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2008 Dsce-Study Guide
2008 Dsce-Study Guide
2008 Dsce-Study Guide
2008 DSCE
STUDY GUIDE
Self explanatory
2008 (updated) DSCE QUESTIONS & STUDY GUIDE 2
References & Editing by Jodi Pessoa, Marlin & Alan Walker
1. IL-1
4. self explanatory
5. Before trimming teeth on a stone cast for immediate denture fabrication you must
first:
6. Patients with renal disease will be unable to take any of the following
analgesics:
Aspirin
So, even at high levels renal effects may ensue< ACETOMINOPHEN is the only
7. self explanatory
8. self explanatory
154. Best position for a finish line on a short clinical crown is: SUBGINGIVAL
(Finkbeiner, Betty Ladley Finkbeiner. Comprehensive Dental Assisting. Elsevier, 1995. 36.8.4). <vbk:0-8151-3239-5#outline(36.8.4)>
2008 (updated) DSCE QUESTIONS & STUDY GUIDE 3
References & Editing by Jodi Pessoa, Marlin & Alan Walker
Figure 16-45
Tetracycline stain. Note the yellow color (tetracycline) of the posterior teeth
and the gray color of the anterior teeth, in which there has been oxidation of
endogenous tetracycline.
(Regezi, Joseph A. Regezi. Oral Pathology: Clinical Pathologic Correlations, 4th Edition. Elsevier, 2002. 17.8.2). <vbk:0-7216-
9805-0#outline(17.8.2)>
FLUOROSIS STAINING –
(Regezi, Joseph A. Regezi. Oral Pathology: Clinical Pathologic Correlations, 4th Edition. Elsevier, 2002. 17.4.1).
<vbk:0-7216-9805-0#outline(17.4.1)>
Figure 16-30
(Regezi, Joseph A. Regezi. Oral Pathology: Clinical Pathologic Correlations, 4th Edition. Elsevier, 2002. 17.4.2).
<vbk:0-7216-9805-0#outline(17.4.2)>
A pulp stone is a calcification that occurs in the coronal region of the pulp (Fig.
33-8) . A diffuse or linear calcification in the form of a spicule may occur in the
pulp canal and is usually aligned near a vessel or nerve. Typically, pulp stones
have no clinical significance; however, they may increase in frequency and size
with age or with irritation. Large pulp stones can complicate gaining access to a
canal during root canal therapy.
(Finkbeiner, Betty Ladley Finkbeiner. Comprehensive Dental Assisting. Elsevier, 1995. 33.2.11).
Cracked tooth syndrome is transient acute pain experienced occasionally while
chewing. Difficult to locate and reproduce. Likely to occur among individuals
who crack nuts and crush ice with their teeth, and among popcorn eaters. Usually
a vertical crack or split in the tooth extends across a marginal ridge through the
crown and into the root, involving the pulp. Visible by transilluminated light or
with the use of disclosing dyes. (Mosby. Mosby's Dental Dictionary. Elsevier,
2004.). Occurs on minimally restored teeth and extends from the mesial to the
distal direction> you can use the tooth slooth to tell you about cracked tooth also.
Tooth sloth is:
2008 (updated) DSCE QUESTIONS & STUDY GUIDE 5
References & Editing by Jodi Pessoa, Marlin & Alan Walker
FIG. 9-19
A, The tooth Slooth is used to detect an incomplete fracture of a posterior tooth. The end
with the small divot or depression is placed on the suspected cusp. The patient is
instructed to close so that the opposite tooth engages the grooved, flat side of the Tooth
Slooth. B, The patient is instructed to apply biting pressure on the plastic instrument and
to move the mandible slowly side to side and then release quickly. C, Tooth that yielded a
positive response to the rubber wheel test, indicating a possible incomplete fracture. D,
Removal of existing restoration confirms diagnosis of an incomplete fracture.
(Roberson, Theodore Roberson. Sturdevant's Art and Science of Operative Dentistry, 4th Edition. Elsevier, 2002.). vbk:0-323-01087-
Clinically, the gingiva appears initially bluish red and cyanotic, with a rounding and
tenseness of the gingival margin; then it increases in size, most often in the
interdental papilla and partially covering the crowns of the teeth
(Newman, Michael G. Newman. Carranza's Clinical Periodontology, 9th Edition. Elsevier, 2002. 12.3.2).
Staphne defect is also called lingual salivary gland defect. Remember this “lesion” will show up
on the radiograph below the IA canal indicating that it is not of odontogenic origin>
Lab values:
White blood cell count 4,000 to 11,000 cells/ mm3
RBC count 45-60 million cell/mm3
Hgb Men: 14-18 g/dL Women: 12-16 g/dL
Platelets 150,0000- 440,000/ mm3
((Thrombocytopenia <150,0000 platelets/mm3))
((spontaneous bleeding < 20,000 platelets/mm3 ))
Bleeding time 4-9 mins [>15 mins abnormal]
PTT Intrinsic and common pways 40-100 secs [120 secs abnormal]
aPTT PTT wt activator added to test tube25-35 secs
ACT Intrinsic and common pways 90-120 secs
PT Extrinsic and common pways 10-12 secs
INR Extrinsic and common pways 2.0-3.0 secs
2.5-3.5 secs (for pts on high dose
anticoags or with
__________________________________________mechanical heart valves)
Sickle cell anemia- increase in bone trabeculation, hair on end in the skull x-ray
Increased film speed will reduce radiation exposure but decrease clarity; so don’t
use F speed use D or E.
“Varicosities become progressively prominent with age; thus, lingual varicosities are
encountered in elderly individuals. Lingual varicosities appear as tortuous serpentine
2008 (updated) DSCE QUESTIONS & STUDY GUIDE 7
References & Editing by Jodi Pessoa, Marlin & Alan Walker
blue, red, and purple elevations that course over the ventrolateral surface of the
tongue, with extension anteriorly. Even though they may be quite striking in some
patients, they represent a degenerative change in the adventitia of the venous wall and
are of no clinical consequence. They are painless and are not subject to rupture and
hemorrhage.” (Greenberg, Martin S. Greenberg. Burket's Oral Medicine Diagnosis and Treatment, 10th Edition. BC
Decker, 2002. 6.1.2).
FIG. 7-2
Varix; note multiple lingual varicosities. (From Wood NK, Goaz PM: Differential diagnosis of oral lesions ,
ed 3, St Louis, 1985, Mosby.) (Finkbeiner, Betty Ladley Finkbeiner. Comprehensive Dental Assisting. Elsevier, 1995.).<vbk:0-8151-
3239-5#F90>
o Hyperpituiatarism
o Hyperparathyroidism
o Osteomalacia
o Pagets
o Fibrous dysplasia
o Massive osteolysis
Herpangina:
2008 (updated) DSCE QUESTIONS & STUDY GUIDE 8
References & Editing by Jodi Pessoa, Marlin & Alan Walker
Figure 7-23 ♦ Herpangina. Numerous aphthouslike ulcerations of the soft palate. (From Allen CM, Camisa C:
Diseases of the mouth and lips. In Sams WM, Lynch P, editors: Principles of dermatology, New York, 1990, Churchill Livingstone.)
(Neville, Brad Neville. Oral and Maxillofacial Pathology, 2nd Edition. Elsevier, 2002.).
Herpangina begins with an acute onset of significant sore throat, dysphagia, and fever,
occasionally accompanied by cough, rhinorrhea, anorexia, vomiting, diarrhea, myalgia,
and headache. Most cases, however, are mild or subclinical. A small number of oral
lesions, usually two to six, develop in the posterior areas of the mouth, usually the soft
palate or tonsillar pillars (Figure 7-23). The affected areas begin as red macules, which
form fragile vesicles that rapidly ulcerate. The ulcerations average 2 to 4 mm in diameter.
The systemic symptoms resolve within a few days; as would be expected, the ulcerations
usually take 7 to 10 days to heal. (Neville, Brad Neville. Oral and Maxillofacial Pathology, 2nd Edition. Elsevier,
2002. 7.6.1). <vbk:0-7216-9003-3#outline(7.6.1)>
Because herpangina is self-limiting, is mild and of short duration, and causes few
complications, treatment usually is not required. (Regezi, Joseph A. Regezi. Oral Pathology: Clinical
Pathologic Correlations, 4th Edition. Elsevier, 2002. 2.1.4.3). <vbk:0-7216-9805-0#outline(2.1.4.3)>
EAGLE”S SYNDROME
FIG. 9-70
2008 (updated) DSCE QUESTIONS & STUDY GUIDE 9
References & Editing by Jodi Pessoa, Marlin & Alan Walker
Even when extensive ossification of one or both stylohyoid ligaments is seen, more than
50% of patients are clinically asymptomatic. The ossified ligament usually can be
detected by palpation over the tonsil as a hard, pointed structure. Very little correlation
exists between the extent of ossification and the intensity of the accompanying
symptoms. One symptom is vague, nagging to intense pain in the pharynx on swallowing,
turning the head, or opening the mouth, especially on yawning. When this entity is
associated with discomfort and the patient has a recent history of neck trauma (e.g.,
tonsillectomy), the condition is called Eagle's syndrome. (White, Stuart C. White. Oral Radiology, 5th
Edition. Elsevier, 2003. 31.7.1.2). patient ay also complain of dizziness.
CPR:
Although KS is a malignant tumor, in its classic form it is a localized and slowly growing
lesion. The KS that occurs in HIV-infected patients presents different clinical features. In
these individuals, it is a much more aggressive lesion and the majority (71%) develop
lesions of the oral mucosa, particularly the palate and gingival. The oral cavity may often
be the first or only site of the lesion. GUESS it can occur on the tongue…
(Newman, Michael G. Newman. Carranza's Clinical Periodontology, 9th Edition. Elsevier, 2002. 29.4.3).
AIDS and the periodontium. A, Hairy leukoplakia on the lateral margin of the tongue,
causing a corrugated appearance. B, Painless ANUG-like lesion of several months'
duration. The patient had a second ANUG-like lesion that was painful. C-E, ANUG-like
lesion and candidiasis of the palate and tongue in a 29-year-old woman. F, Kaposi's
sarcoma involving the anterior hard palate and right and left palatal mucosa. Candidiasis
is also noted on the hard palate. G, Same patient as in F, with Kaposi's sarcoma of the
labial gingiva presenting as a small purple nodule next to a parulis. H, Kaposi's sarcoma
involving the anterior facial gingiva and producing a gingival enlargement. (Courtesy Dr.
Frank Lucatorto.)
(Newman, Michael G. Newman. Carranza's Clinical Periodontology, 9th Edition. Elsevier, 2002.). <vbk:0-7216-8331-2#P32>
OSTEORADIONECROSIS
2008 (updated) DSCE QUESTIONS & STUDY GUIDE 12
References & Editing by Jodi Pessoa, Marlin & Alan Walker
FIG. 18-2
Progressive course of osteoradionecrosis. A, Radiograph showing radiolucencies in
right mandible and around apex of molar tooth. B, Six months later, during which time
antibiotics and local irrigations were used, radiolucent process is spreading into ramus.
Molar was removed at this time. C, Five months after tooth removal, extraction site did
not heal and destructive process spread, resulting in pathologic fracture of mandible. D,
Radiograph after removal of devitalized bone, showing extent of process. (Courtesy Dr.
Richard Scoot, Ann Arbor, MI.)
(Peterson, Larry J. Peterson. Contemporary Oral and Maxillofacial Surgery, 4th Edition. Elsevier, 2002.).
-----------------------------------------------------------------------------------------
ECTODERMAL DYSPLASIA
FIGURE 5-10
A, a child with ectodermal dysplasia, in addition to the characteristic thin and
lightcolored hair, is likely to have an overclosed appearance because of lack of
development of the alveolar processes; B, panoramic radiograph of the same boy,
showing the multiple missing teeth. Oligodontia of this extent is almost pathognomonic
of ectodermal dysplasia.
(Proffit, William R. Proffit. Contemporary Orthodontics, 3rd Edition. Elsevier, 1999.).
>their crown shapes are characteristically abnormal (Figure 16-3). The incisor
crowns usually appear tapered, conical, or pointed, and the molar crowns are
reduced in diameter. Complete lack of tooth development (anodontia) has also
been reported, but this appears to be uncommon.
(Neville, Brad Neville. Oral and Maxillofacial Pathology, 2nd Edition. Elsevier, 2002. 16.1.1).
2008 (updated) DSCE QUESTIONS & STUDY GUIDE 14
References & Editing by Jodi Pessoa, Marlin & Alan Walker
Immediate denture things
These posterior extraction and other operatedon areas are allowed to heal for a short
time, usually only 3 to 4 weeks, before the preliminary impressions are made.
(Zarb, George A. Zarb. Prosthodontic Treatment for Edentulous Patients, 12th Edition. Elsevier, 2003. 9.5.1).
FIG. 21-48.
A, Root fracture in middle third of root with a 5-mm separation of the parts. B,
Approximately 1 hour after the injury the coronal portion of the tooth had been
repositioned under local anesthesia with finger pressure. The mesial-incisal-labial areas
of both central incisors were etched and self-curing resin was applied to hold the tooth in
position while this radiograph was made to confirm proper reapproximation of the
fractured root surfaces. C, Appearance of stabilized left central incisor at the time
radiograph was made to confirm good reapproximation. After satisfactory repositioning
was confirmed, the tooth was further stabilized using the bonded resin and wire
technique. This injury did not result in significant soft tissue trauma, and hemorrhage was
controlled easily. D and E, Radiograph and photograph reveal satisfactory progress 2
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weeks after the injury. F, Nine months after the injury the tooth was vital, sound in the
alveolus, and asymptomatic.
(McDonald, Ralph E. McDonald. Dentistry for the Child and Adolescent, 8th Edition. Elsevier, 2004.).
FIG. 21-47.
A, Root fracture is evident in the apical half of the central incisor, but parent did not seek
treatment until 2 weeks after the injury. The tooth was stabilized with a splint for 1
month. B, The tooth responded favorably to pulp testing and had this radiographic
appearance 1 month later. C, More than 2 years had elapsed when this radiograph
revealed a normal periapical appearance. The tooth responded normally to vitality tests,
and there was slight mobility but no sensitivity to percussion.
(McDonald, Ralph E. McDonald. Dentistry for the Child and Adolescent, 8th Edition. Elsevier, 2004.).
Telangiectasias on tongue
FIGURE 6-3 Multiple small purple papules of
hereditary hemorrhagic telangiectasia.
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References & Editing by Jodi Pessoa, Marlin & Alan Walker
(Greenberg, Martin S. Greenberg. Burket's Oral Medicine Diagnosis and Treatment, 10th Edition. BC Decker, 2002. 6.1.5).
Plate I.
1, Squamous cell carcinoma. 2, Same lesion as 1. 3, Squamous cell carcinoma. 4,
Squamous cell carcinoma. 5, Squamous cell carcinoma. 6, Squamous cell carcinoma. 7,
Squamous cell carcinoma. 8, Basal cell carcinoma. 9, Basal cell carcinoma. 10, Basal cell
carcinoma. 11, Adenoid cystic carcinoma. 12, Malignant mixed tumor of the parotid
gland. (5 courtesy S. Silverman, San Francisco; 11 courtesy D. Smith, North Conway, NH.) (Wood, Norman K. Wood.
Differential Diagnosis of Oral & Maxillofacial Lesions, 5th Edition. Elsevier, 1997.).
Rinn system
2008 (updated) DSCE QUESTIONS & STUDY GUIDE 19
References & Editing by Jodi Pessoa, Marlin & Alan Walker