Educate The Patient Then Go With The Patient To A Safe Place

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Remembered questions 20-11-2020 Ran

1-Did anesthesia for patient then they announced a tornado is coming and patient is local told you to
don't worry about it, it happens every year you can continue, what should you do next?

Continue the dental procedure

Let the patient sign a consent then continue

Educate the patient then go with the patient to a safe place

Leave the patient and clinic and go to a safe place

2-Dental hygienist works in an office owned by 3 dentist, the hygiene injured a patient how's responsible

Hygienist and the 3 doctors. (if is the assistant then the responsibility is the doctor in charge that day)

3-Got a picture of a red spot in the middle of the tongue and question was p had the same spot in the
palate for 6 weeks what is the diagnosis? (Depend on the picture)

Kaposi

Candida

syphilis

possible HPV or geographic tongue


4-Contraindication of Calcium hydroxide:

Symptomatic teeth, primary teeth,

5-Got question about PICO theory example with sealants

Population Intervention Comparison Outcome

6-Many questions about herpes simplex with pictures

7-Also about root sensitivity least effective: iontophoresis

8-1 mg of fluoride is how many ppm in water?

a. 10

b. .01

c. 1pp

9- Main function for sleep apnea device: CPAP machine delivers a constant flow of air through tubing
and a mask and into your airway. The CPAP machine creates enough pressure in your airway to hold the
tissue open, so your airway doesn't collapse. The soft, steady jet of air from the CPAP machine creates
enough pressure to keep the airway open.
10-Multiple myeloma Xray: Scoop lesion or pouch out lesion (first sign of MM is pain) affect the plasma
cells, patient major of 50, pain, inflammation, anemia, poor prognosis, Most common in black male,
Bence jones protein and amyloidosis (10%)

11- Scleroderma features the opinions were very confusing: autoimmune disorder, caused by excess
collagen production that leads to thickening and tightening of the skin, can also affect internal organs,
Mona Liza, microsomia. Widening of the PDL around the roots, bilateral reabsorption of the angle of the
mandible ramus, or complete reabsorption of the mandibular condyles and or coronoid process.

12 – In class 3 decrease with time: ANB (increase with time SNB)

13- Best to diagnose horizontal cervical bone loss and heath of the pulp? Pano, mesial angled (also can
be seen in a periapical or bitewing)

14-Couple of questions about pterygomandibular raphe: Insertion of the buccinator and superior
constrictor.

15- What not to give with Myocardial infarction: Epinephrine (you can give MONA, morphine, oxygen,
nitroglycerin, aspirin.

16- hyperventilation patient what not seen? (what happen: tingling, confusion, no give oxygen,
tachycardia, position the patient upright carpopedal spasm

17- DM patient what not seen? Postoperative bleeding, another option about the veins
18- why choose the color of restoration before you but the rubber dam? Because the tooth appears
more glossy or the rubber dam reflects different colors (also tooth look dehydrate)

19- Rubber dam show the soft tissue? Holes too close

20-Kids feel hyperactive after you give one carpule of lido with epinephrine? You inject the anesthesia
into a vessel

21 -6mm intruder max primary center what do you do: let it re-erupt unless is affecting the permanent
tooth buds.

21-Maximum time you can wait after traumatic necrotic primary teeth to do the treatment?

One Week, 2 weeks, 3 weeks, 4 weeks

22-Questions and lateral flap? When not to do it and also the advantage of it: not to do it: when donor
side have not enough attached gingiva, when you need to cover more than one tooth. When to do it:
when you have enough attaches gingiva, when you need to cover a single tooth, when the esthetic is
important.

23-Min space bet 2 implants: 3mm (1.5 bt implant and tooth)

24-Dentin dysplasia picture:

25-Neuropraxia: Mild injury with not axonal damage. Spontaneous recovery within 4 weeks.

26 type 1 bone where? Anterior mandible

27-Best place for implants: Anterior mandible

28- Male in his 30s, Pano with mixed diffuse radiopacity in the lower premolars area with vital teeth?
Condensing ostites, osteosarcoma

29- Question about adult kid responsible of his parents? When they have powder of attorney

30-do every year? TB test

31-bacteria in the root apex ? strict anaerobic

facultative anaerobic

32-subcondyle neck fracture the lateral pterygoid moves? Moving to the affecting side

33- non working interference what muscle? Pterygoid Lateral

34-lingual incline of buccal mand What movement? Working, non working LUBL

35-codeine works on what? Mu receptors

36 - opioids receptors? Kappa, Mu, Delta


37-Diphenhydramine MOA: inhibit the receptor to histamine

39-Different bet angina and MI: duration

40-Articaine (septo) metabolism: Blood plasma, Blood stream

41- Not a reason for a restoration to fall after 1 day? Too much light cure

42- Sealed bond by? Locking in the groove and pits, micromechanical bonds bet composite and enamel

43-A lot of questions about papilloma:

44- Case about AOT: less recurrent, related to impacted canine, beyond CEJ, 2/3max, 2/3 anterior, 2/3
female

45- questions about unbundling dentist separate treatments, upcoding: insurance charges more
sensitivity (same in Very importance file please revise it well)

46- max molar in cross bite you want to cement a band where the hook in the band should be?

lingual upper, buccal lower

47-DMF what The F common in?

White most

,black,Hispanic

48-how long you wait after bleaching to do a composite restoration

1 week

49- doesn't have fluoride?

.7ppm-1.2ppm?
50- white spot decay what do you do?

SDF, fluoride varnish

51- functions of tin and all small metal in amalgam

52-major connecter for what? Stability and rigidity


53-why we make the metal between the clasp and rest thick, for horizontal movement, or pervert
fracture: prevent fracture

54-what convex in denture polished side?

Buccal y labial flange

55-pt is after came to you after he fall and after treatment pt feels nauseous and headache what the
next step? Monitor for 24h if the pt still feel the symptoms should go the hospital, or should go
immediately because it's may be head concussion

56- pt slow pulse and unconciseness you put the pt in spine position and cleared the airway and gave
oxygen after 1 minute nothing changed what to do next? Increase the oxygen,pt may experience
hypoglycemia

57 - what with neuro lesions and has many phases or type? Seizures I forgot the rest of the opinions 😐
Most common seizure in children ʹ grand mal seizures (febrile) ͻ Febrile seizures, which occur in young
children & are provoked by fever, are the most common type of provoked seizures in childhood. Then,
generalized tonic-clonic (grand mal)

58-calculations of the max carpules of lidocaine

60-picture of compound odontoma

61-sialoliths in what gland

Subm warton duct


62- not bilateral swelling? Xerostomia , mumps, others tyroglosal cyst , sialolytiasis

63-description of arrested caries ?

remineralized lesions, hard, black/brown .

64-most recurrent? OKC

65 pt prognosis? SSC in lip - adenosacroma in palate

66-pt alcohol you ordered? INR

67-uses of Bupropion (Zyban) ?smoking quit

68 Xray sensor? Is critical or semi or non critical

69 - not true about cleft plate? Treat at birth- usually need ortho , start speech therapy at age 1.5 - 2

Cleft lip rule of 10’s: Surgery is performed when the child is at least 10 weeks of age, weighs at least 10
lb, and has at least 10 g/dL hemoglobin.

70- not true about combination syndrome? Increased vd

71- tight tongue what's the problem with mand denture? Speaking, mastication

Speaking

72 - best description of MB Root amputation

indicated in clas 3 furcation an first we need to do endo


73- glucocorticoids contraindication

diabetes, ulceras petic, VIH. ACTIVE INFECTIONS

74-pano for eagle syndrome

75- Diastema – reciprocal anchorage

76- Pagets and hypercementosis


77- Moa of penicillin – bactericidal , cell wall synthesis inhibitor, beta lactamase synthesis inhibitor

78-How much max NO can be given to kids50 %

79-pt eats 3-4 lemons a day ??erosion

80-• What is not included in ethics ada –

cost, credentials?

81-min root-crown ratio: 1:1

(ideal 1:2, but 2:3 is more realistic)

82-min space btw 2 implants:

3mm

entre diente y implante 1.5mm

83-Critical PH for caries :

5.5 (demineralization pH=5.5, remineralization pH>5.5) <pH es mas acido

84- 2 questions about paraphrasing

paraphrasing: repeat back what you hear


Q) which of the following is an effective means of ensuring the patient understands concepts
discussed at the treatment planning appointment? Ask the patient to explain the concept in their
own words

Q1) When should the dentist NOT use paraphrasing?


a. When trying to speak to a patient in his secon language
b. When the dentist is unset whit what patient says
c. When giving factual values
Ans C
Q) Which statement is NOT correct about “paraphrasing “? To put in your own words ( creo que
para ser correcta tiene que ser: “in one’s own words”, what someone has said

in fulcrum doc

85-collimator in xray?

collimation is the reason lead is used in x-ray machines

Its a metallic barrier with an aperture to reduce the size of the x-ray beam and therefore
the volume of irradiated patient tissue (reduce further unnecessary patient exposure)

86- dentist should stand how far from the patient ?

4-6 ft?

87- all can cause bacteremia except ?

Apex of root , scaling and curettage

88- pt with 3 mand molars ?

Fusion

concresence?

89- 54. Fear of choking because of dental treatment =

associated with anxiety


Busque
‍🤷🏻‍
‍‍ en Facebook y solo encontré esta pero todos difieren con la respuesta ♂
‍ ‍
Fear of choking related to dental treatment:
A.Needle phobia
B.Anxiety
C.Catastrophe
Pseudodysphagia, or the fear of choking, is sometimes confused with phagophobia, or the fear
of swallowing. Although both conditions involve the act of swallowing, the difference is in the
precise nature of the fear. Those with phagophobia are afraid of the swallowing process, while
those with pseudodysphagia worry that swallowing might lead to choking.
Pseudodysphagia and the Dentist
Many people who do not otherwise suffer from pseudodysphagia are afraid of choking during or
after a dental procedure. Those who have a more generalized fear of choking may find it difficult
or impossible to visit the dentist at all. These fears often contribute to a more generalized fear of
dentists.3
Common dental choking fears include choking on saliva, choking on dental instruments, and
choking on gauze. Some people are afraid that they will be unable to breathe or swallow while
their mouths are numb. Many people find that their fears worsen when the chair is tipped all the
way back.

90- rectangular wire use for what?

ortho wires

Root torque
Rectangular/square wires: gives root torque (more on rectangular)
Corrects vertical discrepancies (working arch wires), control crown & root movement
round 1: aliviar

round2: stainless steel mainenance

91-Patient has a new amalgam restoration, most likely experience after 1 day? Cold

92- you did a root canal for a pt after 2 years there is a preapical lesion no symptoms present? Root
canal, or monitor, only do treatment if it becomes symptomatic

93-Which of the following is correct about intra-pulpal anesthesia?

Back pressure

94-where to place an ant implant for better esthetics?

2-3 apical to the adjacent CEJ tooth , or coronal 1 m

95-INR for implant: 2.5

Esta pregunta esta más completa


You can only place one implant in a patient taking warfarin. The INR should be
A. 2.5
B. 3.5- when patient is not taking anticoagulant
C. 7
D. 12

Ans A
>4 cannot treat send to doctor
heparin ptt

96- You suspect a drug allergy of dental product problem- whom do you report to –

FDA

osha

epa

97- Gtr for which furcation 2

3 wall defect gtr also

98-penicillin allergy what type of hypersensitivity

type 1 hypersensitivity reaction

all four?

99- turner from?

Birth trauma , local trauma and infection

para mi es esta porq tiene que ver dientes primarios)


Canine/premolar: by infection
Anteriors: by traumatic injury
If Turner's hypoplasia is found on a canine or a premolar, the most likely cause is an infection
that was present when the primary tooth was still in the mouth. Most likely, the primary tooth
was heavily decayed and an area of inflamed tissues around the root of the tooth affected the
development of the permanent tooth. The appearance of the abnormality will depend on the
severity and longevity of the infection.
If Turner's hypoplasia is found in the anterior area of the mouth, the most likely cause is a
traumatic injury to a primary tooth. The traumatized tooth, which is usually a maxillary central
incisor, is pushed into the developing tooth underneath it and consequently affects the formation
of enamel. Because of the location of the permanent tooth's developing tooth bud in relation to
the primary tooth, the most likely affected area on the permanent tooth is the facial surface.[3]
White or yellow discoloration may accompany Turner's hypoplasia.

100- Gives color to teeth: dentin

101- main reason for maintenance phase? Prevent recurrent - assets from the initial treatment

to achieve long-term stability of results and to minimise recurrence.

102-. Which of the following is Point A (Description)?

deepest concavity on anterior profile maxilla


103-first 24 hours after a free gingival graft, the graft gets its vascularization from

after a free gingival graft, primary source of nutrition for graft during the first 24 hours

Esta es la pregunta correcta:


After a free gingival graft, primary source of nutrition for graft during the first 24 hours is

A primary vascular anastomosis


B residual nutrients within the graft
C diffusion of nutrients from the underlying connective tissue
D diffusion of nutrients from adjacent vessels of the periodontal ligament
Ans: C

104-. Which of the following is reported to a side effect of antihypertensive drugs and is a

reason that there is low compliance with taking the medication? a. Baldness

b. Sexual dysfunction

105-For a patient with asthma which is the best position for the patient?

a. Supine

b. Sitting up and slightly forward

upright position

dont give patient opiods, ibuprofen,

The steps in emergency management of an acute asthmatic episode are: Terminate treatment
and remove all dental materials and instruments from the patient's mouth. Sit the patient upright
or in a comfortable position with the arms thrown forward over a chair back

106- whats the use of surgical template for implants: to know proper angle position and diagnose and
treatment plan, and 2nd surgical procedure

NOT for # of implants

A surgical template is a guide used to assist in the proper surgical placement and angulation of
dental implants. However, a surgical guide not only facilitates implant placement but can also be
used for other purposes, including diagnosis, treatment planning, and even second-stage
surgery.
107-7 y girl first dental visit, mom complains about bleeding with brushing? Leukemia ALL , choric
gingivitis

108- Which of the following provides pulpal analgesia? eugenol

109- question about beading complete denture

Purpose - Fabricate a cast that Preserves the peripheral role


Provides a protective rim around the cast (land). This rim (land) must be 4-6 mm wide.
Produce a dense cast of vacuum mixed stone of the proper dimension (10- 15 mm thick) to
permit flasking.

Beading is done to preserve width and height of sulcus in a cast and boxing is done to obtain a
uniform smooth well shaped base of the cast.
Boxing can be defined as the enclosure (box) of an impression to produce the desired size and
form of the base of the cast and to preserve desired details.
Beading and boxing final impressions before pouring preserves the extension, as well as the
thickness, of the border; controls the form and thickness of the base of the cast; and boxing also
facilitates placing remounting plates in the cast; and conserves artificial stone. It ensures the
capture of the mucobuccal and mucolingual borders of impression.

bps impression, once ran on yeso add wax. PAD roll up wax like snail

to fabricate a cast impression with proper measurements and preserve periphery?

boxing: cera aplanada

beading: floor of wax

110- stage 2 hypertension?


140/90

stage 1: 130-139/80-89

141-159/

120/80 normal

111- Fordyce granules are intraoral sebaceous glands: ectopic sebaceous gland

1) Hyperparathyroidism oral manifestations

brown tumor, loss of bone density, soft tissue calcification, and dental abnormalities

Fragile teeth with widened pulp chamber. Loss of lamina dura, loss of teeth, soft tissue calcifications, loss
of jawbone density
2) radiograph questions were hard - where they showed xray, pointed to a structure and asked what it
was. I got an "oropharnyx" air space on my radiograph that I did not identify correctly

Adenoid hypertrophy?

3) It is important to balance occlusion in all eccentric movements in a denture during

1) bruxism 2) swallowing 3) at rest

Chewing or bruxism?
4) prophylaxis for angina

Calcium channel blockers: amlodipine, verapimi SR, diliatezam SR,

Nitrates: isosorbide mononitrate 20mg/12hrs, transdermal NTG

Isosorbide dinitrate (oral) 10-40mg TDS

Nitroglycerin oral SR (6.25-12mg) 2-4 times/day

Acute attack: NTG 0.5mg S/L sublingual bucal spray

(not beta blockers for vasospastic angina)

5) Sturge Weber oral manifestations

rare, congenital, neuro-oculo-cutaneous disorder which is characterised extra-orally by unilateral port


wine stains on the face, glaucoma, seizures and mental retardation, and intra-orally by ipsilateral
gingival haemangioma which frequently affects the maxilla or mandible
7) Bells Palsy nerve involved, what supplies taste sensation and how to treat it

Supplied by chords tympani facial nerve

Tx with antiviral and steroids like prednisone

8) Tramadol drug interactions

Oxycodone

Fentanyl

Benzos

Opiods

9) **ORAL** bisphosphonates, what precautions do you take -- ask patient to reduce dosage before
extractions or do you go ahead and do all the extractions

All patients should be asked about the current or past use of bisphosphonate drugs and the
mode of administration because IV bisphosphonate have a longer half life and patients on
IV mode are at more risk for development of ONJ than patients on oral bisphosphonate.
2.
Patients yet to start with bisphosphonate therapy should be first examined for requirement of
any surgical dental procedures prior to the therapy, if the risk factors allows. Hopeless
teeth should be removed. Subgingival scaling should be performed. Poorly fitting
dentures should be replaced to avoid soft tissue trauma. Comprehensive treatment should
be performed to minimize the need for future dental treatment.
3.
For patients who have already started with the therapy, any elective procedures should be
avoided if possible to avoid the risk of bisphosphonate induced osteonecrosis of jaw.
Root canal treatment should be done rather than dental extraction when possible.
4.
Patient should be routinely examined radio graphically for osteonecrosis and baseline data
should be recorded for the patient. Certain laboratory test may help to monitor markers of
bone turnover and can help in diagnosis and risk assessment of developing
bisphosphonate associated osteonecrosis. Bisphosphonates reduces the level of CTx (C-
telopeptides) which are fragment of collagen released during remodelling and skeleton
turnover. So by assessing the serum CTx levels risk assessment can be done34 (Table 1).
Table 1
CTx serum value and risk factors for osteonecrosis.

CTx serum value (pg/ml) Risk for osteonecrosis

300–600 (normal) None

150–299 None to minimal

101–149 Moderate

<100 High

Patients in which dental extractions are unavoidable should be first consulted with the prescriber of
bisphosphonate therapy for possible temporary interruption of drug if beneficial. Extraction should be
done as atraumatically as possible and flap raising should be avoided. Sterile technique has to be
followed. Patient should be kept on chlorhexidine mouthwash twice daily for two months and
postoperatively 2 month follow up should be done. In some cases it has been recommended to do
root canal of the teeth followed by coronal amputation and leave the roots

The basic mechanism of development of osteonecrosis is that due to osteoclastic inhibition necrotic bone
cannot be resorbed by the osteoclast during normal course of healing and the necrotic bone which
remains, affects the blood supply to the area. If the patient has been taking medicine for more than 3
years it has been recommended to stop the medicine for at least 3 months before carrying out any
surgical procedure and once the healing is complete the drug can be taken.

Zolendronate (IV) is the most potent bisphosphonate because of its high mineral binding
affinity and FPPS enzyme inhibition whereas pamidronate is less potent.17–20 Orally
administered bisphosphonates include etidronate, risedronate, tiludronate, alendronate.

10) Tx of class II furcation --> resorbable collagen Guided tissue regeneration

The resorbable GTR membrane with bone material was more effective than open debridement alone, in
the treatment of furcation defects

11) 8 year old Pt with asthma taking albuterol --> what oral manifestations is a side effect

Candidiasis

Xerostomia

● Xerostomia: Dry mouth or xerostomia is defined as an overall reduction of salivary


output. It is an adverse effect observed with use of beta-2 agonists,[7] anticholinergic
inhalers,[21] and inhaled corticosteroids.[22] Patients complain of difficulty in talking or
swallowing, altered taste, generalized oral discomfort, mouth soreness, burning sensation,
and poor retention of artificial dentures. Also seen are generalized erythema of the oral
mucosa and a lobulated appearance on the dorsum of the tongue.[23] Xerostomia is
clinically presented as oral fissuring, ulceration, and epithelial atrophy.
● Dental caries: An infectious microbiologic disease of the teeth that results in localized
dissolution and destruction of the calcified tissues.[24] Under normal conditions, the
tooth is continually bathed in saliva. Saliva is supersaturated with calcium and phosphate
ions and capable of remineralizing the very early stages of caries formation, particularly
when the fluoride ion is present.[25] Fluoride slows down the progression of caries.
When salivary flow is diminished or absent, there is increased food retention. Since
salivary buffering capacity has been lost, an acidic environment is encouraged and
persists longer. This in turn encourages aciduric bacteria which relish the acid conditions
and continue to metabolize carbohydrate in the low pH environment. The stage is set for
uncontrolled carious attack. Dental caries is thus caused due to imbalance between saliva,
plaque, tooth, microflora, and dietary substrate over a certain period of time.
It has been found that higher risk of caries is seen in the more developed mixed or
permanent dentition of the individuals on the inhalation therapy.[26,27] The initial lesion
is a reversible incipient caries seen as chalky white appearance on the tooth surface. This
can progress rapidly into a grayish or blackish discoloration finally leading to cavitated
caries. It is seen frequently on the labial surfaces of the anterior teeth and on the occlusal
surfaces of the posterior teeth.
● Oral mucosal changes: With the use of inhaled corticosteroids, oropharyngeal
candidiasis[28] occurs as a potential adverse effect. Most commonly, seen as
pseudomembraneous lesion (thrush), it clinically presents as white, soft plaques that
leaves a painful erythematous, eroded, or ulcerated surface. The common sites are buccal
mucosa, oropharynx, and lateral aspects of tongue. Patients may complain of tenderness,
burning, and dysphagia once the pseudomembrane gets disrupted. The use of inhaled
corticosteroids can also result in throat irritation, dysphonia, cough,[5] dryness of oral
cavity and rarely, tongue enlargement.[29]
● Ulceration: Ulceration of the oral mucosa is seen mainly due to xerostomia and
immunosuppression caused by inhaled drugs.[30,31] Before the appearance of an ulcer,
the involved area produces burning or tingling sensation. A lesion is surrounded by an
erythematous halo and is covered by a yellowish fibrinous membrane. They develop over
the movable mucosa of the oral cavity.
● Taste disturbances: Oral mucosal diseases including candidiasis, prophylactic drugs
such as nedocromil, and anticholinergics[18] are important etiologic factors. Xerostomia
produces taste changes secondary to incomplete food solubilization and by diminished
transport of tastant molecules to taste buds.
● Halitosis: Bad breath could be due to oral infections and xerostomia.[32]
● Gingivitis and periodontitis: Increased level of gingivitis is observed with the use of
inhaled corticosteroids.[33,34] Mouth breathing habit in these patients further increases
gingivitis due to dehydration of the alveolar mucosa. Higher prevalence of periodontal
diseases has been reported possibly due to the pathological activation of the immune
system, inhaled drugs, or an interaction between them.[30] Saliva plays an important role
in restricting the periodontal disease, thus any drug affecting the salivary secretion
remarkably affects the severity of the periodontal disease.[3,35]
● Others: Adrenal insufficiency and growth impairment[36] are possible adverse effects of
inhaled corticosteroids. Acute adrenal insufficiency has been significantly reported in
individuals on inhaled corticosteroid therapy.[37] It may lead to severe hypotension,
nausea, and shock

Gastro-esophageal reflux due to beta-2 agonists increases the chances of dental erosions.

Bronchospasm B2-agonists – short acting Albuterol (Proventil, Ventolin, Oral candidiasis, xerostomia,
decreased AccuNeb) salivary flow rate, increased heart rate, nervousness, tremor, headache,
palpitations, elevated blood pressure, nausea, dizziness, heartburn, throat irritation, and nosebleeds.

12) Drug class C for pregnancy

A No risk in controlled human studies: Adequate and well-controlled human studies have failed
to demonstrate a risk to the fetus in the first trimester of pregnancy (and there is no evidence of
risk in later trimesters).

B No risk in other studies: Animal reproduction studies have failed to demonstrate a risk to the
fetus and there are no adequate and well-controlled studies in pregnant women, or animal
studies have shown adverse effects, but adequate and well-controlled studies in pregnant women
have failed to demonstrate a risk to the fetus in any trimester.

C Risk not ruled out: Animal reproduction studies have shown an adverse effect on the fetus
and there are no adequate and well-controlled studies in humans, but potential benefits
may warrant use of the drug in pregnant women despite potential risks.

D Positive evidence of risk: There is positive evidence of human fetal risk based on adverse
reaction data from investigational or marketing experience or studies in humans, but potential
benefits may warrant use of the drug in pregnant women despite potential risks.

X Contraindicated in pregnancy: Studies in animals or humans have demonstrated fetal


abnormalities and/or there is positive evidence of human fetal risk based on adverse reaction
data from investigational or marketing experience, and the risks involved in use of the drug in
pregnant women clearly outweigh potential benefits.

N FDA has not yet classified the drug into a specified pregnancy category.
13) I had an x-ray on Stafne inclusion

The Stafne defect (also termed Stafne's idiopathic bone cavity, Stafne bone cavity, Stafne bone
cyst (misnomer), lingual mandibular salivary gland depression, lingual mandibular cortical defect,
latent bone cyst, or static bone cyst) is a depression of the mandible, most commonly located on the
lingual surface (the side nearest the tongue). The Stafne defect is thought to be a normal anatomical
variant, as the depression is created by ectopic salivary gland tissue associated with the submandibular
gland and does not represent a pathologic lesion as such.

14) How to differentitate between radicular cyst and lateral perio cyst 1) location 2) vitality 3) appearance

Radicular cyst (periapical cyst): Odontogenic cyst derived from cell rests of Malassez that proliferate in
response to inflammation

Most common location: maxillary anterior region, maxillary posterior region, mandibular
posterior region, mandibular anterior region (i believe in this order)

Constitutes ½ to ¾ of all cysts in the jaws. Frequency is 60-70% between ages 20-60 rare in <10 year

olds. male/female ratio: 3:2


Maxilla is 3 times more affected than mandible

Tx: root canal, extract, enucleation, marsupilization

Lateral periodontal cyst: developmental.

“Lateral periodontal cysts (LPCs) are defined as non-keratinized and non-inflammatory developmental
cysts located adjacent or lateral to the root of a vital tooth.” LPCs are a rare form of jaw cysts, with the
same histopathological characteristics as gingival cysts of adults (GCA).

More common in mandibular premolar region


Always with vital teeth

tx.excision

15) If condylar guidance is VERY steep, and normal incisal guidance, what happens to compensating
curve 1) flat 2) Reversed 3) steep, they were more options
16) best way to diagnose pulpitis on primary tooth

1. Visual and tactile examination of carious dentin and associated periodontium

2. Radiographic examination of a. periradicular and furcation areas b. pulp canals c. periodontal space d.
developing succedaneous teeth

3. History of spontaneous unprovoked pain

4. Pain from percussion

5. Pain from mastication

6. Degree of mobility

7. Palpation of surrounding soft tissues

8. Size, appearance, and amount of hemorrhage associated with pulp exposures From the diagnostic
factors, the pulpal conditio

3. Reversible Pulpitis (Syn: hyperemia, inflamed-reversible) A pulpal condition is commonly induced by


dental caries and operative procedures, in which the patient responds to thermal or osmotic stimuli, but
the symptoms disappear when the etiology is eliminated.

4. Irreversible Pulpitis a) Irreversible pulpitis without periapical pathosis A pulpal condition, usually caused
by deep dental caries or restorations, in which spontaneous pain may occur or be precipitated by thermal
or other stimuli. Radiographs show no periapical changes. The pain lasts for several minutes to hours. b)
Irreversible pulpitis with periapical pathosis A pulpal condition similar to above, but in which periapical or
lateral radiographic changes are evident.

5. Necrotic Pulp a) Necrotic pulp without periapical pathosis A pulpal condition in which there may or may
not be spontaneous, moderate to severe pain or pain elicited by various stimuli. Response to various
testing modalities is usually absent. Radiographic changes are not evident.

b) Necrotic pulp with periapical pathosis A pulpal condition similar to above, except that in
this category periapical or lateral lesions are evident in radiographs.

read ALL questions from El-Maestro and mental dental Know them EXTREMELY well before the exam. I
got many repeated questions but also got many questions I've never seen before.

Pray for me, please! I'll post more questions from what I remember.. thats all I remember for now. hehe

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