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Lecture 4 PharmD 2022 Part I
Lecture 4 PharmD 2022 Part I
1- Suppurative inflammation
i. Localized
ii. Diffused
2- Nonsuppurative inflammation
i. Catarrhal inflammation
ii. Membranous inflammation
iii. Fibrinous inflammation
iv. Serous inflammation
v. Sero-fibrinous inflammation
MD 406, 2022
1- Suppurative inflammation
(Septic, Purulent, Pyogenic
inflammation):
◦ Pus formation
◦ Caused by pyogenic
organisms:
Staphylococcus aureus
Streptococci
Pneumococci
Gonococci
Meningococci
MD 406, 2022
Pyogenic organisms TOXINS
Pus cells
Localized: Diffused:
e.g. Abscess, boil e.g. Cellulitis
(furuncle), carbuncle
Staphylococcus Streptococcus
aureus Hemolyticus
Coagulase Streptokinase (fibrinolysin)
Hyaluronidase (spreading
factor)
Abscess = cavity containing pus,
showing 3 zones:
MD 406, 2022
Localized Suppurative Inflammation
Abscess commonly occurs in the
subcutaneous tissue, but may occur in any
organ as lung, brain.
Sites of carbuncle
include back of neck,
scalp,more common
in diabetics.
MD 406, 2022
Fistula is a tract communicating the abscess
cavity & a hollow organ or between two hollow
organs e.g. ano-rectal fistula complicating a
preanal abscess.
MD 406, 2022
1- Catarrhal inflammation: thick exudate of
mucus and white blood cells caused by the
swelling of the mucous membranes usually
associated with common cold
MD 406, 2022
2- Serous inflammation: outpouring of watery,
low protein exudate/ Burns.
MD 406, 2022
4- Sero-fibrinous inflammation:
Excess fluid exudate rich in fibrinogen
MD 406, 2022
5- Membranous inflammation:
◦ Yellowish gray
Pseudomembrane
◦ Composed of necrotic
patches and excessive fibrin
due to severe inflammation
of the mucus membranes
MD 406, 2022
Fate of acute inflammation
Resolution Regression/Repair
Acute Chronic
Injury Inflammation Inflammation
Progression
Septicaemia
and spread
MD 406, 2022