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REVIEWER2
REVIEWER2
Strong forceful Displaced Very strong pulsation Not palpable *turn pt. Apical Beat that
pulsation/impulse apical Pushes the steth/Hands up on lateral decubitus-bring retracts during systole
5th ICS beat Displaced nearer to the chest wall*
MCL/10cm horizontally Horizontally/Vertically 5th/6th ICS Symptomatic patients:
Thick wall 5th ICS Very faintly palpable Mitral stenosis
Small cavity LAAL/12cm Seen in Patient with: beat on the middle of Congested heart disease
Thick wall Volume overload axilla Cor pulmonale
Seen in Patient with: Thick septum due to valvular disease *Global hypokinesia Pulmonary embolism
Chronic HPN Mitral regurgitation moving in 2D echo Rheumatic heart disease
Seen in Patient with: Aortic regurgitation o Left atrial enlargement
Chronic HPN Congenital Heart Seen in patient with: o Right ventricular
Disease Cardiomyopathy hypertrophy
Eg. Patent Ductus CAD
Arteriosus (PDA) Chronic Ischemia
Symptomatic patients: Heart Disease
DOB Coronary Bypass
Orthopnea Cox Virus
Peripheral edema Echo virus-viral
w/ jugular vein distention cardiomyopathy
On ECG Use of alcohol
o Small QRS- Hypokinetic Taking illicit drugs
On Xray
o Markedly enlarged heart
*Chronic Constrictive
Pericarditis
Carvallos’s sign – Tricuspid regurgitation Ebstein Anomaly – Sail Sound
Kussmauls sign – Constrictive pericarditis and Pericardial effusion Graham Steele – Pulmonary regurgitation
Hepatojugular – CHF Carey Coombs – Rheumatic heart Disease
Austin Flint – Chronic Aortic regurgitation Coartation of Aorta – BP elevated in UE and low in LE
Gallavardin’s sign – Aortic Stenosis ASD – Fixed Splitting of S2
Machinery like – PDA VSD – Holosystolic murmur at the Left parasternal area to right sternal
Intermittent Claudication – pain in the right calf upon walking can be border
relieve by rest
2nd RIGHT ICS PSL 2nd LEFT ICS PSL 3rd LEFT ICS 4th LEFT ICS PSB 5th LEFT ICS MAL
(AORTIC) (PULMONIC) (ERB’S POINT) (TRICUSPID) (MITRAL)
1. Paradoxical Splitting 1. Physiologic Splitting of S2 1. Mitral Valve Prolapse 1. Tricuspid Regurgitation 1. Mitral Regurgitation
of S2 2. Persistent Splitting of S2 - (MVP) 2. ASD 2. Mitral Stenosis
2. Aortic Stenosis 3. Pulmonic Aneurysm 2. Ventricular Septal 3. Physiologic Splitting of S1 3. S3
3. Aortic Aneurysm 4. Pulmonary Stenosis Defect (VSD) 4. Ebstein Anomaly 4. S4
5. PDA 3. Aortic Regurgitation 5. Hypertrophic 5. Dilated Cardiomyopathy
6. ASD 4. Quadruple Rhythm Cardiomyopathy
A. HEART SOUNDS:
S1 S2
LOUD A2 (LA-SA) LOUD P2 (LP-PA)
S1 S2
SPLITTING 1st-Closure of mitral valve 1st – Aortic
2nd – Pulmonic
WIDENING Complete RBBB RBBB
Delayed onset of RV pressure pulse Delayed closure of PV
Mitral regurgitation
REVERSED Severe mitral stenosis
LBBB
Left atrial myxoma
PHYSIOLOGIC Delayed closure does not vary with pulmonic
valve
ASD
RBBB
FIXED Does not vary with respiration
ASD
RVF
PARADOXICAL Does not vary on expiration and disappears
on inspiration
Delayed closure at AV
Aortic stenosis
LBBB
Hypertrophic Cardiomyopathy
C. SYSTOLIC SOUND
OPENING SNAP S3 S4
- Brief high pitch - Low pitched - Low pitched
- LLSB - Normal in children - Pre-systolic sound
- Radiates to the base of heart
REVIEW:
Normal
Apex: Loud S1 Soft S2
Based: Loud S2 Soft S1
The intensity of first heart sound is being affected by heart rate. In patient with very fast heart (FEVER, PREGNANCY, and HYPERTHYROIDISM –
S1 becomes loud all over
S3
-Mitral Regurgitation
-Hypertropic Cardiomyopathy
-Restrictive in early diastole (ventricular filling)
HEART MURMUR
Systolic Diastolic
MITRAL VALCE
TRICUSPID VALVE REGURGITATTION STENOSIS
(Holosystolic) (MID SYSTOLIC MURMUR)
SYSTOLIC DIASTOLIC
MIDSYSTOLIC (APAH) EARLY DIASTOLIC (APA)
- Aortic Stenosis - AORTIC REGURGITATION
- Pulmonic stenosis - PULMONIC REGURGITATION
- ASD - AUSTIN FLINT
- HOCM – Hypertrophic Cardiomyopathy
HOLOSYSTOLIC (MTV) MID-LATE DIASTOLIC (MT)
- Miral Valve Regurgitation - Mitral Stenosis
- Tricuspid Regurgitation - Tricuspid Stenosis
- VSD
LATE SYSTOLIC
- MVP
POSITIONING
Valsalva Increase hypertrophic cardiomyopathy murmur
Deep breathing Right sided murmur
Hand grip Left sided murmur
Standing from squatting MVP
Squatting from standing Decrease hypertrophic cardiomyopathy murmur
Squatting and leg raising Increase venous return to the heart
AUSCULTATION
Sitting and leaning Mild aortic
regurgitation
HEART
Aortic Stenosis Ejection systolic murmur transmitted into carotids
Crescendo-decrescendo murmur
Ejection clicks or sound
Paradoxical splitting of S2
Aortic regurgitation Diastolic blowing murmur
Wide systolic pressure
Hypertrophic
Cardiomyopathy
Atrial Septal Defect Fixed/persistent splitting of S2
Ventricular Septal Defect Holosystolic murmur → Heard at Erb’s, transmitted to the right sternal border
Tricuspid regurgitation 4th ICS parasternal line
Pulmonic stenosis RBBB
Mid-diastolic murmur → radiates to left shoulder
Crescendo-decrescendo
Persistent splitting of S2
Pulmonic regurgitation
Mitral stenosis Opening snap
Diastolic murmur
Loud S1
Mitral regurgitation Holosystolic murmur displaced to the left axilla/intrascapular
Diminished S1
Rumbling murmur
Mitral valve prolapses Mid-systolic click
Late systolic murmur
Non-ejection sound
Coarctation of aorta Greater BP in the UE compared to LE