Professional Documents
Culture Documents
Nursing Care Plan
Nursing Care Plan
Name of Drugs Dosage/ Mechanism of Action Indication Contraindication Adverse Effect Nursing responsibilities
Frequency/
Timing/
Route
Generic Name: Below 12 The mucolytic effect of Acute & chronic Known Significant: Maintaining Patent Airway
N- years: 5-10 acetylcysteine (Fluimicil) resp tract hypersensitivity to The most frequent adverse Clearance by Assessing the rate,
acetylcysteine ml of is linked to the sulfhydryl infections w/ acetylcysteine or to events associated with the rhythm, and depth of respiration,
10/20% group in the molecule. abundant mucus any of the excipients. oral administration of chest movement, and use of
Brand Name: solution by This group is likely to secretions due to Acetylcysteine acetylcysteine are accessory muscles
Fluimucil nebulizer open disulphide bonds in acute bronchitis, (Fluimucil) granules gastrointestinal in nature.
every 6-8 hr the mucosa, reducing chronic and tablets are Hypersensitivity reactions Monitoring cough effectiveness
Classification: PRN. viscosity. bronchitis & its contraindicated in including anaphylactic and productivity
Mucolytics exacerbations, patients suffering shock,
Acetylcysteine (Fluimucil) pulmonary from anaphylactic/anaphylactoi Auscultate lung fields, noting areas
Restores hepatic emphysema, phenylketonuria due d reaction, bronchospasm, of decreased or absent airflow and
glutathione levels by mucoviscidosis to the aspartame angioedema, rash and adventitious breath sounds:
producing the glutathione & bronchiectasis. content. pruritus have been crackles, wheezes. And observe the
precursor L-cysteine. To clear phlegm Children below 2 reported less frequently. sputum color, viscosity, and odor.
Glutathione must and strengthen years of age. Having headache, nausea, Report changes.
inactivate an intermediate lung functions. vomiting, tachycardia,
metabolite (N-acetyl-p- Assess the patient’s hydration
benzoquinoneimine) of status and proper hygiene.
paracetamol, which is
believed to be hepatotoxic.
In the case of
acetaminophen overdose,
excessive levels of this
metabolite are formed
because the primary
metabolic pathways
(glucuronide and sulfate
conjugation) are saturated.