Drug Action Mechanism of Side Indications Contraindication Nursing

Action Effects/ Responsibility

Adverse
Effects

Generic Proton Omeprazole Side Effects: Short-term treatment of active  Assess vital
Name: Pump suppresses CNS: duodenal ulcer; First-line signs before and
Omeprazol Inhibitors stomach acids Headache, therapy in treatment of after
e (PPI) ecretion bys dizziness, heartburn or symptoms of administration
pecific somnolence gastroesophageal reflux disease  Assess client
Brand inhibition of GI: nausea, (GERD);. history for any
Name: the H vomiting, contraindications
Omepron + /K + - diarrhea,  Assess for any
ATPase constipation, hypersensitivity
Dosage: (hydrogen- flatulence, to any
20mg potassium abdominal component of
adenosine pain the drug
Route: triphosphatase) Skin: Rash,  Assess drugs
Oral enzyme dermatitis, already being
system found pruritus taken for any
Frequency: at the secretory adverse drug
OD surface of interactions
gastric parietal  Explain the side
cells. effects that may
occur and also
danger signs for
adverse
reactions to
 Administer drug
with a whole
glass of water
 Caution patient
 to swallow
capsules whole
—not to open,
chew, or crush
them.
 Take the drug
before meals.
ASSESSMENT NURSING PLANNING INTERVENTION CORE NURSING EVALUATION
DIAGNOSIS VALUES THEORY

Subjective: Impaired Short term Goal: Independent:  Service  Theory of Human GOAL MET
Skin  Respect Caring by Jean
Integrity After 24 hrs of  Simplicity Watson Short term Goal:
Objective: related to nursing 1. Repostion the  Vincentian
 (+) prolonged intervention the patient every Excellence  After 24 hrs of nursing
Grade 2 pressure client will be able 2 hours.  Watson's intervention the client
bed to: Philosophy was able to:
sores on Rationale: and Science of
left and  This is to Caring
right  Prevent relieve revolves  Prevent further
buttocks further pressure and around the worsening of
 Blisters worsening of avoid way in which existing pressure
on both existing constant nurses ulcers.
buttocks pressure contanct with manifest care  Show signs of
 Redness ulcers. the bed and towards their improvement
 Swelling  Show signs prevent patients. Her regarding
of pressure theory existing
improvemen ulcer underscores bedsores.
Diagnostic t regarding development. the integration
Tests: existing of humanistic PARTIALLY GOAL
bedsores. elements into MET
GCS: 7 2. Use pressure nursing,
Long term relieving alongside Long term
devices such scientific
After 3 days of as pillows knowledge and After 3 days of nursing
nursing and cushions. nursing intervention the client
intervention the techniques. In was able to:
client will be able Rationale: accordance
to:  The use of with her
these devices theory, nurses  Prevent
prevent have the development of
 Prevent development capacity to new pressure
development of new exhibit and ulcers.
of new pressure apply care in
pressure ulcers their practice.
ulcers. especially if The act of  Reduce the
bony caring for appearance of
prominces patients fosters existing pressure
 Reduce the such as the personal ulcers.
appearance sacrum will development,
of existing be protected. and within a
pressure caring
ulcers. environment,
3. Conduct individuals are
regular skin embraced for
assessment,  who they are,
monitor for with a focus on
any signs of their potential
infection and for
document any transformation.
changes.

Rationale:
 To give
appropriate
measures
regarding the
condition to
existing
 pressure
ulcer and
relay
information
to the client’s
attending
physician.

4. Monitor the
client’s
laboratory
results.

Rationale:
 Abnormalitie
s in
laboratory
results may
be an
indicator of
infection and
causes of
delayed
wound
healing.

Dependent

1. Apply
Calmoseptine
to affected
 areas as
ordered.

Rationale: 
 This drug is
used to
soothe and
helps
promote
healing of
impaired skin
integrity
related to bed
sores.

Collaborative

1. Collaborate
with a
dietitian
inorder to
give adequate
nutrition that
is appropriate
to the needs
of the client.

Rationale:
 Poor
nutrition may
interfere with
the immune
function that
cause the
delayed the
wound
healing
process of
existing
pressure
ulcers.