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  • Patho Acute Bronchi

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    Marianne Bagui Dinglasan
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    The document discusses the etiological factors and progression of bronchitis. It begins with common causes like cough, cold, and fever which can allow bacteria like during childhood to become trapped in mucus in the lungs. The bacteria then release toxins that damage cilia and epithelial cells, impairing the lungs' ability to clear mucus and making the person more susceptible to chronic bronchitis. As bronchitis progresses, cilia are replaced with goblet cells, causing inflammation of the bronchial walls, thickening, and excess mucus production that obstruct airflow.

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    PATHO ACUTE BRONCHI

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    The document discusses the etiological factors and progression of bronchitis. It begins with common causes like cough, cold, and fever which can allow bacteria like during childhood to become trapped in mucus in the lungs. The bacteria then release toxins that damage cilia and epithelial cells, impairing the lungs' ability to clear mucus and making the person more susceptible to chronic bronchitis. As bronchitis progresses, cilia are replaced with goblet cells, causing inflammation of the bronchial walls, thickening, and excess mucus production that obstruct airflow.

    Copyright:

    © All Rights Reserved
    Download as DOCX, PDF, TXT or read online from Scribd
    Flag for inappropriate content
    Download as docx, pdf, or txt
    10 views1 page

    Patho Acute Bronchi

    Uploaded by

    Marianne Bagui Dinglasan
    The document discusses the etiological factors and progression of bronchitis. It begins with common causes like cough, cold, and fever which can allow bacteria like during childhood to become trapped in mucus in the lungs. The bacteria then release toxins that damage cilia and epithelial cells, impairing the lungs' ability to clear mucus and making the person more susceptible to chronic bronchitis. As bronchitis progresses, cilia are replaced with goblet cells, causing inflammation of the bronchial walls, thickening, and excess mucus production that obstruct airflow.

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    of 1

    ETIOLOGICAL FACTOR

    Cough
    Cold
    Fever

    Compromised Immune System


    Bacteria
    esp. in children

    Becomes trapped in sticky mucus &


    swept upwards and outwards by cilia

    Building of Cilia slows down and Bacteria release toxins to further damage
    becomes paralyzed and disappear the cilia and epithelial cells

    Cilia cells are replaced by goblet


    cells as bronchitis progress

    Constant Coughing tends to


    be a combination of:

    Air Inflammation Excessive Bronchial


    Increased Cough
    Secretion
    Receptor Sensitivity

    Inflammatory cells
    begin with chemical
    alarm

    Release of Macrophages release


    Released by intruder stress
    Interleukin 4,5 & cytokines & interferon
    tissue cells & immune cells
    histamine gamma

    Inflammation of Bronchial Wall

    Goblet Cell Hyperplasia and Thickening of wall


    Hypertrophy narrows passages

    Increase obstruction to Air passages becomes clogged


    airflow

    Crackles/Crackling
    Hypersecretion of Mucus Sound

    Diminishing Bronchial Mucociliary Function

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