wr ete aay lets Wel G Yo rae UL SES AT | SA SAV CEE ah Sn, CA ie ie Bh ae = Geese kph, MP OSE L 59 eshte Ghoge panpnit pp ph AEE Xe nin RPI CRIN Ried Se ees Sake 6 BNR 88a BR Cid ee ‘oe see 21 Phosphe eral pyruvde 7 SP Ral REE A — 18c) — IWnoliteacid J teh cere tialtlet pb ors i Atu¥S ws O «tabled ADU Soe ¢ , ete = (PEK) Phaiphe fraclo Kinase P) S cly Cush > wi Sou Dh Se oe Soy 7 te 2 Nes nnn bandana) / Ge Wes abtoo rl Pua Let pits Pian San ce » pyravate AN Glycine ty doe DN Sed! poy (0 iL phe ) iriguali GAF del, Mee oes + Cand protine oo cael (ress ne Xe Tai Sule . ‘gous THE puke we Mee ta dalek 56 cs 7 ~ Cihsine — Guefrtaed ead bel We nas L Mucleases JEL eo reel BRU AE IB Lol ate mv)Fa the ces of the following table with the suitabla required Information Pyruvate hinase ‘Giycogen synniase Glocounase ver veoren shosohorvlase sobitrate ‘roster natvator “Alosterc inhibitor Phosphorylation (yes (Na inhibition oF activation ‘Sie of pathway~ a Predict we Enzyme TATP NADH | Citrate | Acetyle F2,6 | In (X) no effects in| ple ‘cAMP | Co-A | bis p_| _ | | = Glycogen Citrate synthase Phosphofructokinasel i = Fructose Glyceraldehyde-3-p Dehydrogenase | _ Pyruvate dehydrogenase Enolase aez Isocitrate dehydrogenase __| Lt OY show the Ye taken ° glycogen sn nbyase curd phospho wat ar ylose a ete yee & 9 Weer pins Ghee tennetm FAAad OW rch om F Me i baer 5 uO x ASV 52 Zb-p 6 @ IF sys, Case BTN Bd Dy yea cad Ye odd Sy Vai tie ay os 72e é SOS 5 ASAD ow SoD tb lt MD Ce SU - spear te) Gayla, wo ay - wii euts\ Bid A yaa e Oi Asa sua. PAW NWSE Lp ole Ht 2 Saii Ai) gett) PN = d+— is a Zs whe ae Mie lt eg See