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  • Visual Pathway Lesion

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    Msd Kishor
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    1. Visual pathway lesions can occur at multiple locations, including the optic nerve, chiasm, and optic tracts. 2. Complete optic nerve lesions result in total blindness on the side of the lesion, while partial lesions cause corresponding visual field defects such as a central scotoma. 3. Lesions of the optic chiasm or beyond can result in hemianopic visual field defects, depending on the location and extent of the lesion. Bitemporal hemianopia indicates a more central chiasmal lesion.

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    1. Visual pathway lesions can occur at multiple locations, including the optic nerve, chiasm, and optic tracts. 2. Complete optic nerve lesions result in total blindness on the side of the lesion, while partial lesions cause corresponding visual field defects such as a central scotoma. 3. Lesions of the optic chiasm or beyond can result in hemianopic visual field defects, depending on the location and extent of the lesion. Bitemporal hemianopia indicates a more central chiasmal lesion.

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    5 views9 pages

    Visual Pathway Lesion

    Uploaded by

    Msd Kishor
    1. Visual pathway lesions can occur at multiple locations, including the optic nerve, chiasm, and optic tracts. 2. Complete optic nerve lesions result in total blindness on the side of the lesion, while partial lesions cause corresponding visual field defects such as a central scotoma. 3. Lesions of the optic chiasm or beyond can result in hemianopic visual field defects, depending on the location and extent of the lesion. Bitemporal hemianopia indicates a more central chiasmal lesion.

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    of 9

    Visual pathway lesions

    ⑪ optic Nerve

    ⑦
    complete it optic Nerve lesion:
    ↳ R

    IIIIII),
    I/ L Anopia

    ⑬
    Equliest lesion:
    ↳
    only affect fibre
    Mostsensitive
    xeehemia
    ↳ R

    ↳ R

    II central scotoma

    complete optic
    #
    Nerve lesion # Partial optic Nerve lesion
    ↓
    ↓
    corresponding
    IIL Anopiq scotoma

    Amaurotic pupil (AA4D)


    I

    Marcus run pupil


    (RAPD)

    central scotoma
    internathy
    causes of

    comes dystrophy [stargardt's d15]


    H
    colour Blindness + c.scotoma
    Toptic=Heteronymous]
    chiasma
    Middle chiasmal
    syndrome
    [BIL Nasal fibre]

    ↳ R

    1, "I
    Bitemporal HemiAnopia
    (0R)
    Temporal Heteronymous
    Hemi Anopia

    variat : Equliest lesion in Pitutary Adenoma


    ↓
    Inferior BIL Nasal fibre
    Graniophary
    ↓
    I *M ⑭ optic
    chiasma

    tutawy
    Adenoma
    Temp Heteronymous superior quadrant Anopia

    variat :Earliest: craniopharyngeama


    ↳
    B/L superior Nasal fibre
    ↳ R

    III "I
    Temp Hetero Inferior QudAnopia
    ③ Ant chiasmal syndrome
    H

    I/ optic Nerve C/L


    +
    Inferonasal fibre
    1- Ant. Knee of you willebrand)

    *I
    ↳

    "l
    Junctional Scotoma of Traquir

    Post chiasmal
    #
    synd
    H
    B/L Nasal Macular fibre

    ↳ R

    ⑱ ⑲

    ④ Lateral optic chiasma


    ↓
    BIL temporal fibre
    ↳R

    My M
    BN Hemianopia/ Nasal Heteronymous
    Anopia
    3
    5 of
    =
    complete
    Lesion
    6 24B
    =

    l
    ⑦:OR C/ Homonymous
    Anopiq

    I
    Ill Temporal fibre it side
    I

    CIL Nasal fibre

    ↳R
    Photo R - 1st order

    "p "s
    ↓
    Bipolar Neuron -> and order
    ↓
    Ganglionic cell
    Iheader Right Homonymus
    Last - order
    4th
    damage
    ↳> optic Atrophy
    Hemianopia

    Feature OT LGB OR

    ① Pupil
    wernicke's
    ⑪ ⑪
    Pupil

    ② optic
    B/L 0. Atrophy B/L 0. Atrophy NO
    Atrophy

    ③ congruity Incongruous congrus


    Incongruous

    Lesion
    =>
    More towards occipital visual costex->
    -
    Move the
    congruent
    visual loss
    Partial
    # LGB Lesion #

    due to Thrombosis of due to Thrombosis


    of
    Lpch A (lateral post choroidal
    Ant choroidal
    Astery) Astery
    H
    H
    CL key Hole visual field defect
    /L sector sparing
    E.g. Lt LGB: Lpch A Homonymous
    Hemianopia

    ev eRr E. g. Lt LGB: ACGOA

    IIII IIII ↳R

    " "
    [Difference.
    Key Holevision

    BIL PCA lesion


    ↓
    affect BIL visual contex
    Except Tip]
    #Partial OR lesion#
    Distal OR/ Ant. OR
    =>
    L [Lt sides I

    ⑨ Bown's Lesion
    ⑧ Meyer's
    loop
    loop Lesion

    ↳ R
    ↳ R

    "III "I
    ClL "IIII "II
    CIL

    POTF:
    pie the floor
    pie in the
    PTS= sky
    on

    Boun
    Mnemonics: IT Meyer's loop ↓
    XI I Behind
    Inferior supreme ↓
    temporal Leades
    OR Inferior
    Lobe ↓
    superior
    Quadrantanopia

    Proximal
    ->
    =>
    OR/ Post OR E

    ↓ visual
    side) Ass Area
    & OR-Cortex Junction (It
    ↓ A V

    ↳ R
    OR occipital OR-Parietal
    -

    cortex lesion
    (Tumor
    ↓
    "I "II contex lesion
    (Trauma)
    ↓

    OkN Test
    OKN Test
    CIL Homonymous HemiAnopia
    · symmetric Erratic pursuit
    :

    m/V
    ⑪OkMyst I
    I
    Homo HemiAnopia
    Homony HemiAnopia
    ·OKN Test: optokinetic Mystagmus (Induced Nystagmus)

    ↳ Optokinetic Response -
    -> Fast phase Eye myxe saccade my

    (Normal)
    CIn opposite
    I
    directionof
    moving object)

    > Erratic (Ab -> slow phase Eye m/- persuit


    ↳ Irregular (smooth my

    saccade in directof
    moving obj)

    -
    Persuit staccade
    ⑩ visual cortexLesion (it

    ↓ ↓
    [Lt)
    Post Middle
    cerebral cerebral
    Astery Astere
    Lesion Lesion
    ↓
    ↓ Macular
    only
    Entire visual Involvement
    cortexExcept Tip
    ↓
    (Macular sparing)

    ⑧
    cookie cutter
    ↳ R
    I defect R

    IH,I " I D

    Tunnelvisions
    et ene
    Lesion: Anterior
    # Most Part
    of visual cortex
    ↓
    Parito -
    occipital fissue

    9
    ↳

    CIL Monoocular Temporal crescent

    Altitudinal visual field


    #

    defect

    G glaucoma
    =

    0 optic disc drysen


    =

    A AION (Ant Ischemic


    optic Neuropathy)
    =

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