CARE, vention for both the mother and the fetus.17
AND PATIENT EDUCATION When comparing causes of death in the cohort of women
undergoing perimortem cesarean delivery with all maternal
Obstetric consultation should be obtained on an emergency deaths, trauma accounts for a larger proportion in the former
basis to assist in the delivery and postpartum care of the group.18 Perimortem cesarean delivery is one of the oldest
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SECTION XII WOMEN’S HEALTH AND GYNECOLOGIC DISEASES
• ABCs
Is UI acute?
Uterine • 2 large-bore IV lines
(before cervical
inversion • STOP any oxytocin
ring contracts)
• Notify OB
Yes
• Manually support uterus
for several minutes Uterus Immediate attempt No
• Discontinue tocolytics in place to reduce uterus
• Start uterotonic meds
(see Table 122.1) Failed
attempt
Remove placenta if
necessary (if not Reattempt to
abnormally adherent) reduce uterus when
once uterus contracts patient is stable
and bleeding is controlled
Failed
attempt
Surgical
repositioning
of uterus
Fig. 122.2 Algorithm for uterine inversion management strategies. ABCs, Airway, breathing, and circulation; IV, intravenous; OB,
obstetrics; UI, uterine inversion.
surgical procedures in history, with the first reference to a DIFFERENTIAL DIAGNOSIS AND MEDICAL
successful postmortem cesarean section recorded in 237 DECISION MAKING
bce.19 Under the emperors of Rome, the Caesars, a law
decreeing that a child be excised from the womb of any Perimortem cesarean delivery is indicated for gravid patients
woman who died late in pregnancy became known as the “lex more than 24 weeks pregnant (or the uterine fundus is four
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CHAPTER 122 Emergency Delivery and Peripartum Emergencies
60
40 50
tissue. Fetal-maternal tissue transfer is common and probably
20 normal. It is proposed that when fetal antigens breach a mater-
0 0 nal immunologic barrier in some women, release of endoge-
0-5 6-10 11-15 16-25 nous mediators is triggered, and an anaphylactoid syndrome
can occur.24
Time interval (minutes)
Neurologic damage is seen in as many as 85% of survivors
% Survival of anaphylactoid syndrome.25 The mechanism of neurologic
% Neuro intact injury is thought to be severe hypoxia leading to encephalopa-
thy and seizures. The increased metabolic demand concurrent
Fig. 122.3 Perimortem cesarean delivery: fetal outcomes in with seizures (seen in 50%) may worsen the brain injury,
relation to time interval from maternal arrest to delivery. especially in the setting of hypoxia.3 Disseminated intravas-
cular coagulation developed within 4 hours of initial evalua-
tion in more than 80% of patients.25 If diffuse bleeding occurs,
fingerwidths above the umbilicus if the gestational age is hemorrhagic shock can contribute to the hypotension.
unknown) if arrest continues after 4 to 5 minutes of cardio-
pulmonary resuscitation. In a study by Katz et al.,17 among
the cases with available data on maternal hemodynamics PRESENTING SIGNS AND SYMPTOMS
during resuscitation, more than half the women had a “sudden
and often profound improvement, including return of pulse The classic manifestation of anaphylactoid syndrome of preg-
and blood pressure at the time the uterus was emptied.” The nancy is acute hypoxia or respiratory arrest with associated
study concluded that perimortem delivery should be per- cardiovascular failure, altered mental status, seizures, and
formed within 4 minutes of maternal arrest if resuscitation coagulopathy in the immediate peripartum period. It typically
was ineffective. An update published in 2005 strongly sup- occurs abruptly and with catastrophic outcomes, but less
ported this conclusion,17 and the 2010 American Heart Asso- severe forms of this syndrome have been described. In about
ciation guidelines for cardiac arrest associated with pregnancy 10% of patients the diagnosis is made postpartum, and it has
agree with this window (even sooner if it is clear that the been diagnosed as late as 48 hours postpartum.
mother has a grave or nonsurvivable injury).21
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SECTION XII WOMEN’S HEALTH AND GYNECOLOGIC DISEASES
TREATMENT
REFERENCES
No studies have shown that targeted intervention improves
maternal prognosis. Management includes early definitive References can be found on Expert Consult @
airway control with endotracheal intubation, IV fluids, www.expertconsult.com.
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CHAPTER 122 Emergency Delivery and Peripartum Emergencies
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SECTION XII WOMEN’S HEALTH AND GYNECOLOGIC DISEASES
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CHAPTER 122 Emergency Delivery and Peripartum Emergencies
A B
Fig. 122.5 When the scapulae appear under the symphysis, the
operator reaches over the left shoulder, sweeps the arm across the
chest (A), and delivers the arm (B). Gentle rotation of the shoulder
girdle facilitates delivery of the right arm (C). (From Gabbe SG,
Niebyl JR, Simpson JL, et al. Obstetrics—normal and problem
pregnancies. 5th ed. Philadelphia: Churchill Livingstone; 2007.)
C
occurs as the cord passes through the cervix or, rarely, from
tension applied around an entangled fetal part. The most
important factor in these situations is patency of the umbilical
vessels with sufficient blood flow. If a cord is tightly entrapped
or impacted on a structure, blood flow will be inadequate and
fetal hypoxia will result. Functionally, the only way to pre
vent fetal hypoxia in this situation is to restore umbilical
blood flow either by manually decompressing the cord or by
completing delivery so that the fetus is capable of oxygenating
independently.
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SECTION XII WOMEN’S HEALTH AND GYNECOLOGIC DISEASES
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CHAPTER 122 Emergency Delivery and Peripartum Emergencies
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SECTION XII WOMEN’S HEALTH AND GYNECOLOGIC DISEASES
Fig. 122.9 The least invasive maneuver to disimpact the shoulders is the McRoberts maneuver. Sharp ventral flexion of the maternal hips
results in ventral rotation of the maternal pelvis and an increase in the useful size of the outlet. (From Gabbe SG, Niebyl JR, Simpson JL,
et al. Obstetrics—normal and problem pregnancies. 5th ed. Philadelphia: Churchill Livingstone; 2007.)
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CHAPTER 122 Emergency Delivery and Peripartum Emergencies
A B
Fig. 122.11 Rubin or reverse Wood screw maneuver for shoulder dystocia. A, Rotate the posterior shoulder. B, Deliver the rotated
shoulder. (From Roberts JR, Hedges JR. Clinical procedures in emergency medicine. 5th ed. Philadelphia: Saunders; 2009.)
PREMATURE RUPTURE OF
MEMBRANES AND PRETERM
A
PREMATURE RUPTURE
OF MEMBRANES
EPIDEMIOLOGY
Rupture of fetal membranes occurring without evidence of
labor is called PROM. It occurs infrequently—less than 5%
of pregnancies are affected. If this event takes place before 37
weeks’ gestation, it is called PPROM. Factors that have been
associated with PROM include infections, maternal smoking,
placentation abnormalities such as abruption or previa, mal-
nutrition, and connective tissue disorders.
B PATHOPHYSIOLOGY
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SECTION XII WOMEN’S HEALTH AND GYNECOLOGIC DISEASES
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CHAPTER 122 Emergency Delivery and Peripartum Emergencies
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SECTION XII WOMEN’S HEALTH AND GYNECOLOGIC DISEASES
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