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Neuroscience 4th Edition Lundy Test Bank
Neuroscience 4th Edition Lundy Test Bank
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Lundy-Ekman: Neuroscience: Fundamentals for Rehabilitation, 4th Edition
Chapter 9: Autonomic Nervous System
Test Bank
ANS: E
Rationale: Stimulation of nociceptive second-order neurons results in pain sensation referred
to the umbilical region (see Figure 9-4).
2. Areas of the central nervous system (CNS) that modulate autonomic control include which
one of the following?
A. Thalamus
B. Hypothalamus
C. Limbic system
D. Both A and B
E. A, B, and C
ANS: E
Rationale: The hypothalamus, thalamus, and limbic system modulate brainstem autonomic
control.
3. Which one of the following is NOT a characteristic of the autonomic efferent system?
A. Innervates blood vessels in skeletal muscle.
B. Hormones regulate effector control.
C. Is usually a two-neuron pathway with a synapse outside the CNS.
D. Activation of effectors is usually voluntary.
E. Neurotransmitters are used for signaling.
ANS: D.
Rationale: Unlike the somatic nervous system, regulation of autonomic functions is typically
nonconscious and can be exerted by hormones.
D. Dilation of bronchioles
E. Increased heart rate
ANS: C
Rationale: The role of the sympathetic nervous system is often illustrated by describing the
physiologic responses to fear. When an individual feels threatened, the sympathetic nervous
system prepares for vigorous muscle activity; that is, it prepares for fight or flight.
Vasoconstriction in the skin and gut increases blood flow to active muscles. Blood glucose
levels increase, bronchi and coronary vessels dilate, and blood pressure and heart rate
increase. Simultaneously, sympathetic firing reduces activity in the digestive system.
ANS: A
Rationale: Skeletal muscle veins and venules are called capacitance vessels because blood
pools in these vessels when their walls are relaxed.
6. Drugs that block beta-1 (β1) receptors are used to treat which one of the following?
A. Asthma
B. Excess sweating
C. Metabolic disorders
D. Tachycardia
E. Vagus nerve disorders
ANS: D
Rationale: β1-blockers decrease heart rate and contractility without affecting the airways.
7. Where are the cell bodies of the sympathetic preganglionic neurons located?
A. Autonomic nuclei of the brainstem
B. Lateral horn of spinal cord T1-L2 levels
C. Lateral horn of spinal cord S2-S4 levels
D. Ganglia adjacent to the spinal cord
E. Ganglia near the thoracic cavity
ANS: B
Rationale: Cell bodies of the sympathetic preganglionic neurons are located in the lateral horn
of the spinal cord gray matter from the T1 to L2 levels.
C. Periaqueductal gray
D. Both A and B
E. A, B, and C
ANS: D
Rationale: Sympathetic effects on the thoracic viscera include increasing heart rate and
contractility (see Figure 9-11). Parasympathetic efferent fibers in the vagus nerve innervate
the heart and the smooth muscle of the lungs and digestive system. Vagus nerve activity to the
heart produces either bradycardia (slowing of the heart rate) or decreased cardiac contraction
force.
ANS: E
Rationale: If a lesion affects the sympathetic pathway to the head, then sympathetic activity
on one side of the head is decreased. This reduction leads to ipsilateral drooping of the upper
eyelid, constriction of the pupil, and skin vasodilation, with the absence of sweating on the
ipsilateral side of the face and neck.
ANS: C
Rationale: Syncope (fainting) is a brief loss of consciousness caused by inadequate blood flow
to the brain. Causes include powerful emotions, insufficient cardiac output, hypoxia, and
hypoglycemia.