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HEALTH ASSESSMENT Assessment of the Nose

- Inspect and palpate the external nose for


color, shape, consistency, tenderness, and
Learning Outcome patency of airflow.
- Inspect the internal nose for color, swelling,
- knowing how to answer and ask logical exudate, bleeding, ulcers, perforated septum,
questions (communication) or polyps.
- develop clinical eye (observation)
Clinical Tip
- develop critical thinking
- Position the otoscopes handle to the side to
improve your view of the structure. If an
NOSE AND SINUSES otoscope is unavailable, use a penlight and
hold the tip of the nose slightly up. A nasal
speculum with a penlight also facilitates
Collecting Subjective Data good visualization.
- History of present health concern Structure of the Sinus
- Past health history
- Four pairs of paranasal sinuses—frontal,
- Family history
maxillary, ethmoidal, and sphenoidal are in
- Lifestyle and health practices the skull.
Equipment for Assessment of the Upper Respiratory - Sinuses decrease the weight of the skull and
Tract act as resonance chambers during speech.

- Gloves Assessment of the Sinuses


- Cotton gauze pads - Palpation for tenderness
- Penlight - Percussion and transillumination for air
- Nasal speculum attached to otoscope versus fluid or pus
- Tongue blade (Palpating the frontal sinuses and maxillary
sinuses.)
Physical Assessment Techniques
Clinical Tip
- Inspection
- Palpation - Upper dentures should be removed is that
- Percussion the light is not blocked

Structure of the Nose Abnormal Findings of the Nose and Sinuses


- Nasal Polyp
- Consists of an external portion covered with
- are soft, painless, noncancerous growths on
skin and an internal nasal cavity
the lining of your nasal passages or sinuses.
- External Nose: a bridge, tip, and two ovals’
- Perforated Septum
openings called nares
- A perforated septum is usually caused by
- Internal Nose: nasal cavity; nasal septum;
the loss of blood flow to both sides of the
Kiesselbach area; superior, middle, and
septum. This causes the cartilage to
inferior turbinate’s
deteriorate, which prevents proper airflow
and worsens as air slips through from one
nostril to the other through the hole.
THORAX AND LUNGS edema, medications to treat pulmonary
fibrosis.
- Cough up blood?
Structure of the Thorax and Lungs - Description of cough – dry, hacking
- Associative and Alleviating factors
- Thorax - Painful?
- Extends from the base of the neck - Shortness of Breath (SOB)
superiorly to the level of the diaphragm - Onset, associative factors
inferiorly • Determine how much activity precipitates
- Lungs, distal portion of the trachea, SOB
bronchi - Affected by position?
- Sternum • Orthopnea – difficulty breathing when
- Lies in the center of the chest anteriorly supine (heart failure?)
- Has three parts: - Time of day/night
• Manubrium, the body, xiphoid process • Paroxysmal nocturnal dyspnea –
- 12 pairs of ribs—thoracic cage awakening from sleep with SOB and
- Mediastinum needing to be upright to achieve comfort
- Central area in the thoracic cavity - Allergies?
- Lungs • Asthma attacks
- two cone-shaped, elastic structures - Alleviating factors
- Pleura - Chest pain with breathing?
- thin, double-layered serous membrane that • Location, onset, duration, frequency,
lines the thoracic cavity intensity, associative and alleviative factors.
- Past history of respiratory infections
- Smoking history
Vertical Reference Lines - Environmental exposure
- Self-care behaviors
- Anterior Chest • Immunizations, TB skin tests, chest X-rays
• midsternal, right and left midclavicular
lines Assessment-Inspection
- Posterior Thorax
- Inspect Thorax
• vertebral line, right and left scapular lines
- Symmetry
- Lateral Thorax
- AP Diameter
• midaxillary line, anterior and posterior
• Normal 1:2
axillary lines
• AP diameter = transverse diameter, “barrel
Health History chest”. Occurs with normal aging, chronic
emphysema, and asthma
- Cough - Symmetry and normal development of
- Onset? Gradual or sudden? Frequency? trapezius muscle
- Continuous throughout day – acute illness •Hypertrophied in COPD
(respiratory infection) - Position person takes to breathe
- Afternoon/evening – may reflect exposure • COPD – tripod position
to irritants at work
- Night – postnasal drip, sinusitis Assessing the Anterior Chest
- Early morning – chronic bronchial
- Symmetric chest expansion
inflammation of smokers
- Abnormally wide costal angle occurs with
- Sputum? How much? Characteristics?
emphysema.
• Chronic bronchitis – productive cough for
3 months of the year for 2 years in a row.
Yellow or green – bacterial infection, strep
throat. Rust colored – TB, pneumococcal
pneumonia. Pink, frothy – pulmonary
Posterior Chest
- Symmetric chest expansion Percussing and Auscultating Anterior Chest
- Ask person to place warmed hands-on
posterolateral chest wall with thumbs at - Begin percussing the apices in
level of T9 or T10 and pinch up small fold supraclavicular ares, continuing down in
of skin between thumbs. As person inhales, intercostal spaces
ask person to move thumbs apart - Note cardiac and liver dullness and stomach
symmetrically and take a deep breath. tympany
• Chronic emphysema leads to
Tactile Fremitus hyperinflation of lungs, resulting in
hyperresonance where you would expect
- Fremitus is a palpable vibration transmitted cardiac dullness
through the bronchi and lung parenchyma to - Auscultate lung fields down to the 6th rib.
the chest wall. Progress from side to side moving
- Abnormalities in fremitus downward and listen for one full respiration
- Decrease fremitus occurs when anything at each location
obstructs transmission of vibrations
• Obstructed bronchus Auscultating Posterior Chest
• Pleural effusion or thickening
• Pneumothorax - Breath sounds
• Emphysema - Instruct the person to breathe through the
- Increased fremitus occurs with compression mouth a little deeper than usual, but to
or consolidation of lung tissue stop if they feel dizzy. Hyperventilation
• Lobar pneumonia may lead to fainting!
- Rhonchal fremitus palpable with thick - Use the flat diaphragm endpiece of the
secretions stethoscope and listen for at least one full
- Crepitus occurs in subcutaneous respiration in each location
emphysema, when air escapes from the - Continue to think:
lungs and enters the body's nervous system - What am I hearing?
through the skin (subcutaneous), sometimes - What should I expect to be hearing?
called anaphylactic shock. • Bronchial
• Bronchovesicular
Percussion • Vesicular
- Do not confuse background noise with lung
- Start at the apices and percuss across tops of sounds
both shoulders and down the lung region at • Stethoscope tubing bumping together
approx. 5cm intervals. • Shivering
- Make a side-to-side comparison • Hairy chest
- Avoid damping effect of scapulae and ribs. • Rustling of gown
- Resonance predominates in healthy lungs. - Characteristics of normal breath sounds
• Hyperresonance is found when too much • High > loud > inspiration\expiration >
air is present (emphysema or pneumothorax) harsh, hollow tubular > Trachea and larynx
• Dullness signals abnormal density • Moderate > moderate >
(pneumonia, pleural effusion, atelectasis, inspiration=expiration > mixed > over major
tumor) bronchi where fewer alveoli are located
- Expected percussion notes • low > soft > inspiration/expiration >
• Resonance rusting like the sound of the wind in the
• Flat over scapula trees > over peripheral lung fields where air
• Visceral dullness flows through smaller bronchioles and
• liver dullness alveoli.
- Percussion notes
- Clinical example:
- Pulmonary edema, pneumonia, pulmonary
Auscultation fibrosis, depressed cough reflex
- Abnormal findings
- Decreased breath sounds Pleural Friction Rub
• Obstruction of bronchial tree (by - Description
secretions, mucous plug, foreign body) - Coarse and low-pitched superficial sound.
• In emphysema due to loss of elasticity in Both inspiratory and expiratory.
the lung fibers and decreased force of - Mechanism
inspired air. The lungs are already - Caused when pleurae become inflamed and
hyperinflated so not much air will be coming lose normal lubricating fluid. Pleural
in. surfaces rub together during respiration.
• Obstruction of sound by pleural thickening Heard best in anterolateral wall.
• Silent chest – no air moving in or out - Clinical example:
- Increased breath sounds louder than normal - Pleuritis
- Bronchial sounds
• Heard in abnormal location, such as Wheeze
periphery - Description
• High pitched, with prolonged expiratory - High pitched musical squeaking sound
phase predominantly during expiration
• Occur in consolidation (pneumonia) or - Mechanism
compression (fluid in intrapleural space). - Air squeezed or compressed through
Dense lung tissue enhances transmission narrowed airways (collapsing, swelling,
of sound. secretions, tumors)
Auscultating Adventitious Sounds - Clinical example:
- Acute asthma or chronic emphysema
- Adventitious sounds
- Sounds not normally heard in the lungs Rhonchi (sonorous)
- Caused by moving air colliding with - Description
secretions in trachea or bronchi, or from - Low-pitched, musical snoring
popping open of previously deflated - Mechanism
airways - Airflow obstruction
- Crackles (fine) - Clinical example:
- Description: popping sounds heard during - Bronchitis, obstruction of bronchus from
inspiration. May be stimulated by rolling a obstruction or tumor
strand of hair between fingers near the ear.
- Mechanism: Inhaled air collides with Stridor
previously deflated airway
- Description
- Clinical example:
- High pitched, inspiratory, crowing sound,
• Early inspiratory – COPD
louder in neck than over chest wall
• Late inspiratory – Pneumonia, heart
- Mechanism
failure, interstitial fibrosis
- Originates in larynx or trachea. Upper
Crackles (coarse) airway obstruction from inflamed tissue or
obstruction
- Description - Clinical example:
- loud, low-pitched, bubbling and gurgling - Croup and acute epiglottitis. Obstructed
sounds early in inspiration. Sound like airway.
Velcro.
- Mechanism
- Inhaled air collides with secretions in
trachea and large bronchi
Developmental Consideration
- Infants
- While infant is sleeping, can inspect and
auscultate the lungs
- Infants normally have a rounded thorax,
reaching a 1:2 (anteroposterior to transverse)
diameter by age 6
• If a barrel shape persists after age 6,
possible chronic asthma or cystic fibrosis
- If baby begins to cry, it actually enhances
the palpation of tactile fremitus.
- Pregnancy
- Wider thoracic cage
- The enlarging uterus elevates the
diaphragm 4 cm during pregnancy, but the
increased estrogen relaxes thoracic
ligaments allowing compensation by
increasing the transverse diameter
- Mother’s tidal volume increases to meet
demands of fetus
- Aging
- kyphosis
- calcification of costal cartilage
- decreased vital capacity
- decreased number of alveoli
- decreased mucous production

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